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classassstb-sticker-stickertickerb-ISBN9785970438831-0046-000ISBN9785970438831-0046-000lass="">Inflammation is a universal response of the body to the influence of various exogenic and endogenous damaging factors, for example bacterial, viral, parasitic infections as well as allergens, physical and chemical stimuli. They cause both local and systemic (generalized) reactions. Inflammatory reactions occur in many pathological conditions of infectious and non-infectious aetiology; they may be very severe, leading to organs and tissue function disturbances. Therefore, it is obvious that, besides etiotropic therapy (if it is available), proper management of the inflammatory process is of great practical importance. Nowadays anti-inflammatory agents are the integral components of pharmacotherapy of quite a few diseases and pathological conditions.ss="">Inflammation is a universal response of the body to the influence of various exogenic and endogenous damaging factors, for example bacterial, viral, parasitic infections as well as allergens, physical and chemical stimuli. They cause both local and systemic (generalized) reactions. Inflammatory reactions occur in many pathological conditions of infectious and non-infectious aetiology; they may be very severe, leading to organs and tissue function disturbances. Therefore, it is obvious that, besides etiotropic therapy (if it is available), proper management of the inflammatory process is of great practical importance. Nowadays anti-inflammatory agents are the integral components of pharmacotherapy of quite a few diseases and pathological conditions.="">Inflammation is a universal response of the body to the influence of various exogenic and endogenous damaging factors, for example bacterial, viral, parasitic infections as well as allergens, physical and chemical stimuli. They cause both local and systemic (generalized) reactions. Inflammatory reactions occur in many pathological conditions of infectious and non-infectious aetiology; they may be very severe, leading to organs and tissue function disturbances. Therefore, it is obvious that, besides etiotropic therapy (if it is available), proper management of the inflammatory process is of great practical importance. Nowadays anti-inflammatory agents are the integral components of pharmacotherapy of quite a few diseases and pathological conditions.">Inflammation is a universal response of the body to the influence of various exogenic and endogenous damaging factors, for example bacterial, viral, parasitic infections as well as allergens, physical and chemical stimuli. They cause both local and systemic (generalized) reactions. Inflammatory reactions occur in many pathological conditions of infectious and non-infectious aetiology; they may be very severe, leading to organs and tissue function disturbances. Therefore, it is obvious that, besides etiotropic therapy (if it is available), proper management of the inflammatory process is of great practical importance. Nowadays anti-inflammatory agents are the integral components of pharmacotherapy of quite a few diseases and pathological conditions.Inflammation is a universal response of the body to the influence of various exogenic and endogenous damaging factors, for example bacterial, viral, parasitic infections as well as allergens, physical and chemical stimuli. They cause both local and systemic (generalized) reactions. Inflammatory reactions occur in many pathological conditions of infectious and non-infectious aetiology; they may be very severe, leading to organs and tissue function disturbances. Therefore, it is obvious that, besides etiotropic therapy (if it is available), proper management of the inflammatory process is of great practical importance. Nowadays anti-inflammatory agents are the integral components of pharmacotherapy of quite a few diseases and pathological conditions.flammation is a universal response of the body to the influence of various exogenic and endogenous damaging factors, for example bacterial, viral, parasitic infections as well as allergens, physical and chemical stimuli. They cause both local and systemic (generalized) reactions. Inflammatory reactions occur in many pathological conditions of infectious and non-infectious aetiology; they may be very severe, leading to organs and tissue function disturbances. Therefore, it is obvious that, besides etiotropic therapy (if it is available), proper management of the inflammatory process is of great practical importance. Nowadays anti-inflammatory agents are the integral components of pharmacotherapy of quite a few diseases and pathological conditions.ammation is a universal response of the body to the influence of various exogenic and endogenous damaging factors, for example bacterial, viral, parasitic infections as well as allergens, physical and chemical stimuli. They cause both local and systemic (generalized) reactions. Inflammatory reactions occur in many pathological conditions of infectious and non-infectious aetiology; they may be very severe, leading to organs and tissue function disturbances. Therefore, it is obvious that, besides etiotropic therapy (if it is available), proper management of the inflammatory process is of great practical importance. Nowadays anti-inflammatory agents are the integral components of pharmacotherapy of quite a few diseases and pathological conditions.tion is a universal response of the body to the influence of various exogenic and endogenous damaging factors, for example bacterial, viral, parasitic infections as well as allergens, physical and chemical stimuli. They cause both local and systemic (generalized) reactions. Inflammatory reactions occur in many pathological conditions of infectious and non-infectious aetiology; they may be very severe, leading to organs and tissue function disturbances. Therefore, it is obvious that, besides etiotropic therapy (if it is available), proper management of the inflammatory process is of great practical importance. Nowadays anti-inflammatory agents are the integral components of pharmacotherapy of quite a few diseases and pathological conditions.on is a universal response of the body to the influence of various exogenic and endogenous damaging factors, for example bacterial, viral, parasitic infections as well as allergens, physical and chemical stimuli. They cause both local and systemic (generalized) reactions. Inflammatory reactions occur in many pathological conditions of infectious and non-infectious aetiology; they may be very severe, leading to organs and tissue function disturbances. Therefore, it is obvious that, besides etiotropic therapy (if it is available), proper management of the inflammatory process is of great practical importance. Nowadays anti-inflammatory agents are the integral components of pharmacotherapy of quite a few diseases and pathological conditions. is a universal response of the body to the influence of various exogenic and endogenous damaging factors, for example bacterial, viral, parasitic infections as well as allergens, physical and chemical stimuli. They cause both local and systemic (generalized) reactions. Inflammatory reactions occur in many pathological conditions of infectious and non-infectious aetiology; they may be very severe, leading to organs and tissue function disturbances. Therefore, it is obvious that, besides etiotropic therapy (if it is available), proper management of the inflammatory process is of great practical importance. Nowadays anti-inflammatory agents are the integral components of pharmacotherapy of quite a few diseases and pathological conditions.a universal response of the body to the influence of various exogenic and endogenous damaging factors, for example bacterial, viral, parasitic infections as well as allergens, physical and chemical stimuli. They cause both local and systemic (generalized) reactions. Inflammatory reactions occur in many pathological conditions of infectious and non-infectious aetiology; they may be very severe, leading to organs and tissue function disturbances. Therefore, it is obvious that, besides etiotropic therapy (if it is available), proper management of the inflammatory process is of great practical importance. Nowadays anti-inflammatory agents are the integral components of pharmacotherapy of quite a few diseases and pathological conditions.universal response of the body to the influence of various exogenic and endogenous damaging factors, for example bacterial, viral, parasitic infections as well as allergens, physical and chemical stimuli. They cause both local and systemic (generalized) reactions. Inflammatory reactions occur in many pathological conditions of infectious and non-infectious aetiology; they may be very severe, leading to organs and tissue function disturbances. Therefore, it is obvious that, besides etiotropic therapy (if it is available), proper management of the inflammatory process is of great practical importance. Nowadays anti-inflammatory agents are the integral components of pharmacotherapy of quite a few diseases and pathological conditions.sal response of the body to the influence of various exogenic and endogenous damaging factors, for example bacterial, viral, parasitic infections as well as allergens, physical and chemical stimuli. They cause both local and systemic (generalized) reactions. Inflammatory reactions occur in many pathological conditions of infectious and non-infectious aetiology; they may be very severe, leading to organs and tissue function disturbances. Therefore, it is obvious that, besides etiotropic therapy (if it is available), proper management of the inflammatory process is of great practical importance. Nowadays anti-inflammatory agents are the integral components of pharmacotherapy of quite a few diseases and pathological conditions.l response of the body to the influence of various exogenic and endogenous damaging factors, for example bacterial, viral, parasitic infections as well as allergens, physical and chemical stimuli. They cause both local and systemic (generalized) reactions. Inflammatory reactions occur in many pathological conditions of infectious and non-infectious aetiology; they may be very severe, leading to organs and tissue function disturbances. Therefore, it is obvious that, besides etiotropic therapy (if it is available), proper management of the inflammatory process is of great practical importance. Nowadays anti-inflammatory agents are the integral components of pharmacotherapy of quite a few diseases and pathological conditions.response of the body to the influence of various exogenic and endogenous damaging factors, for example bacterial, viral, parasitic infections as well as allergens, physical and chemical stimuli. They cause both local and systemic (generalized) reactions. Inflammatory reactions occur in many pathological conditions of infectious and non-infectious aetiology; they may be very severe, leading to organs and tissue function disturbances. Therefore, it is obvious that, besides etiotropic therapy (if it is available), proper management of the inflammatory process is of great practical importance. Nowadays anti-inflammatory agents are the integral components of pharmacotherapy of quite a few diseases and pathological conditions. body to the influence of various exogenic and endogenous damaging factors, for example bacterial, viral, parasitic infections as well as allergens, physical and chemical stimuli. They cause both local and systemic (generalized) reactions. Inflammatory reactions occur in many pathological conditions of infectious and non-infectious aetiology; they may be very severe, leading to organs and tissue function disturbances. Therefore, it is obvious that, besides etiotropic therapy (if it is available), proper management of the inflammatory process is of great practical importance. Nowadays anti-inflammatory agents are the integral components of pharmacotherapy of quite a few diseases and pathological conditions.ody to the influence of various exogenic and endogenous damaging factors, for example bacterial, viral, parasitic infections as well as allergens, physical and chemical stimuli. They cause both local and systemic (generalized) reactions. Inflammatory reactions occur in many pathological conditions of infectious and non-infectious aetiology; they may be very severe, leading to organs and tissue function disturbances. Therefore, it is obvious that, besides etiotropic therapy (if it is available), proper management of the inflammatory process is of great practical importance. Nowadays anti-inflammatory agents are the integral components of pharmacotherapy of quite a few diseases and pathological conditions.y to the influence of various exogenic and endogenous damaging factors, for example bacterial, viral, parasitic infections as well as allergens, physical and chemical stimuli. They cause both local and systemic (generalized) reactions. Inflammatory reactions occur in many pathological conditions of infectious and non-infectious aetiology; they may be very severe, leading to organs and tissue function disturbances. Therefore, it is obvious that, besides etiotropic therapy (if it is available), proper management of the inflammatory process is of great practical importance. Nowadays anti-inflammatory agents are the integral components of pharmacotherapy of quite a few diseases and pathological conditions.o the influence of various exogenic and endogenous damaging factors, for example bacterial, viral, parasitic infections as well as allergens, physical and chemical stimuli. They cause both local and systemic (generalized) reactions. Inflammatory reactions occur in many pathological conditions of infectious and non-infectious aetiology; they may be very severe, leading to organs and tissue function disturbances. Therefore, it is obvious that, besides etiotropic therapy (if it is available), proper management of the inflammatory process is of great practical importance. Nowadays anti-inflammatory agents are the integral components of pharmacotherapy of quite a few diseases and pathological conditions.the influence of various exogenic and endogenous damaging factors, for example bacterial, viral, parasitic infections as well as allergens, physical and chemical stimuli. They cause both local and systemic (generalized) reactions. Inflammatory reactions occur in many pathological conditions of infectious and non-infectious aetiology; they may be very severe, leading to organs and tissue function disturbances. Therefore, it is obvious that, besides etiotropic therapy (if it is available), proper management of the inflammatory process is of great practical importance. Nowadays anti-inflammatory agents are the integral components of pharmacotherapy of quite a few diseases and pathological conditions.e influence of various exogenic and endogenous damaging factors, for example bacterial, viral, parasitic infections as well as allergens, physical and chemical stimuli. They cause both local and systemic (generalized) reactions. Inflammatory reactions occur in many pathological conditions of infectious and non-infectious aetiology; they may be very severe, leading to organs and tissue function disturbances. Therefore, it is obvious that, besides etiotropic therapy (if it is available), proper management of the inflammatory process is of great practical importance. Nowadays anti-inflammatory agents are the integral components of pharmacotherapy of quite a few diseases and pathological conditions.c and endogenous damaging factors, for example bacterial, viral, parasitic infections as well as allergens, physical and chemical stimuli. They cause both local and systemic (generalized) reactions. Inflammatory reactions occur in many pathological conditions of infectious and non-infectious aetiology; they may be very severe, leading to organs and tissue function disturbances. Therefore, it is obvious that, besides etiotropic therapy (if it is available), proper management of the inflammatory process is of great practical importance. Nowadays anti-inflammatory agents are the integral components of pharmacotherapy of quite a few diseases and pathological conditions.and endogenous damaging factors, for example bacterial, viral, parasitic infections as well as allergens, physical and chemical stimuli. They cause both local and systemic (generalized) reactions. Inflammatory reactions occur in many pathological conditions of infectious and non-infectious aetiology; they may be very severe, leading to organs and tissue function disturbances. Therefore, it is obvious that, besides etiotropic therapy (if it is available), proper management of the inflammatory process is of great practical importance. Nowadays anti-inflammatory agents are the integral components of pharmacotherapy of quite a few diseases and pathological conditions.d endogenous damaging factors, for example bacterial, viral, parasitic infections as well as allergens, physical and chemical stimuli. They cause both local and systemic (generalized) reactions. Inflammatory reactions occur in many pathological conditions of infectious and non-infectious aetiology; they may be very severe, leading to organs and tissue function disturbances. Therefore, it is obvious that, besides etiotropic therapy (if it is available), proper management of the inflammatory process is of great practical importance. Nowadays anti-inflammatory agents are the integral components of pharmacotherapy of quite a few diseases and pathological conditions.endogenous damaging factors, for example bacterial, viral, parasitic infections as well as allergens, physical and chemical stimuli. They cause both local and systemic (generalized) reactions. Inflammatory reactions occur in many pathological conditions of infectious and non-infectious aetiology; they may be very severe, leading to organs and tissue function disturbances. Therefore, it is obvious that, besides etiotropic therapy (if it is available), proper management of the inflammatory process is of great practical importance. Nowadays anti-inflammatory agents are the integral components of pharmacotherapy of quite a few diseases and pathological conditions.dogenous damaging factors, for example bacterial, viral, parasitic infections as well as allergens, physical and chemical stimuli. They cause both local and systemic (generalized) reactions. Inflammatory reactions occur in many pathological conditions of infectious and non-infectious aetiology; they may be very severe, leading to organs and tissue function disturbances. Therefore, it is obvious that, besides etiotropic therapy (if it is available), proper management of the inflammatory process is of great practical importance. Nowadays anti-inflammatory agents are the integral components of pharmacotherapy of quite a few diseases and pathological conditions.genous damaging factors, for example bacterial, viral, parasitic infections as well as allergens, physical and chemical stimuli. They cause both local and systemic (generalized) reactions. Inflammatory reactions occur in many pathological conditions of infectious and non-infectious aetiology; they may be very severe, leading to organs and tissue function disturbances. Therefore, it is obvious that, besides etiotropic therapy (if it is available), proper management of the inflammatory process is of great practical importance. Nowadays anti-inflammatory agents are the integral components of pharmacotherapy of quite a few diseases and pathological conditions.s damaging factors, for example bacterial, viral, parasitic infections as well as allergens, physical and chemical stimuli. They cause both local and systemic (generalized) reactions. Inflammatory reactions occur in many pathological conditions of infectious and non-infectious aetiology; they may be very severe, leading to organs and tissue function disturbances. Therefore, it is obvious that, besides etiotropic therapy (if it is available), proper management of the inflammatory process is of great practical importance. Nowadays anti-inflammatory agents are the integral components of pharmacotherapy of quite a few diseases and pathological conditions.damaging factors, for example bacterial, viral, parasitic infections as well as allergens, physical and chemical stimuli. They cause both local and systemic (generalized) reactions. Inflammatory reactions occur in many pathological conditions of infectious and non-infectious aetiology; they may be very severe, leading to organs and tissue function disturbances. Therefore, it is obvious that, besides etiotropic therapy (if it is available), proper management of the inflammatory process is of great practical importance. Nowadays anti-inflammatory agents are the integral components of pharmacotherapy of quite a few diseases and pathological conditions.maging factors, for example bacterial, viral, parasitic infections as well as allergens, physical and chemical stimuli. They cause both local and systemic (generalized) reactions. Inflammatory reactions occur in many pathological conditions of infectious and non-infectious aetiology; they may be very severe, leading to organs and tissue function disturbances. Therefore, it is obvious that, besides etiotropic therapy (if it is available), proper management of the inflammatory process is of great practical importance. Nowadays anti-inflammatory agents are the integral components of pharmacotherapy of quite a few diseases and pathological conditions.as well as allergens, physical and chemical stimuli. They cause both local and systemic (generalized) reactions. Inflammatory reactions occur in many pathological conditions of infectious and non-infectious aetiology; they may be very severe, leading to organs and tissue function disturbances. Therefore, it is obvious that, besides etiotropic therapy (if it is available), proper management of the inflammatory process is of great practical importance. Nowadays anti-inflammatory agents are the integral components of pharmacotherapy of quite a few diseases and pathological conditions. well as allergens, physical and chemical stimuli. They cause both local and systemic (generalized) reactions. Inflammatory reactions occur in many pathological conditions of infectious and non-infectious aetiology; they may be very severe, leading to organs and tissue function disturbances. Therefore, it is obvious that, besides etiotropic therapy (if it is available), proper management of the inflammatory process is of great practical importance. Nowadays anti-inflammatory agents are the integral components of pharmacotherapy of quite a few diseases and pathological conditions.ell as allergens, physical and chemical stimuli. They cause both local and systemic (generalized) reactions. Inflammatory reactions occur in many pathological conditions of infectious and non-infectious aetiology; they may be very severe, leading to organs and tissue function disturbances. Therefore, it is obvious that, besides etiotropic therapy (if it is available), proper management of the inflammatory process is of great practical importance. Nowadays anti-inflammatory agents are the integral components of pharmacotherapy of quite a few diseases and pathological conditions. allergens, physical and chemical stimuli. They cause both local and systemic (generalized) reactions. Inflammatory reactions occur in many pathological conditions of infectious and non-infectious aetiology; they may be very severe, leading to organs and tissue function disturbances. Therefore, it is obvious that, besides etiotropic therapy (if it is available), proper management of the inflammatory process is of great practical importance. Nowadays anti-inflammatory agents are the integral components of pharmacotherapy of quite a few diseases and pathological conditions.llergens, physical and chemical stimuli. They cause both local and systemic (generalized) reactions. Inflammatory reactions occur in many pathological conditions of infectious and non-infectious aetiology; they may be very severe, leading to organs and tissue function disturbances. Therefore, it is obvious that, besides etiotropic therapy (if it is available), proper management of the inflammatory process is of great practical importance. Nowadays anti-inflammatory agents are the integral components of pharmacotherapy of quite a few diseases and pathological conditions.ergens, physical and chemical stimuli. They cause both local and systemic (generalized) reactions. Inflammatory reactions occur in many pathological conditions of infectious and non-infectious aetiology; they may be very severe, leading to organs and tissue function disturbances. Therefore, it is obvious that, besides etiotropic therapy (if it is available), proper management of the inflammatory process is of great practical importance. Nowadays anti-inflammatory agents are the integral components of pharmacotherapy of quite a few diseases and pathological conditions.and chemical stimuli. They cause both local and systemic (generalized) reactions. Inflammatory reactions occur in many pathological conditions of infectious and non-infectious aetiology; they may be very severe, leading to organs and tissue function disturbances. Therefore, it is obvious that, besides etiotropic therapy (if it is available), proper management of the inflammatory process is of great practical importance. Nowadays anti-inflammatory agents are the integral components of pharmacotherapy of quite a few diseases and pathological conditions.d chemical stimuli. They cause both local and systemic (generalized) reactions. Inflammatory reactions occur in many pathological conditions of infectious and non-infectious aetiology; they may be very severe, leading to organs and tissue function disturbances. Therefore, it is obvious that, besides etiotropic therapy (if it is available), proper management of the inflammatory process is of great practical importance. Nowadays anti-inflammatory agents are the integral components of pharmacotherapy of quite a few diseases and pathological conditions.chemical stimuli. They cause both local and systemic (generalized) reactions. Inflammatory reactions occur in many pathological conditions of infectious and non-infectious aetiology; they may be very severe, leading to organs and tissue function disturbances. Therefore, it is obvious that, besides etiotropic therapy (if it is available), proper management of the inflammatory process is of great practical importance. Nowadays anti-inflammatory agents are the integral components of pharmacotherapy of quite a few diseases and pathological conditions.c (generalized) reactions. Inflammatory reactions occur in many pathological conditions of infectious and non-infectious aetiology; they may be very severe, leading to organs and tissue function disturbances. Therefore, it is obvious that, besides etiotropic therapy (if it is available), proper management of the inflammatory process is of great practical importance. Nowadays anti-inflammatory agents are the integral components of pharmacotherapy of quite a few diseases and pathological conditions.(generalized) reactions. Inflammatory reactions occur in many pathological conditions of infectious and non-infectious aetiology; they may be very severe, leading to organs and tissue function disturbances. Therefore, it is obvious that, besides etiotropic therapy (if it is available), proper management of the inflammatory process is of great practical importance. Nowadays anti-inflammatory agents are the integral components of pharmacotherapy of quite a few diseases and pathological conditions.eneralized) reactions. Inflammatory reactions occur in many pathological conditions of infectious and non-infectious aetiology; they may be very severe, leading to organs and tissue function disturbances. Therefore, it is obvious that, besides etiotropic therapy (if it is available), proper management of the inflammatory process is of great practical importance. Nowadays anti-inflammatory agents are the integral components of pharmacotherapy of quite a few diseases and pathological conditions.eralized) reactions. Inflammatory reactions occur in many pathological conditions of infectious and non-infectious aetiology; they may be very severe, leading to organs and tissue function disturbances. Therefore, it is obvious that, besides etiotropic therapy (if it is available), proper management of the inflammatory process is of great practical importance. Nowadays anti-inflammatory agents are the integral components of pharmacotherapy of quite a few diseases and pathological conditions.alized) reactions. Inflammatory reactions occur in many pathological conditions of infectious and non-infectious aetiology; they may be very severe, leading to organs and tissue function disturbances. Therefore, it is obvious that, besides etiotropic therapy (if it is available), proper management of the inflammatory process is of great practical importance. Nowadays anti-inflammatory agents are the integral components of pharmacotherapy of quite a few diseases and pathological conditions.ized) reactions. Inflammatory reactions occur in many pathological conditions of infectious and non-infectious aetiology; they may be very severe, leading to organs and tissue function disturbances. Therefore, it is obvious that, besides etiotropic therapy (if it is available), proper management of the inflammatory process is of great practical importance. Nowadays anti-inflammatory agents are the integral components of pharmacotherapy of quite a few diseases and pathological conditions.ed) reactions. Inflammatory reactions occur in many pathological conditions of infectious and non-infectious aetiology; they may be very severe, leading to organs and tissue function disturbances. Therefore, it is obvious that, besides etiotropic therapy (if it is available), proper management of the inflammatory process is of great practical importance. Nowadays anti-inflammatory agents are the integral components of pharmacotherapy of quite a few diseases and pathological conditions.reactions. Inflammatory reactions occur in many pathological conditions of infectious and non-infectious aetiology; they may be very severe, leading to organs and tissue function disturbances. Therefore, it is obvious that, besides etiotropic therapy (if it is available), proper management of the inflammatory process is of great practical importance. Nowadays anti-inflammatory agents are the integral components of pharmacotherapy of quite a few diseases and pathological conditions.actions. Inflammatory reactions occur in many pathological conditions of infectious and non-infectious aetiology; they may be very severe, leading to organs and tissue function disturbances. Therefore, it is obvious that, besides etiotropic therapy (if it is available), proper management of the inflammatory process is of great practical importance. Nowadays anti-inflammatory agents are the integral components of pharmacotherapy of quite a few diseases and pathological conditions.tions. Inflammatory reactions occur in many pathological conditions of infectious and non-infectious aetiology; they may be very severe, leading to organs and tissue function disturbances. Therefore, it is obvious that, besides etiotropic therapy (if it is available), proper management of the inflammatory process is of great practical importance. Nowadays anti-inflammatory agents are the integral components of pharmacotherapy of quite a few diseases and pathological conditions.s. Inflammatory reactions occur in many pathological conditions of infectious and non-infectious aetiology; they may be very severe, leading to organs and tissue function disturbances. Therefore, it is obvious that, besides etiotropic therapy (if it is available), proper management of the inflammatory process is of great practical importance. Nowadays anti-inflammatory agents are the integral components of pharmacotherapy of quite a few diseases and pathological conditions. Inflammatory reactions occur in many pathological conditions of infectious and non-infectious aetiology; they may be very severe, leading to organs and tissue function disturbances. Therefore, it is obvious that, besides etiotropic therapy (if it is available), proper management of the inflammatory process is of great practical importance. Nowadays anti-inflammatory agents are the integral components of pharmacotherapy of quite a few diseases and pathological conditions.mmatory reactions occur in many pathological conditions of infectious and non-infectious aetiology; they may be very severe, leading to organs and tissue function disturbances. Therefore, it is obvious that, besides etiotropic therapy (if it is available), proper management of the inflammatory process is of great practical importance. Nowadays anti-inflammatory agents are the integral components of pharmacotherapy of quite a few diseases and pathological conditions.atory reactions occur in many pathological conditions of infectious and non-infectious aetiology; they may be very severe, leading to organs and tissue function disturbances. Therefore, it is obvious that, besides etiotropic therapy (if it is available), proper management of the inflammatory process is of great practical importance. Nowadays anti-inflammatory agents are the integral components of pharmacotherapy of quite a few diseases and pathological conditions.ory reactions occur in many pathological conditions of infectious and non-infectious aetiology; they may be very severe, leading to organs and tissue function disturbances. Therefore, it is obvious that, besides etiotropic therapy (if it is available), proper management of the inflammatory process is of great practical importance. Nowadays anti-inflammatory agents are the integral components of pharmacotherapy of quite a few diseases and pathological conditions.ccur in many pathological conditions of infectious and non-infectious aetiology; they may be very severe, leading to organs and tissue function disturbances. Therefore, it is obvious that, besides etiotropic therapy (if it is available), proper management of the inflammatory process is of great practical importance. Nowadays anti-inflammatory agents are the integral components of pharmacotherapy of quite a few diseases and pathological conditions.ur in many pathological conditions of infectious and non-infectious aetiology; they may be very severe, leading to organs and tissue function disturbances. Therefore, it is obvious that, besides etiotropic therapy (if it is available), proper management of the inflammatory process is of great practical importance. Nowadays anti-inflammatory agents are the integral components of pharmacotherapy of quite a few diseases and pathological conditions. in many pathological conditions of infectious and non-infectious aetiology; they may be very severe, leading to organs and tissue function disturbances. Therefore, it is obvious that, besides etiotropic therapy (if it is available), proper management of the inflammatory process is of great practical importance. Nowadays anti-inflammatory agents are the integral components of pharmacotherapy of quite a few diseases and pathological conditions. many pathological conditions of infectious and non-infectious aetiology; they may be very severe, leading to organs and tissue function disturbances. Therefore, it is obvious that, besides etiotropic therapy (if it is available), proper management of the inflammatory process is of great practical importance. Nowadays anti-inflammatory agents are the integral components of pharmacotherapy of quite a few diseases and pathological conditions.any pathological conditions of infectious and non-infectious aetiology; they may be very severe, leading to organs and tissue function disturbances. Therefore, it is obvious that, besides etiotropic therapy (if it is available), proper management of the inflammatory process is of great practical importance. Nowadays anti-inflammatory agents are the integral components of pharmacotherapy of quite a few diseases and pathological conditions.y pathological conditions of infectious and non-infectious aetiology; they may be very severe, leading to organs and tissue function disturbances. Therefore, it is obvious that, besides etiotropic therapy (if it is available), proper management of the inflammatory process is of great practical importance. Nowadays anti-inflammatory agents are the integral components of pharmacotherapy of quite a few diseases and pathological conditions.nfectious and non-infectious aetiology; they may be very severe, leading to organs and tissue function disturbances. Therefore, it is obvious that, besides etiotropic therapy (if it is available), proper management of the inflammatory process is of great practical importance. Nowadays anti-inflammatory agents are the integral components of pharmacotherapy of quite a few diseases and pathological conditions.ectious and non-infectious aetiology; they may be very severe, leading to organs and tissue function disturbances. Therefore, it is obvious that, besides etiotropic therapy (if it is available), proper management of the inflammatory process is of great practical importance. Nowadays anti-inflammatory agents are the integral components of pharmacotherapy of quite a few diseases and pathological conditions.tious and non-infectious aetiology; they may be very severe, leading to organs and tissue function disturbances. Therefore, it is obvious that, besides etiotropic therapy (if it is available), proper management of the inflammatory process is of great practical importance. Nowadays anti-inflammatory agents are the integral components of pharmacotherapy of quite a few diseases and pathological conditions.ous and non-infectious aetiology; they may be very severe, leading to organs and tissue function disturbances. Therefore, it is obvious that, besides etiotropic therapy (if it is available), proper management of the inflammatory process is of great practical importance. Nowadays anti-inflammatory agents are the integral components of pharmacotherapy of quite a few diseases and pathological conditions.s and non-infectious aetiology; they may be very severe, leading to organs and tissue function disturbances. Therefore, it is obvious that, besides etiotropic therapy (if it is available), proper management of the inflammatory process is of great practical importance. Nowadays anti-inflammatory agents are the integral components of pharmacotherapy of quite a few diseases and pathological conditions.and non-infectious aetiology; they may be very severe, leading to organs and tissue function disturbances. Therefore, it is obvious that, besides etiotropic therapy (if it is available), proper management of the inflammatory process is of great practical importance. Nowadays anti-inflammatory agents are the integral components of pharmacotherapy of quite a few diseases and pathological conditions.on-infectious aetiology; they may be very severe, leading to organs and tissue function disturbances. Therefore, it is obvious that, besides etiotropic therapy (if it is available), proper management of the inflammatory process is of great practical importance. Nowadays anti-inflammatory agents are the integral components of pharmacotherapy of quite a few diseases and pathological conditions.-infectious aetiology; they may be very severe, leading to organs and tissue function disturbances. Therefore, it is obvious that, besides etiotropic therapy (if it is available), proper management of the inflammatory process is of great practical importance. Nowadays anti-inflammatory agents are the integral components of pharmacotherapy of quite a few diseases and pathological conditions.nfectious aetiology; they may be very severe, leading to organs and tissue function disturbances. Therefore, it is obvious that, besides etiotropic therapy (if it is available), proper management of the inflammatory process is of great practical importance. Nowadays anti-inflammatory agents are the integral components of pharmacotherapy of quite a few diseases and pathological conditions. tissue function disturbances. Therefore, it is obvious that, besides etiotropic therapy (if it is available), proper management of the inflammatory process is of great practical importance. Nowadays anti-inflammatory agents are the integral components of pharmacotherapy of quite a few diseases and pathological conditions.issue function disturbances. Therefore, it is obvious that, besides etiotropic therapy (if it is available), proper management of the inflammatory process is of great practical importance. Nowadays anti-inflammatory agents are the integral components of pharmacotherapy of quite a few diseases and pathological conditions.sue function disturbances. Therefore, it is obvious that, besides etiotropic therapy (if it is available), proper management of the inflammatory process is of great practical importance. Nowadays anti-inflammatory agents are the integral components of pharmacotherapy of quite a few diseases and pathological conditions.nction disturbances. Therefore, it is obvious that, besides etiotropic therapy (if it is available), proper management of the inflammatory process is of great practical importance. Nowadays anti-inflammatory agents are the integral components of pharmacotherapy of quite a few diseases and pathological conditions.tion disturbances. Therefore, it is obvious that, besides etiotropic therapy (if it is available), proper management of the inflammatory process is of great practical importance. Nowadays anti-inflammatory agents are the integral components of pharmacotherapy of quite a few diseases and pathological conditions.on disturbances. Therefore, it is obvious that, besides etiotropic therapy (if it is available), proper management of the inflammatory process is of great practical importance. Nowadays anti-inflammatory agents are the integral components of pharmacotherapy of quite a few diseases and pathological conditions.Therefore, it is obvious that, besides etiotropic therapy (if it is available), proper management of the inflammatory process is of great practical importance. Nowadays anti-inflammatory agents are the integral components of pharmacotherapy of quite a few diseases and pathological conditions.erefore, it is obvious that, besides etiotropic therapy (if it is available), proper management of the inflammatory process is of great practical importance. Nowadays anti-inflammatory agents are the integral components of pharmacotherapy of quite a few diseases and pathological conditions.efore, it is obvious that, besides etiotropic therapy (if it is available), proper management of the inflammatory process is of great practical importance. Nowadays anti-inflammatory agents are the integral components of pharmacotherapy of quite a few diseases and pathological conditions.therapy (if it is available), proper management of the inflammatory process is of great practical importance. Nowadays anti-inflammatory agents are the integral components of pharmacotherapy of quite a few diseases and pathological conditions.erapy (if it is available), proper management of the inflammatory process is of great practical importance. Nowadays anti-inflammatory agents are the integral components of pharmacotherapy of quite a few diseases and pathological conditions.apy (if it is available), proper management of the inflammatory process is of great practical importance. Nowadays anti-inflammatory agents are the integral components of pharmacotherapy of quite a few diseases and pathological conditions.y (if it is available), proper management of the inflammatory process is of great practical importance. Nowadays anti-inflammatory agents are the integral components of pharmacotherapy of quite a few diseases and pathological conditions.(if it is available), proper management of the inflammatory process is of great practical importance. Nowadays anti-inflammatory agents are the integral components of pharmacotherapy of quite a few diseases and pathological conditions.f it is available), proper management of the inflammatory process is of great practical importance. Nowadays anti-inflammatory agents are the integral components of pharmacotherapy of quite a few diseases and pathological conditions.it is available), proper management of the inflammatory process is of great practical importance. Nowadays anti-inflammatory agents are the integral components of pharmacotherapy of quite a few diseases and pathological conditions.s available), proper management of the inflammatory process is of great practical importance. Nowadays anti-inflammatory agents are the integral components of pharmacotherapy of quite a few diseases and pathological conditions.available), proper management of the inflammatory process is of great practical importance. Nowadays anti-inflammatory agents are the integral components of pharmacotherapy of quite a few diseases and pathological conditions.ailable), proper management of the inflammatory process is of great practical importance. Nowadays anti-inflammatory agents are the integral components of pharmacotherapy of quite a few diseases and pathological conditions.ble), proper management of the inflammatory process is of great practical importance. Nowadays anti-inflammatory agents are the integral components of pharmacotherapy of quite a few diseases and pathological conditions.e), proper management of the inflammatory process is of great practical importance. Nowadays anti-inflammatory agents are the integral components of pharmacotherapy of quite a few diseases and pathological conditions.oper management of the inflammatory process is of great practical importance. Nowadays anti-inflammatory agents are the integral components of pharmacotherapy of quite a few diseases and pathological conditions.er management of the inflammatory process is of great practical importance. Nowadays anti-inflammatory agents are the integral components of pharmacotherapy of quite a few diseases and pathological conditions. management of the inflammatory process is of great practical importance. Nowadays anti-inflammatory agents are the integral components of pharmacotherapy of quite a few diseases and pathological conditions.the inflammatory process is of great practical importance. Nowadays anti-inflammatory agents are the integral components of pharmacotherapy of quite a few diseases and pathological conditions.e inflammatory process is of great practical importance. Nowadays anti-inflammatory agents are the integral components of pharmacotherapy of quite a few diseases and pathological conditions.inflammatory process is of great practical importance. Nowadays anti-inflammatory agents are the integral components of pharmacotherapy of quite a few diseases and pathological conditions.lammatory process is of great practical importance. Nowadays anti-inflammatory agents are the integral components of pharmacotherapy of quite a few diseases and pathological conditions.mmatory process is of great practical importance. Nowadays anti-inflammatory agents are the integral components of pharmacotherapy of quite a few diseases and pathological conditions.atory process is of great practical importance. Nowadays anti-inflammatory agents are the integral components of pharmacotherapy of quite a few diseases and pathological conditions.tical importance. Nowadays anti-inflammatory agents are the integral components of pharmacotherapy of quite a few diseases and pathological conditions.cal importance. Nowadays anti-inflammatory agents are the integral components of pharmacotherapy of quite a few diseases and pathological conditions.l importance. Nowadays anti-inflammatory agents are the integral components of pharmacotherapy of quite a few diseases and pathological conditions.importance. Nowadays anti-inflammatory agents are the integral components of pharmacotherapy of quite a few diseases and pathological conditions.portance. Nowadays anti-inflammatory agents are the integral components of pharmacotherapy of quite a few diseases and pathological conditions.rtance. Nowadays anti-inflammatory agents are the integral components of pharmacotherapy of quite a few diseases and pathological conditions.e. Nowadays anti-inflammatory agents are the integral components of pharmacotherapy of quite a few diseases and pathological conditions. Nowadays anti-inflammatory agents are the integral components of pharmacotherapy of quite a few diseases and pathological conditions.owadays anti-inflammatory agents are the integral components of pharmacotherapy of quite a few diseases and pathological conditions.rmacotherapy of quite a few diseases and pathological conditions.acotherapy of quite a few diseases and pathological conditions.otherapy of quite a few diseases and pathological conditions.py of quite a few diseases and pathological conditions. of quite a few diseases and pathological conditions.f quite a few diseases and pathological conditions.eases and pathological conditions.ses and pathological conditions.s and pathological conditions.classasss="txtt>Inflammation is a complex process regulated by a number of endogenous substances. Endogenous substances are produced by various cells participating in inflammation (mast cells, polymorphonuclear leukocytes, monocytes/macrophages, endothelial cells, platelets). They secrete such biologically active substances as prostanoids, leukotrienes, NO, platelet activating factor (PAF¹), histamine, some interleukins, etc.² Therefore, there are many ways to regulate inflammation pharmacologically. Usually they are aimed at the suppression of the production and release of the substances stimulating the inflammatory process.pan class="">Inflammation is a complex process regulated by a number of endogenous substances. Endogenous substances are produced by various cells participating in inflammation (mast cells, polymorphonuclear leukocytes, monocytes/macrophages, endothelial cells, platelets). They secrete such biologically active substances as prostanoids, leukotrienes, NO, platelet activating factor (PAF¹), histamine, some interleukins, etc.² Therefore, there are many ways to regulate inflammation pharmacologically. Usually they are aimed at the suppression of the production and release of the substances stimulating the inflammatory process.class="">Inflammation is a complex process regulated by a number of endogenous substances. Endogenous substances are produced by various cells participating in inflammation (mast cells, polymorphonuclear leukocytes, monocytes/macrophages, endothelial cells, platelets). They secrete such biologically active substances as prostanoids, leukotrienes, NO, platelet activating factor (PAF¹), histamine, some interleukins, etc.² Therefore, there are many ways to regulate inflammation pharmacologically. Usually they are aimed at the suppression of the production and release of the substances stimulating the inflammatory process.ass="">Inflammation is a complex process regulated by a number of endogenous substances. Endogenous substances are produced by various cells participating in inflammation (mast cells, polymorphonuclear leukocytes, monocytes/macrophages, endothelial cells, platelets). They secrete such biologically active substances as prostanoids, leukotrienes, NO, platelet activating factor (PAF¹), histamine, some interleukins, etc.² Therefore, there are many ways to regulate inflammation pharmacologically. Usually they are aimed at the suppression of the production and release of the substances stimulating the inflammatory process.>Inflammation is a complex process regulated by a number of endogenous substances. Endogenous substances are produced by various cells participating in inflammation (mast cells, polymorphonuclear leukocytes, monocytes/macrophages, endothelial cells, platelets). They secrete such biologically active substances as prostanoids, leukotrienes, NO, platelet activating factor (PAF¹), histamine, some interleukins, etc.² Therefore, there are many ways to regulate inflammation pharmacologically. Usually they are aimed at the suppression of the production and release of the substances stimulating the inflammatory process.nflammation is a complex process regulated by a number of endogenous substances. Endogenous substances are produced by various cells participating in inflammation (mast cells, polymorphonuclear leukocytes, monocytes/macrophages, endothelial cells, platelets). They secrete such biologically active substances as prostanoids, leukotrienes, NO, platelet activating factor (PAF¹), histamine, some interleukins, etc.² Therefore, there are many ways to regulate inflammation pharmacologically. Usually they are aimed at the suppression of the production and release of the substances stimulating the inflammatory process.lammation is a complex process regulated by a number of endogenous substances. Endogenous substances are produced by various cells participating in inflammation (mast cells, polymorphonuclear leukocytes, monocytes/macrophages, endothelial cells, platelets). They secrete such biologically active substances as prostanoids, leukotrienes, NO, platelet activating factor (PAF¹), histamine, some interleukins, etc.² Therefore, there are many ways to regulate inflammation pharmacologically. Usually they are aimed at the suppression of the production and release of the substances stimulating the inflammatory process.complex process regulated by a number of endogenous substances. Endogenous substances are produced by various cells participating in inflammation (mast cells, polymorphonuclear leukocytes, monocytes/macrophages, endothelial cells, platelets). They secrete such biologically active substances as prostanoids, leukotrienes, NO, platelet activating factor (PAF¹), histamine, some interleukins, etc.² Therefore, there are many ways to regulate inflammation pharmacologically. Usually they are aimed at the suppression of the production and release of the substances stimulating the inflammatory process.mplex process regulated by a number of endogenous substances. Endogenous substances are produced by various cells participating in inflammation (mast cells, polymorphonuclear leukocytes, monocytes/macrophages, endothelial cells, platelets). They secrete such biologically active substances as prostanoids, leukotrienes, NO, platelet activating factor (PAF¹), histamine, some interleukins, etc.² Therefore, there are many ways to regulate inflammation pharmacologically. Usually they are aimed at the suppression of the production and release of the substances stimulating the inflammatory process.lex process regulated by a number of endogenous substances. Endogenous substances are produced by various cells participating in inflammation (mast cells, polymorphonuclear leukocytes, monocytes/macrophages, endothelial cells, platelets). They secrete such biologically active substances as prostanoids, leukotrienes, NO, platelet activating factor (PAF¹), histamine, some interleukins, etc.² Therefore, there are many ways to regulate inflammation pharmacologically. Usually they are aimed at the suppression of the production and release of the substances stimulating the inflammatory process. process regulated by a number of endogenous substances. Endogenous substances are produced by various cells participating in inflammation (mast cells, polymorphonuclear leukocytes, monocytes/macrophages, endothelial cells, platelets). They secrete such biologically active substances as prostanoids, leukotrienes, NO, platelet activating factor (PAF¹), histamine, some interleukins, etc.² Therefore, there are many ways to regulate inflammation pharmacologically. Usually they are aimed at the suppression of the production and release of the substances stimulating the inflammatory process.rocess regulated by a number of endogenous substances. Endogenous substances are produced by various cells participating in inflammation (mast cells, polymorphonuclear leukocytes, monocytes/macrophages, endothelial cells, platelets). They secrete such biologically active substances as prostanoids, leukotrienes, NO, platelet activating factor (PAF¹), histamine, some interleukins, etc.² Therefore, there are many ways to regulate inflammation pharmacologically. Usually they are aimed at the suppression of the production and release of the substances stimulating the inflammatory process.cess regulated by a number of endogenous substances. Endogenous substances are produced by various cells participating in inflammation (mast cells, polymorphonuclear leukocytes, monocytes/macrophages, endothelial cells, platelets). They secrete such biologically active substances as prostanoids, leukotrienes, NO, platelet activating factor (PAF¹), histamine, some interleukins, etc.² Therefore, there are many ways to regulate inflammation pharmacologically. Usually they are aimed at the suppression of the production and release of the substances stimulating the inflammatory process.endogenous substances. Endogenous substances are produced by various cells participating in inflammation (mast cells, polymorphonuclear leukocytes, monocytes/macrophages, endothelial cells, platelets). They secrete such biologically active substances as prostanoids, leukotrienes, NO, platelet activating factor (PAF¹), histamine, some interleukins, etc.² Therefore, there are many ways to regulate inflammation pharmacologically. Usually they are aimed at the suppression of the production and release of the substances stimulating the inflammatory process.dogenous substances. Endogenous substances are produced by various cells participating in inflammation (mast cells, polymorphonuclear leukocytes, monocytes/macrophages, endothelial cells, platelets). They secrete such biologically active substances as prostanoids, leukotrienes, NO, platelet activating factor (PAF¹), histamine, some interleukins, etc.² Therefore, there are many ways to regulate inflammation pharmacologically. Usually they are aimed at the suppression of the production and release of the substances stimulating the inflammatory process.genous substances. Endogenous substances are produced by various cells participating in inflammation (mast cells, polymorphonuclear leukocytes, monocytes/macrophages, endothelial cells, platelets). They secrete such biologically active substances as prostanoids, leukotrienes, NO, platelet activating factor (PAF¹), histamine, some interleukins, etc.² Therefore, there are many ways to regulate inflammation pharmacologically. Usually they are aimed at the suppression of the production and release of the substances stimulating the inflammatory process.nous substances. Endogenous substances are produced by various cells participating in inflammation (mast cells, polymorphonuclear leukocytes, monocytes/macrophages, endothelial cells, platelets). They secrete such biologically active substances as prostanoids, leukotrienes, NO, platelet activating factor (PAF¹), histamine, some interleukins, etc.² Therefore, there are many ways to regulate inflammation pharmacologically. Usually they are aimed at the suppression of the production and release of the substances stimulating the inflammatory process.us substances. Endogenous substances are produced by various cells participating in inflammation (mast cells, polymorphonuclear leukocytes, monocytes/macrophages, endothelial cells, platelets). They secrete such biologically active substances as prostanoids, leukotrienes, NO, platelet activating factor (PAF¹), histamine, some interleukins, etc.² Therefore, there are many ways to regulate inflammation pharmacologically. Usually they are aimed at the suppression of the production and release of the substances stimulating the inflammatory process. substances. Endogenous substances are produced by various cells participating in inflammation (mast cells, polymorphonuclear leukocytes, monocytes/macrophages, endothelial cells, platelets). They secrete such biologically active substances as prostanoids, leukotrienes, NO, platelet activating factor (PAF¹), histamine, some interleukins, etc.² Therefore, there are many ways to regulate inflammation pharmacologically. Usually they are aimed at the suppression of the production and release of the substances stimulating the inflammatory process.tances. Endogenous substances are produced by various cells participating in inflammation (mast cells, polymorphonuclear leukocytes, monocytes/macrophages, endothelial cells, platelets). They secrete such biologically active substances as prostanoids, leukotrienes, NO, platelet activating factor (PAF¹), histamine, some interleukins, etc.² Therefore, there are many ways to regulate inflammation pharmacologically. Usually they are aimed at the suppression of the production and release of the substances stimulating the inflammatory process.nces. Endogenous substances are produced by various cells participating in inflammation (mast cells, polymorphonuclear leukocytes, monocytes/macrophages, endothelial cells, platelets). They secrete such biologically active substances as prostanoids, leukotrienes, NO, platelet activating factor (PAF¹), histamine, some interleukins, etc.² Therefore, there are many ways to regulate inflammation pharmacologically. Usually they are aimed at the suppression of the production and release of the substances stimulating the inflammatory process.es. Endogenous substances are produced by various cells participating in inflammation (mast cells, polymorphonuclear leukocytes, monocytes/macrophages, endothelial cells, platelets). They secrete such biologically active substances as prostanoids, leukotrienes, NO, platelet activating factor (PAF¹), histamine, some interleukins, etc.² Therefore, there are many ways to regulate inflammation pharmacologically. Usually they are aimed at the suppression of the production and release of the substances stimulating the inflammatory process.ng in inflammation (mast cells, polymorphonuclear leukocytes, monocytes/macrophages, endothelial cells, platelets). They secrete such biologically active substances as prostanoids, leukotrienes, NO, platelet activating factor (PAF¹), histamine, some interleukins, etc.² Therefore, there are many ways to regulate inflammation pharmacologically. Usually they are aimed at the suppression of the production and release of the substances stimulating the inflammatory process. in inflammation (mast cells, polymorphonuclear leukocytes, monocytes/macrophages, endothelial cells, platelets). They secrete such biologically active substances as prostanoids, leukotrienes, NO, platelet activating factor (PAF¹), histamine, some interleukins, etc.² Therefore, there are many ways to regulate inflammation pharmacologically. Usually they are aimed at the suppression of the production and release of the substances stimulating the inflammatory process.n inflammation (mast cells, polymorphonuclear leukocytes, monocytes/macrophages, endothelial cells, platelets). They secrete such biologically active substances as prostanoids, leukotrienes, NO, platelet activating factor (PAF¹), histamine, some interleukins, etc.² Therefore, there are many ways to regulate inflammation pharmacologically. Usually they are aimed at the suppression of the production and release of the substances stimulating the inflammatory process.ammation (mast cells, polymorphonuclear leukocytes, monocytes/macrophages, endothelial cells, platelets). They secrete such biologically active substances as prostanoids, leukotrienes, NO, platelet activating factor (PAF¹), histamine, some interleukins, etc.² Therefore, there are many ways to regulate inflammation pharmacologically. Usually they are aimed at the suppression of the production and release of the substances stimulating the inflammatory process.mation (mast cells, polymorphonuclear leukocytes, monocytes/macrophages, endothelial cells, platelets). They secrete such biologically active substances as prostanoids, leukotrienes, NO, platelet activating factor (PAF¹), histamine, some interleukins, etc.² Therefore, there are many ways to regulate inflammation pharmacologically. Usually they are aimed at the suppression of the production and release of the substances stimulating the inflammatory process.tion (mast cells, polymorphonuclear leukocytes, monocytes/macrophages, endothelial cells, platelets). They secrete such biologically active substances as prostanoids, leukotrienes, NO, platelet activating factor (PAF¹), histamine, some interleukins, etc.² Therefore, there are many ways to regulate inflammation pharmacologically. Usually they are aimed at the suppression of the production and release of the substances stimulating the inflammatory process. polymorphonuclear leukocytes, monocytes/macrophages, endothelial cells, platelets). They secrete such biologically active substances as prostanoids, leukotrienes, NO, platelet activating factor (PAF¹), histamine, some interleukins, etc.² Therefore, there are many ways to regulate inflammation pharmacologically. Usually they are aimed at the suppression of the production and release of the substances stimulating the inflammatory process.olymorphonuclear leukocytes, monocytes/macrophages, endothelial cells, platelets). They secrete such biologically active substances as prostanoids, leukotrienes, NO, platelet activating factor (PAF¹), histamine, some interleukins, etc.² Therefore, there are many ways to regulate inflammation pharmacologically. Usually they are aimed at the suppression of the production and release of the substances stimulating the inflammatory process.ymorphonuclear leukocytes, monocytes/macrophages, endothelial cells, platelets). They secrete such biologically active substances as prostanoids, leukotrienes, NO, platelet activating factor (PAF¹), histamine, some interleukins, etc.² Therefore, there are many ways to regulate inflammation pharmacologically. Usually they are aimed at the suppression of the production and release of the substances stimulating the inflammatory process.ytes, monocytes/macrophages, endothelial cells, platelets). They secrete such biologically active substances as prostanoids, leukotrienes, NO, platelet activating factor (PAF¹), histamine, some interleukins, etc.² Therefore, there are many ways to regulate inflammation pharmacologically. Usually they are aimed at the suppression of the production and release of the substances stimulating the inflammatory process.es, monocytes/macrophages, endothelial cells, platelets). They secrete such biologically active substances as prostanoids, leukotrienes, NO, platelet activating factor (PAF¹), histamine, some interleukins, etc.² Therefore, there are many ways to regulate inflammation pharmacologically. Usually they are aimed at the suppression of the production and release of the substances stimulating the inflammatory process., monocytes/macrophages, endothelial cells, platelets). They secrete such biologically active substances as prostanoids, leukotrienes, NO, platelet activating factor (PAF¹), histamine, some interleukins, etc.² Therefore, there are many ways to regulate inflammation pharmacologically. Usually they are aimed at the suppression of the production and release of the substances stimulating the inflammatory process.monocytes/macrophages, endothelial cells, platelets). They secrete such biologically active substances as prostanoids, leukotrienes, NO, platelet activating factor (PAF¹), histamine, some interleukins, etc.² Therefore, there are many ways to regulate inflammation pharmacologically. Usually they are aimed at the suppression of the production and release of the substances stimulating the inflammatory process.nocytes/macrophages, endothelial cells, platelets). They secrete such biologically active substances as prostanoids, leukotrienes, NO, platelet activating factor (PAF¹), histamine, some interleukins, etc.² Therefore, there are many ways to regulate inflammation pharmacologically. Usually they are aimed at the suppression of the production and release of the substances stimulating the inflammatory process.cytes/macrophages, endothelial cells, platelets). They secrete such biologically active substances as prostanoids, leukotrienes, NO, platelet activating factor (PAF¹), histamine, some interleukins, etc.² Therefore, there are many ways to regulate inflammation pharmacologically. Usually they are aimed at the suppression of the production and release of the substances stimulating the inflammatory process.tes/macrophages, endothelial cells, platelets). They secrete such biologically active substances as prostanoids, leukotrienes, NO, platelet activating factor (PAF¹), histamine, some interleukins, etc.² Therefore, there are many ways to regulate inflammation pharmacologically. Usually they are aimed at the suppression of the production and release of the substances stimulating the inflammatory process.macrophages, endothelial cells, platelets). They secrete such biologically active substances as prostanoids, leukotrienes, NO, platelet activating factor (PAF¹), histamine, some interleukins, etc.² Therefore, there are many ways to regulate inflammation pharmacologically. Usually they are aimed at the suppression of the production and release of the substances stimulating the inflammatory process.crophages, endothelial cells, platelets). They secrete such biologically active substances as prostanoids, leukotrienes, NO, platelet activating factor (PAF¹), histamine, some interleukins, etc.² Therefore, there are many ways to regulate inflammation pharmacologically. Usually they are aimed at the suppression of the production and release of the substances stimulating the inflammatory process.ophages, endothelial cells, platelets). 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They secrete such biologically active substances as prostanoids, leukotrienes, NO, platelet activating factor (PAF¹), histamine, some interleukins, etc.² Therefore, there are many ways to regulate inflammation pharmacologically. Usually they are aimed at the suppression of the production and release of the substances stimulating the inflammatory process.othelial cells, platelets). They secrete such biologically active substances as prostanoids, leukotrienes, NO, platelet activating factor (PAF¹), histamine, some interleukins, etc.² Therefore, there are many ways to regulate inflammation pharmacologically. Usually they are aimed at the suppression of the production and release of the substances stimulating the inflammatory process.helial cells, platelets). They secrete such biologically active substances as prostanoids, leukotrienes, NO, platelet activating factor (PAF¹), histamine, some interleukins, etc.² Therefore, there are many ways to regulate inflammation pharmacologically. 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They secrete such biologically active substances as prostanoids, leukotrienes, NO, platelet activating factor (PAF¹), histamine, some interleukins, etc.² Therefore, there are many ways to regulate inflammation pharmacologically. Usually they are aimed at the suppression of the production and release of the substances stimulating the inflammatory process.ts). They secrete such biologically active substances as prostanoids, leukotrienes, NO, platelet activating factor (PAF¹), histamine, some interleukins, etc.² Therefore, there are many ways to regulate inflammation pharmacologically. Usually they are aimed at the suppression of the production and release of the substances stimulating the inflammatory process.. They secrete such biologically active substances as prostanoids, leukotrienes, NO, platelet activating factor (PAF¹), histamine, some interleukins, etc.² Therefore, there are many ways to regulate inflammation pharmacologically. Usually they are aimed at the suppression of the production and release of the substances stimulating the inflammatory process.They secrete such biologically active substances as prostanoids, leukotrienes, NO, platelet activating factor (PAF¹), histamine, some interleukins, etc.² Therefore, there are many ways to regulate inflammation pharmacologically. Usually they are aimed at the suppression of the production and release of the substances stimulating the inflammatory process.ey secrete such biologically active substances as prostanoids, leukotrienes, NO, platelet activating factor (PAF¹), histamine, some interleukins, etc.² Therefore, there are many ways to regulate inflammation pharmacologically. 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process deserve specialhe inflammatory process deserve speciale specialspecialecialiall/span>> Biologically active lipid. It is formed under the influence of phospholipase A₂ from phospholipids in many cells participating in the inflammatory process (in platelets, neutrophils, monocytes, mast cells, eosinophils, in the kidneys and endothelium). It is related to inflammation mediators. It also participates in the development of allergic reactions. It has a vasodilating effect, increases vascular permeability, causes leukocyte chemotaxis, activates leukocytes, enhances platelet aggregation and causes smooth muscle contraction.n> Biologically active lipid. It is formed under the influence of phospholipase A₂ from phospholipids in many cells participating in the inflammatory process (in platelets, neutrophils, monocytes, mast cells, eosinophils, in the kidneys and endothelium). It is related to inflammation mediators. It also participates in the development of allergic reactions. It has a vasodilating effect, increases vascular permeability, causes leukocyte chemotaxis, activates leukocytes, enhances platelet aggregation and causes smooth muscle contraction. Biologically active lipid. It is formed under the influence of phospholipase A₂ from phospholipids in many cells participating in the inflammatory process (in platelets, neutrophils, monocytes, mast cells, eosinophils, in the kidneys and endothelium). It is related to inflammation mediators. It also participates in the development of allergic reactions. It has a vasodilating effect, increases vascular permeability, causes leukocyte chemotaxis, activates leukocytes, enhances platelet aggregation and causes smooth muscle contraction.class=""> Biologically active lipid. It is formed under the influence of phospholipase A₂ from phospholipids in many cells participating in the inflammatory process (in platelets, neutrophils, monocytes, mast cells, eosinophils, in the kidneys and endothelium). It is related to inflammation mediators. It also participates in the development of allergic reactions. It has a vasodilating effect, increases vascular permeability, causes leukocyte chemotaxis, activates leukocytes, enhances platelet aggregation and causes smooth muscle contraction.ass=""> Biologically active lipid. It is formed under the influence of phospholipase A₂ from phospholipids in many cells participating in the inflammatory process (in platelets, neutrophils, monocytes, mast cells, eosinophils, in the kidneys and endothelium). It is related to inflammation mediators. It also participates in the development of allergic reactions. It has a vasodilating effect, increases vascular permeability, causes leukocyte chemotaxis, activates leukocytes, enhances platelet aggregation and causes smooth muscle contraction.s=""> Biologically active lipid. It is formed under the influence of phospholipase A₂ from phospholipids in many cells participating in the inflammatory process (in platelets, neutrophils, monocytes, mast cells, eosinophils, in the kidneys and endothelium). It is related to inflammation mediators. It also participates in the development of allergic reactions. It has a vasodilating effect, increases vascular permeability, causes leukocyte chemotaxis, activates leukocytes, enhances platelet aggregation and causes smooth muscle contraction.Biologically active lipid. It is formed under the influence of phospholipase A₂ from phospholipids in many cells participating in the inflammatory process (in platelets, neutrophils, monocytes, mast cells, eosinophils, in the kidneys and endothelium). It is related to inflammation mediators. It also participates in the development of allergic reactions. It has a vasodilating effect, increases vascular permeability, causes leukocyte chemotaxis, activates leukocytes, enhances platelet aggregation and causes smooth muscle contraction.span>Biologically active lipid. It is formed under the influence of phospholipase A₂ from phospholipids in many cells participating in the inflammatory process (in platelets, neutrophils, monocytes, mast cells, eosinophils, in the kidneys and endothelium). It is related to inflammation mediators. It also participates in the development of allergic reactions. It has a vasodilating effect, increases vascular permeability, causes leukocyte chemotaxis, activates leukocytes, enhances platelet aggregation and causes smooth muscle contraction.an>Biologically active lipid. It is formed under the influence of phospholipase A₂ from phospholipids in many cells participating in the inflammatory process (in platelets, neutrophils, monocytes, mast cells, eosinophils, in the kidneys and endothelium). It is related to inflammation mediators. It also participates in the development of allergic reactions. It has a vasodilating effect, increases vascular permeability, causes leukocyte chemotaxis, activates leukocytes, enhances platelet aggregation and causes smooth muscle contraction.under the influence of phospholipase A₂ from phospholipids in many cells participating in the inflammatory process (in platelets, neutrophils, monocytes, mast cells, eosinophils, in the kidneys and endothelium). It is related to inflammation mediators. It also participates in the development of allergic reactions. It has a vasodilating effect, increases vascular permeability, causes leukocyte chemotaxis, activates leukocytes, enhances platelet aggregation and causes smooth muscle contraction.der the influence of phospholipase A₂ from phospholipids in many cells participating in the inflammatory process (in platelets, neutrophils, monocytes, mast cells, eosinophils, in the kidneys and endothelium). It is related to inflammation mediators. It also participates in the development of allergic reactions. It has a vasodilating effect, increases vascular permeability, causes leukocyte chemotaxis, activates leukocytes, enhances platelet aggregation and causes smooth muscle contraction.r the influence of phospholipase A₂ from phospholipids in many cells participating in the inflammatory process (in platelets, neutrophils, monocytes, mast cells, eosinophils, in the kidneys and endothelium). It is related to inflammation mediators. It also participates in the development of allergic reactions. It has a vasodilating effect, increases vascular permeability, causes leukocyte chemotaxis, activates leukocytes, enhances platelet aggregation and causes smooth muscle contraction.the influence of phospholipase A₂ from phospholipids in many cells participating in the inflammatory process (in platelets, neutrophils, monocytes, mast cells, eosinophils, in the kidneys and endothelium). It is related to inflammation mediators. It also participates in the development of allergic reactions. It has a vasodilating effect, increases vascular permeability, causes leukocyte chemotaxis, activates leukocytes, enhances platelet aggregation and causes smooth muscle contraction.e influence of phospholipase A₂ from phospholipids in many cells participating in the inflammatory process (in platelets, neutrophils, monocytes, mast cells, eosinophils, in the kidneys and endothelium). It is related to inflammation mediators. It also participates in the development of allergic reactions. It has a vasodilating effect, increases vascular permeability, causes leukocyte chemotaxis, activates leukocytes, enhances platelet aggregation and causes smooth muscle contraction.influence of phospholipase A₂ from phospholipids in many cells participating in the inflammatory process (in platelets, neutrophils, monocytes, mast cells, eosinophils, in the kidneys and endothelium). It is related to inflammation mediators. It also participates in the development of allergic reactions. It has a vasodilating effect, increases vascular permeability, causes leukocyte chemotaxis, activates leukocytes, enhances platelet aggregation and causes smooth muscle contraction.fluence of phospholipase A₂ from phospholipids in many cells participating in the inflammatory process (in platelets, neutrophils, monocytes, mast cells, eosinophils, in the kidneys and endothelium). It is related to inflammation mediators. It also participates in the development of allergic reactions. It has a vasodilating effect, increases vascular permeability, causes leukocyte chemotaxis, activates leukocytes, enhances platelet aggregation and causes smooth muscle contraction.nce of phospholipase A₂ from phospholipids in many cells participating in the inflammatory process (in platelets, neutrophils, monocytes, mast cells, eosinophils, in the kidneys and endothelium). It is related to inflammation mediators. It also participates in the development of allergic reactions. It has a vasodilating effect, increases vascular permeability, causes leukocyte chemotaxis, activates leukocytes, enhances platelet aggregation and causes smooth muscle contraction.e of phospholipase A₂ from phospholipids in many cells participating in the inflammatory process (in platelets, neutrophils, monocytes, mast cells, eosinophils, in the kidneys and endothelium). It is related to inflammation mediators. It also participates in the development of allergic reactions. It has a vasodilating effect, increases vascular permeability, causes leukocyte chemotaxis, activates leukocytes, enhances platelet aggregation and causes smooth muscle contraction.of phospholipase A₂ from phospholipids in many cells participating in the inflammatory process (in platelets, neutrophils, monocytes, mast cells, eosinophils, in the kidneys and endothelium). It is related to inflammation mediators. It also participates in the development of allergic reactions. It has a vasodilating effect, increases vascular permeability, causes leukocyte chemotaxis, activates leukocytes, enhances platelet aggregation and causes smooth muscle contraction.hospholipase A₂ from phospholipids in many cells participating in the inflammatory process (in platelets, neutrophils, monocytes, mast cells, eosinophils, in the kidneys and endothelium). It is related to inflammation mediators. It also participates in the development of allergic reactions. It has a vasodilating effect, increases vascular permeability, causes leukocyte chemotaxis, activates leukocytes, enhances platelet aggregation and causes smooth muscle contraction.spholipase A₂ from phospholipids in many cells participating in the inflammatory process (in platelets, neutrophils, monocytes, mast cells, eosinophils, in the kidneys and endothelium). It is related to inflammation mediators. It also participates in the development of allergic reactions. It has a vasodilating effect, increases vascular permeability, causes leukocyte chemotaxis, activates leukocytes, enhances platelet aggregation and causes smooth muscle contraction.pase A₂ from phospholipids in many cells participating in the inflammatory process (in platelets, neutrophils, monocytes, mast cells, eosinophils, in the kidneys and endothelium). It is related to inflammation mediators. It also participates in the development of allergic reactions. It has a vasodilating effect, increases vascular permeability, causes leukocyte chemotaxis, activates leukocytes, enhances platelet aggregation and causes smooth muscle contraction.se A₂ from phospholipids in many cells participating in the inflammatory process (in platelets, neutrophils, monocytes, mast cells, eosinophils, in the kidneys and endothelium). It is related to inflammation mediators. It also participates in the development of allergic reactions. It has a vasodilating effect, increases vascular permeability, causes leukocyte chemotaxis, activates leukocytes, enhances platelet aggregation and causes smooth muscle contraction. A₂ from phospholipids in many cells participating in the inflammatory process (in platelets, neutrophils, monocytes, mast cells, eosinophils, in the kidneys and endothelium). It is related to inflammation mediators. It also participates in the development of allergic reactions. It has a vasodilating effect, increases vascular permeability, causes leukocyte chemotaxis, activates leukocytes, enhances platelet aggregation and causes smooth muscle contraction.from phospholipids in many cells participating in the inflammatory process (in platelets, neutrophils, monocytes, mast cells, eosinophils, in the kidneys and endothelium). It is related to inflammation mediators. It also participates in the development of allergic reactions. It has a vasodilating effect, increases vascular permeability, causes leukocyte chemotaxis, activates leukocytes, enhances platelet aggregation and causes smooth muscle contraction.om phospholipids in many cells participating in the inflammatory process (in platelets, neutrophils, monocytes, mast cells, eosinophils, in the kidneys and endothelium). It is related to inflammation mediators. It also participates in the development of allergic reactions. It has a vasodilating effect, increases vascular permeability, causes leukocyte chemotaxis, activates leukocytes, enhances platelet aggregation and causes smooth muscle contraction. phospholipids in many cells participating in the inflammatory process (in platelets, neutrophils, monocytes, mast cells, eosinophils, in the kidneys and endothelium). It is related to inflammation mediators. It also participates in the development of allergic reactions. 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It has a vasodilating effect, increases vascular permeability, causes leukocyte chemotaxis, activates leukocytes, enhances platelet aggregation and causes smooth muscle contraction.ammatory process (in platelets, neutrophils, monocytes, mast cells, eosinophils, in the kidneys and endothelium). It is related to inflammation mediators. It also participates in the development of allergic reactions. It has a vasodilating effect, increases vascular permeability, causes leukocyte chemotaxis, activates leukocytes, enhances platelet aggregation and causes smooth muscle contraction.matory process (in platelets, neutrophils, monocytes, mast cells, eosinophils, in the kidneys and endothelium). It is related to inflammation mediators. It also participates in the development of allergic reactions. 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Table of contents

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v class="table-of-contents">class="table-of-contents">ass="table-of-contents">ontents">tents">nts">">-bkt1kt11"> INTRODUCTION. 1. SUBJECT AND AIMS OF PHARMACOLOGY. ITS POSITION AMONG OTHER MEDICAL DISCIPLINES. MAIN STAGES OF PHARMACOLOGY DEVELOPMENTNTRODUCTION. 1. SUBJECT AND AIMS OF PHARMACOLOGY. ITS POSITION AMONG OTHER MEDICAL DISCIPLINES. MAIN STAGES OF PHARMACOLOGY DEVELOPMENTCTION. 1. SUBJECT AND AIMS OF PHARMACOLOGY. ITS POSITION AMONG OTHER MEDICAL DISCIPLINES. MAIN STAGES OF PHARMACOLOGY DEVELOPMENTION. 1. SUBJECT AND AIMS OF PHARMACOLOGY. ITS POSITION AMONG OTHER MEDICAL DISCIPLINES. MAIN STAGES OF PHARMACOLOGY DEVELOPMENTN. 1. SUBJECT AND AIMS OF PHARMACOLOGY. ITS POSITION AMONG OTHER MEDICAL DISCIPLINES. MAIN STAGES OF PHARMACOLOGY DEVELOPMENTND AIMS OF PHARMACOLOGY. ITS POSITION AMONG OTHER MEDICAL DISCIPLINES. MAIN STAGES OF PHARMACOLOGY DEVELOPMENT AIMS OF PHARMACOLOGY. ITS POSITION AMONG OTHER MEDICAL DISCIPLINES. MAIN STAGES OF PHARMACOLOGY DEVELOPMENTIMS OF PHARMACOLOGY. ITS POSITION AMONG OTHER MEDICAL DISCIPLINES. MAIN STAGES OF PHARMACOLOGY DEVELOPMENT OF PHARMACOLOGY. ITS POSITION AMONG OTHER MEDICAL DISCIPLINES. MAIN STAGES OF PHARMACOLOGY DEVELOPMENTF PHARMACOLOGY. ITS POSITION AMONG OTHER MEDICAL DISCIPLINES. MAIN STAGES OF PHARMACOLOGY DEVELOPMENTPHARMACOLOGY. ITS POSITION AMONG OTHER MEDICAL DISCIPLINES. MAIN STAGES OF PHARMACOLOGY DEVELOPMENTNG OTHER MEDICAL DISCIPLINES. MAIN STAGES OF PHARMACOLOGY DEVELOPMENT OTHER MEDICAL DISCIPLINES. MAIN STAGES OF PHARMACOLOGY DEVELOPMENTTHER MEDICAL DISCIPLINES. MAIN STAGES OF PHARMACOLOGY DEVELOPMENTER MEDICAL DISCIPLINES. MAIN STAGES OF PHARMACOLOGY DEVELOPMENT MEDICAL DISCIPLINES. MAIN STAGES OF PHARMACOLOGY DEVELOPMENTEDICAL DISCIPLINES. MAIN STAGES OF PHARMACOLOGY DEVELOPMENTL DISCIPLINES. MAIN STAGES OF PHARMACOLOGY DEVELOPMENTDISCIPLINES. MAIN STAGES OF PHARMACOLOGY DEVELOPMENTSCIPLINES. MAIN STAGES OF PHARMACOLOGY DEVELOPMENT>ERISTICS OF THE HUMAN BODY AND ITS STATE FOR THE MANIFESTATION OF THE DRUG EFFECTISTICS OF THE HUMAN BODY AND ITS STATE FOR THE MANIFESTATION OF THE DRUG EFFECTTICS OF THE HUMAN BODY AND ITS STATE FOR THE MANIFESTATION OF THE DRUG EFFECTCS OF THE HUMAN BODY AND ITS STATE FOR THE MANIFESTATION OF THE DRUG EFFECT OF THE HUMAN BODY AND ITS STATE FOR THE MANIFESTATION OF THE DRUG EFFECTF THE HUMAN BODY AND ITS STATE FOR THE MANIFESTATION OF THE DRUG EFFECTTHE HUMAN BODY AND ITS STATE FOR THE MANIFESTATION OF THE DRUG EFFECTOF THE DRUG EFFECT THE DRUG EFFECTHE DRUG EFFECT DRUG EFFECT EFFECTFFECTECTt-ISBN9785970438831-0012ISBN9785970438831-0012BN9785970438831-00129785970438831-0012970438831-00120438831-0012831-00121-001200125970438831-0012.html70438831-0012.html438831-0012.html31-0012.html-0012.html012.html.   MAIN TYPES OF DRUG TREATMENT  MAIN TYPES OF DRUG TREATMENT MAIN TYPES OF DRUG TREATMENTAIN TYPES OF DRUG TREATMENTN TYPES OF DRUG TREATMENTivlassss38831-0013831-00131-00130013"><-0013.html013.html3.htmlhtmllassssALLERGIC REACTIONS. IDIOSYNCRASY. TOXIC EFFECTSLERGIC REACTIONS. IDIOSYNCRASY. TOXIC EFFECTSRGIC REACTIONS. IDIOSYNCRASY. TOXIC EFFECTS REACTIONS. IDIOSYNCRASY. TOXIC EFFECTSEACTIONS. IDIOSYNCRASY. TOXIC EFFECTSCTIONS. IDIOSYNCRASY. TOXIC EFFECTS"aTCont-ISBN9785970438831-0014Cont-ISBN9785970438831-0014nt-ISBN9785970438831-0014-ISBN9785970438831-0014SBN9785970438831-0014N9785970438831-0014tudentlibrary.ru/en/doc/ISBN9785970438831-0014.htmldentlibrary.ru/en/doc/ISBN9785970438831-0014.htmlntlibrary.ru/en/doc/ISBN9785970438831-0014.htmllibrary.ru/en/doc/ISBN9785970438831-0014.htmlry.ru/en/doc/ISBN9785970438831-0014.html.ru/en/doc/ISBN9785970438831-0014.htmlu/en/doc/ISBN9785970438831-0014.html0. GENERAL PRINCIPLES OF THE TREATMENT OF ACUTE DRUG POISONING 3ENERAL PRINCIPLES OF THE TREATMENT OF ACUTE DRUG POISONING 3ERAL PRINCIPLES OF THE TREATMENT OF ACUTE DRUG POISONING 3 PRINCIPLES OF THE TREATMENT OF ACUTE DRUG POISONING 3RINCIPLES OF THE TREATMENT OF ACUTE DRUG POISONING 3NCIPLES OF THE TREATMENT OF ACUTE DRUG POISONING 3APTER 2. DRUGS STIMULATING SENSORY NERVE ENDINGSTER 2. DRUGS STIMULATING SENSORY NERVE ENDINGSR 2. DRUGS STIMULATING SENSORY NERVE ENDINGS 2. DRUGS STIMULATING SENSORY NERVE ENDINGS. DRUGS STIMULATING SENSORY NERVE ENDINGSDRUGS STIMULATING SENSORY NERVE ENDINGSSTIMULATING SENSORY NERVE ENDINGSIMULATING SENSORY NERVE ENDINGSULATING SENSORY NERVE ENDINGSLATING SENSORY NERVE ENDINGSTING SENSORY NERVE ENDINGSNG SENSORY NERVE ENDINGSas://prior.studentlibrary.ru/en/doc/ISBN9785970438831-0019.html//prior.studentlibrary.ru/en/doc/ISBN9785970438831-0019.htmlior.studentlibrary.ru/en/doc/ISBN9785970438831-0019.htmlr.studentlibrary.ru/en/doc/ISBN9785970438831-0019.htmlentlibrary.ru/en/doc/ISBN9785970438831-0019.htmltlibrary.ru/en/doc/ISBN9785970438831-0019.htmlibrary.ru/en/doc/ISBN9785970438831-0019.htmln/doc/ISBN9785970438831-0019.htmldoc/ISBN9785970438831-0019.htmlc/ISBN9785970438831-0019.htmlSBN9785970438831-0019.htmlN9785970438831-0019.html785970438831-0019.htmlssTCont-row-doc-aont-row-doc-aw-doc-adoc-ac-a">B. Drugs affecting efferent innervationry.ru/en/doc/ISBN9785970438831-0020.html.ru/en/doc/ISBN9785970438831-0020.htmlu/en/doc/ISBN9785970438831-0020.htmlen/doc/ISBN9785970438831-0020.html/doc/ISBN9785970438831-0020.htmloc/ISBN9785970438831-0020.html/ISBN9785970438831-0020.htmlSBN9785970438831-0020.html5970438831-0020.html70438831-0020.html438831-0020.html31-0020.html-0020.html020.htmlCTING CHOLINERGIC SYNAPSESING CHOLINERGIC SYNAPSESG CHOLINERGIC SYNAPSESCHOLINERGIC SYNAPSESOLINERGIC SYNAPSESINERGIC SYNAPSESERGIC SYNAPSESGIC SYNAPSESNAPSESPSESESnt-ISBN9785970438831-0021-ISBN9785970438831-0021SBN9785970438831-0021N9785970438831-0021785970438831-00215970438831-002170438831-0021438831-0021-00210211>

0028.html28.html.htmltmll" class="aTCont-row-doc-ant-row-doc-a-row-doc-aow-doc-as="aTCont-row-docCont-row-docnt-row-doc-row-docow-doc="aTCont-ISBN9785970438831-00299.htmlhtmlmllassssTCont-row-doc-aont-row-doc-arow-doc-aw-doc-adoc-aCont-row-docnt-row-doc-row-docow-doc-dococ="aTCont-ISBN9785970438831-0030nt-ISBN9785970438831-0030-ISBN9785970438831-0030SBN9785970438831-00305970438831-0030.html70438831-0030.html438831-0030.html8831-0030.html31-0030.html-0030.html030.html0.htmlssnt-row-doc-row-docow-doc-dococ"aTCont-ISBN9785970438831-0031Cont-ISBN9785970438831-0031-ISBN9785970438831-0031SBN9785970438831-0031N9785970438831-0031c/ISBN9785970438831-0031.htmlISBN9785970438831-0031.htmlBN9785970438831-0031.html9785970438831-0031.html85970438831-0031.html970438831-0031.html0438831-0031.html38831-0031.html0031.html31.html.htmll" and their systemsd their systemstheir systemseir systemsr systemssystemsstemsems/a>/divvreff="https://prior.studentlibrary.ru/en/doc/ISBN9785970438831-0032.htmltps://prior.studentlibrary.ru/en/doc/ISBN9785970438831-0032.htmls://prior.studentlibrary.ru/en/doc/ISBN9785970438831-0032.htmlior.studentlibrary.ru/en/doc/ISBN9785970438831-0032.htmlr.studentlibrary.ru/en/doc/ISBN9785970438831-0032.htmlstudentlibrary.ru/en/doc/ISBN9785970438831-0032.htmlentlibrary.ru/en/doc/ISBN9785970438831-0032.htmltlibrary.ru/en/doc/ISBN9785970438831-0032.htmlibrary.ru/en/doc/ISBN9785970438831-0032.htmlCHAPTER 13. DRUGS AFFECTING THE RESPIRATORY SYSTEMAPTER 13. DRUGS AFFECTING THE RESPIRATORY SYSTEMTER 13. DRUGS AFFECTING THE RESPIRATORY SYSTEMR 13. DRUGS AFFECTING THE RESPIRATORY SYSTEM13. DRUGS AFFECTING THE RESPIRATORY SYSTEM. DRUGS AFFECTING THE RESPIRATORY SYSTEMDRUGS AFFECTING THE RESPIRATORY SYSTEMUGS AFFECTING THE RESPIRATORY SYSTEMFECTING THE RESPIRATORY SYSTEMCTING THE RESPIRATORY SYSTEMING THE RESPIRATORY SYSTEMTHE RESPIRATORY SYSTEME RESPIRATORY SYSTEMRESPIRATORY SYSTEMtps://prior.studentlibrary.ru/en/doc/ISBN9785970438831-0033.htmls://prior.studentlibrary.ru/en/doc/ISBN9785970438831-0033.html//prior.studentlibrary.ru/en/doc/ISBN9785970438831-0033.htmlprior.studentlibrary.ru/en/doc/ISBN9785970438831-0033.htmlior.studentlibrary.ru/en/doc/ISBN9785970438831-0033.htmlr.studentlibrary.ru/en/doc/ISBN9785970438831-0033.htmlstudentlibrary.ru/en/doc/ISBN9785970438831-0033.htmludentlibrary.ru/en/doc/ISBN9785970438831-0033.htmlibrary.ru/en/doc/ISBN9785970438831-0033.htmlrary.ru/en/doc/ISBN9785970438831-0033.htmlry.ru/en/doc/ISBN9785970438831-0033.htmlu/en/doc/ISBN9785970438831-0033.htmlen/doc/ISBN9785970438831-0033.html/doc/ISBN9785970438831-0033.htmlLAR SYSTEMR SYSTEMSYSTEMSTEMEMiviv//prior.studentlibrary.ru/en/doc/ISBN9785970438831-0034.htmlprior.studentlibrary.ru/en/doc/ISBN9785970438831-0034.htmlior.studentlibrary.ru/en/doc/ISBN9785970438831-0034.htmlr.studentlibrary.ru/en/doc/ISBN9785970438831-0034.htmlstudentlibrary.ru/en/doc/ISBN9785970438831-0034.htmludentlibrary.ru/en/doc/ISBN9785970438831-0034.htmlentlibrary.ru/en/doc/ISBN9785970438831-0034.htmltlibrary.ru/en/doc/ISBN9785970438831-0034.htmlry.ru/en/doc/ISBN9785970438831-0034.html.ru/en/doc/ISBN9785970438831-0034.htmlu/en/doc/ISBN9785970438831-0034.html/doc/ISBN9785970438831-0034.htmloc/ISBN9785970438831-0034.html/ISBN9785970438831-0034.htmlTCont-row-docont-row-doct-row-docrow-docw-docdoc idTCont-ISBN9785970438831-0035ont-ISBN9785970438831-0035PTER 16. DIURETICSER 16. DIURETICS 16. DIURETICS6. DIURETICS DIURETICSIURETICSRETICSTICSiv1-0036.html0036.html36.html.htmltmll" s="Cont-row-doc-ant-row-doc-a-row-doc-assTCont-row-docont-row-doct-row-docrow-docc" tlibrary.ru/en/doc/ISBN9785970438831-0037.htmlibrary.ru/en/doc/ISBN9785970438831-0037.htmlrary.ru/en/doc/ISBN9785970438831-0037.htmlry.ru/en/doc/ISBN9785970438831-0037.html.ru/en/doc/ISBN9785970438831-0037.htmlu/en/doc/ISBN9785970438831-0037.htmlen/doc/ISBN9785970438831-0037.html/doc/ISBN9785970438831-0037.htmlSBN9785970438831-0037.htmlN9785970438831-0037.html785970438831-0037.html70438831-0037.html438831-0037.html8831-0037.htmlCTING HEMOPOIESISING HEMOPOIESISG HEMOPOIESISHEMOPOIESISMOPOIESISPOIESISIESISSIS/a>/divv0388">CHAPTER 19. DRUGS THAT AFFECT PLATELET AGGREGATION, BLOOD COAGULATION AND FIBRINOLYSISR 19. DRUGS THAT AFFECT PLATELET AGGREGATION, BLOOD COAGULATION AND FIBRINOLYSIS19. DRUGS THAT AFFECT PLATELET AGGREGATION, BLOOD COAGULATION AND FIBRINOLYSIS. DRUGS THAT AFFECT PLATELET AGGREGATION, BLOOD COAGULATION AND FIBRINOLYSISUGS THAT AFFECT PLATELET AGGREGATION, BLOOD COAGULATION AND FIBRINOLYSISS THAT AFFECT PLATELET AGGREGATION, BLOOD COAGULATION AND FIBRINOLYSISTHAT AFFECT PLATELET AGGREGATION, BLOOD COAGULATION AND FIBRINOLYSIS/a>v classs="reff="https://prior.studentlibrary.ru/en/doc/ISBN9785970438831-0039.htmltps://prior.studentlibrary.ru/en/doc/ISBN9785970438831-0039.htmls://prior.studentlibrary.ru/en/doc/ISBN9785970438831-0039.htmlprior.studentlibrary.ru/en/doc/ISBN9785970438831-0039.htmlior.studentlibrary.ru/en/doc/ISBN9785970438831-0039.htmltlibrary.ru/en/doc/ISBN9785970438831-0039.htmllibrary.ru/en/doc/ISBN9785970438831-0039.html85970438831-0040970438831-00400438831-004038831-0040-004004005970438831-0040.html70438831-0040.html438831-0040.html8831-0040.html-0040.html040.htmlhtmlmla>" idTCont-ISBN9785970438831-0041ont-ISBN9785970438831-0041 hrefefttps://prior.studentlibrary.ru/en/doc/ISBN9785970438831-0041.html.htmltmll"lassssdivv classs=aTCont-row-docCont-row-docnt-row-doctps://prior.studentlibrary.ru/en/doc/ISBN9785970438831-0042.htmls://prior.studentlibrary.ru/en/doc/ISBN9785970438831-0042.html//prior.studentlibrary.ru/en/doc/ISBN9785970438831-0042.htmlior.studentlibrary.ru/en/doc/ISBN9785970438831-0042.htmlr.studentlibrary.ru/en/doc/ISBN9785970438831-0042.htmlstudentlibrary.ru/en/doc/ISBN9785970438831-0042.html38831-0042.html831-0042.html1-0042.html.htmltmllCHAPTER 22. ANTIATHEROSCLEROTIC DRUGS (DRUGS USED FOR THE TREATMENT OF HYPERLIPOPROTEINEMIA)APTER 22. ANTIATHEROSCLEROTIC DRUGS (DRUGS USED FOR THE TREATMENT OF HYPERLIPOPROTEINEMIA)TER 22. ANTIATHEROSCLEROTIC DRUGS (DRUGS USED FOR THE TREATMENT OF HYPERLIPOPROTEINEMIA)R 22. ANTIATHEROSCLEROTIC DRUGS (DRUGS USED FOR THE TREATMENT OF HYPERLIPOPROTEINEMIA)22. ANTIATHEROSCLEROTIC DRUGS (DRUGS USED FOR THE TREATMENT OF HYPERLIPOPROTEINEMIA). ANTIATHEROSCLEROTIC DRUGS (DRUGS USED FOR THE TREATMENT OF HYPERLIPOPROTEINEMIA)ANTIATHEROSCLEROTIC DRUGS (DRUGS USED FOR THE TREATMENT OF HYPERLIPOPROTEINEMIA)MENT OF HYPERLIPOPROTEINEMIA)NT OF HYPERLIPOPROTEINEMIA) OF HYPERLIPOPROTEINEMIA)F HYPERLIPOPROTEINEMIA)ERLIPOPROTEINEMIA)LIPOPROTEINEMIA)POPROTEINEMIA)d="aTCont-ISBN9785970438831-0043nt-ISBN9785970438831-0043-ISBN9785970438831-0043N9785970438831-0043785970438831-00435970438831-0043doc/ISBN9785970438831-0043.htmlc/ISBN9785970438831-0043.htmlISBN9785970438831-0043.html9785970438831-0043.html85970438831-0043.html970438831-0043.htmlw-doc-adoc-ac-a>CHAPTER 23. DRUGS USED FOR THE TREATMENT OF OBESITYAPTER 23. DRUGS USED FOR THE TREATMENT OF OBESITYBESITYSITYTYTCont-ISBN9785970438831-0044-ISBN9785970438831-0044SBN9785970438831-0044N9785970438831-0044en/doc/ISBN9785970438831-0044.html/doc/ISBN9785970438831-0044.htmloc/ISBN9785970438831-0044.html/ISBN9785970438831-0044.htmlN9785970438831-0044.html785970438831-0044.html70438831-0044.html438831-0044.html8831-0044.htmlS USED FOR THE TREATMENT AND PREVENTION OF OSTEOPOROSISUSED FOR THE TREATMENT AND PREVENTION OF OSTEOPOROSISED FOR THE TREATMENT AND PREVENTION OF OSTEOPOROSISOR THE TREATMENT AND PREVENTION OF OSTEOPOROSIS THE TREATMENT AND PREVENTION OF OSTEOPOROSISHE TREATMENT AND PREVENTION OF OSTEOPOROSISISd="aTCont-ISBN9785970438831-0045Cont-ISBN9785970438831-0045/prior.studentlibrary.ru/en/doc/ISBN9785970438831-0045.htmlrior.studentlibrary.ru/en/doc/ISBN9785970438831-0045.htmlor.studentlibrary.ru/en/doc/ISBN9785970438831-0045.html.studentlibrary.ru/en/doc/ISBN9785970438831-0045.htmlentlibrary.ru/en/doc/ISBN9785970438831-0045.htmltlibrary.ru/en/doc/ISBN9785970438831-0045.htmlibrary.ru/en/doc/ISBN9785970438831-0045.htmlow-doc-a-doc-aoc-a-aPTER 25. DRUGS USED FOR THE TREATMENT OF GOUTER 25. DRUGS USED FOR THE TREATMENT OF GOUT 25. DRUGS USED FOR THE TREATMENT OF GOUTlassssTCont-row-docont-row-doc785970438831-00465970438831-004670438831-004631-0046-0046046r.studentlibrary.ru/en/doc/ISBN9785970438831-0046.htmlstudentlibrary.ru/en/doc/ISBN9785970438831-0046.htmludentlibrary.ru/en/doc/ISBN9785970438831-0046.htmlentlibrary.ru/en/doc/ISBN9785970438831-0046.htmltlibrary.ru/en/doc/ISBN9785970438831-0046.htmlibrary.ru/en/doc/ISBN9785970438831-0046.htmlrary.ru/en/doc/ISBN9785970438831-0046.htmlCont-row-doc-a aTCont-current_docnt-row-doc-a aTCont-current_doc-row-doc-a aTCont-current_doc-doc-a aTCont-current_dococ-a aTCont-current_doc aTCont-current_docTCont-current_docont-current_doclassasss=Cont-row-docnt-row-doc-row-docrow-docw-docdocc"d=ps://prior.studentlibrary.ru/en/doc/ISBN9785970438831-0047.html://prior.studentlibrary.ru/en/doc/ISBN9785970438831-0047.htmlAPTER 27. DRUGS AFFECTING THE IMMUNE SYSTEMPTER 27. DRUGS AFFECTING THE IMMUNE SYSTEM50ttps://prior.studentlibrary.ru/en/doc/ISBN9785970438831-0051.html//prior.studentlibrary.ru/en/doc/ISBN9785970438831-0051.htmlprior.studentlibrary.ru/en/doc/ISBN9785970438831-0051.htmlior.studentlibrary.ru/en/doc/ISBN9785970438831-0051.htmlasss=TCont-row-docont-row-doct-row-docrow-doc0438831-0052.html38831-0052.html1-0052.html0052.htmltmll"t-row-doc-arow-doc-aw-doc-adoc-aa"APTER 31. ANTIFUNGAL AGENTSTER 31. ANTIFUNGAL AGENTSR 31. ANTIFUNGAL AGENTSL AGENTSAGENTSENTSSCHAPTER 34. ANTINEOPLASTIC (ANTIBLASTOMIC) DRUGSTER 34. ANTINEOPLASTIC (ANTIBLASTOMIC) DRUGSR 34. ANTINEOPLASTIC (ANTIBLASTOMIC) DRUGS34. ANTINEOPLASTIC (ANTIBLASTOMIC) DRUGSANTINEOPLASTIC (ANTIBLASTOMIC) DRUGSTINEOPLASTIC (ANTIBLASTOMIC) DRUGSLASTIC (ANTIBLASTOMIC) DRUGSSTIC (ANTIBLASTOMIC) DRUGSIC (ANTIBLASTOMIC) DRUGSIC) DRUGS) DRUGSDRUGSGSiv>

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asss=Cont-row-docnt-row-doc-row-docTCont-ISBN9785970438831-0031ttps://prior.studentlibrary.ru/en/doc/ISBN9785970438831-0031.htmlps://prior.studentlibrary.ru/en/doc/ISBN9785970438831-0031.html://prior.studentlibrary.ru/en/doc/ISBN9785970438831-0031.html/prior.studentlibrary.ru/en/doc/ISBN9785970438831-0031.htmlrior.studentlibrary.ru/en/doc/ISBN9785970438831-0031.htmlor.studentlibrary.ru/en/doc/ISBN9785970438831-0031.htmltudentlibrary.ru/en/doc/ISBN9785970438831-0031.htmldentlibrary.ru/en/doc/ISBN9785970438831-0031.htmlntlibrary.ru/en/doc/ISBN9785970438831-0031.htmlbrary.ru/en/doc/ISBN9785970438831-0031.htmlary.ru/en/doc/ISBN9785970438831-0031.html/en/doc/ISBN9785970438831-0031.htmln/doc/ISBN9785970438831-0031.htmldoc/ISBN9785970438831-0031.html438831-0031.html8831-0031.html31-0031.html0031.html31.html.html-doc-aoc-a-arugs regulating functions of major organs and their systemsgs regulating functions of major organs and their systems regulating functions of major organs and their systemsiviv-row-docow-doc-docior.studentlibrary.ru/en/doc/ISBN9785970438831-0032.htmlr.studentlibrary.ru/en/doc/ISBN9785970438831-0032.htmlstudentlibrary.ru/en/doc/ISBN9785970438831-0032.htmludentlibrary.ru/en/doc/ISBN9785970438831-0032.htmlentlibrary.ru/en/doc/ISBN9785970438831-0032.htmltlibrary.ru/en/doc/ISBN9785970438831-0032.htmlibrary.ru/en/doc/ISBN9785970438831-0032.htmlry.ru/en/doc/ISBN9785970438831-0032.html.ru/en/doc/ISBN9785970438831-0032.htmlu/en/doc/ISBN9785970438831-0032.html/doc/ISBN9785970438831-0032.htmloc/ISBN9785970438831-0032.htmlN9785970438831-0032.html785970438831-0032.html5970438831-0032.html.htmltmlllassCHAPTER 13. DRUGS AFFECTING THE RESPIRATORY SYSTEMAPTER 13. DRUGS AFFECTING THE RESPIRATORY SYSTEMTER 13. DRUGS AFFECTING THE RESPIRATORY SYSTEMR 13. DRUGS AFFECTING THE RESPIRATORY SYSTEM13. DRUGS AFFECTING THE RESPIRATORY SYSTEM. DRUGS AFFECTING THE RESPIRATORY SYSTEMGS AFFECTING THE RESPIRATORY SYSTEM AFFECTING THE RESPIRATORY SYSTEMFFECTING THE RESPIRATORY SYSTEMTCont-row-docont-row-docw-docdoccnt-ISBN9785970438831-0033-ISBN9785970438831-0033SBN9785970438831-0033f="https://prior.studentlibrary.ru/en/doc/ISBN9785970438831-0033.htmltps://prior.studentlibrary.ru/en/doc/ISBN9785970438831-0033.htmls://prior.studentlibrary.ru/en/doc/ISBN9785970438831-0033.html//prior.studentlibrary.ru/en/doc/ISBN9785970438831-0033.htmlior.studentlibrary.ru/en/doc/ISBN9785970438831-0033.htmlr.studentlibrary.ru/en/doc/ISBN9785970438831-0033.htmlstudentlibrary.ru/en/doc/ISBN9785970438831-0033.htmlentlibrary.ru/en/doc/ISBN9785970438831-0033.htmltlibrary.ru/en/doc/ISBN9785970438831-0033.htmlry.ru/en/doc/ISBN9785970438831-0033.html.ru/en/doc/ISBN9785970438831-0033.htmlu/en/doc/ISBN9785970438831-0033.html85970438831-0033.html970438831-0033.html0438831-0033.html8831-0033.html31-0033.html-0033.htmlt-row-doc-arow-doc-aw-doc-adoc-ac-aaHAPTER 14. DRUGS AFFECTING THE CARDIOVASCULAR SYSTEMPTER 14. DRUGS AFFECTING THE CARDIOVASCULAR SYSTEM>7. TYPICAL DERANGEMENTS OF CARBOHYDRATE METABOLISM. DIABETES MELLITUS

8. TYPICAL DERANGEMENTS OF LIPID METABOLISM. ATHEROSCLEROSIS

9. TYPICAL DISORDERS OF WATER-IONS BALANCE. EDEMA

10. TYPICAL DISTURBANCES OF ACID-BASE BALANCE

11. PATHOPHYSIOLOGY OF HYPOXIA

12. TYPICAL DISORDERS OF THE DEFENCE SYSTEM

13. TYPICAL FORMS OF ABNORMAL TISSUE GROWTH. TUMORS