Электронная библиотека технического вуза
us and articular tuberculous lesions include the epiphyses of the long bone shafts, bodies of the short bones: f.e. vertebrae and diaphyses of the digital phalanges. Anatomically, this type of tuberculosis falls into three major forms:nd articular tuberculous lesions include the epiphyses of the long bone shafts, bodies of the short bones: f.e. vertebrae and diaphyses of the digital phalanges. Anatomically, this type of tuberculosis falls into three major forms: articular tuberculous lesions include the epiphyses of the long bone shafts, bodies of the short bones: f.e. vertebrae and diaphyses of the digital phalanges. Anatomically, this type of tuberculosis falls into three major forms:icular tuberculous lesions include the epiphyses of the long bone shafts, bodies of the short bones: f.e. vertebrae and diaphyses of the digital phalanges. Anatomically, this type of tuberculosis falls into three major forms:ular tuberculous lesions include the epiphyses of the long bone shafts, bodies of the short bones: f.e. vertebrae and diaphyses of the digital phalanges. Anatomically, this type of tuberculosis falls into three major forms:ar tuberculous lesions include the epiphyses of the long bone shafts, bodies of the short bones: f.e. vertebrae and diaphyses of the digital phalanges. Anatomically, this type of tuberculosis falls into three major forms:g bone shafts, bodies of the short bones: f.e. vertebrae and diaphyses of the digital phalanges. Anatomically, this type of tuberculosis falls into three major forms:bone shafts, bodies of the short bones: f.e. vertebrae and diaphyses of the digital phalanges. Anatomically, this type of tuberculosis falls into three major forms:ne shafts, bodies of the short bones: f.e. vertebrae and diaphyses of the digital phalanges. Anatomically, this type of tuberculosis falls into three major forms:hafts, bodies of the short bones: f.e. vertebrae and diaphyses of the digital phalanges. Anatomically, this type of tuberculosis falls into three major forms:fts, bodies of the short bones: f.e. vertebrae and diaphyses of the digital phalanges. Anatomically, this type of tuberculosis falls into three major forms:s, bodies of the short bones: f.e. vertebrae and diaphyses of the digital phalanges. Anatomically, this type of tuberculosis falls into three major forms:, bodies of the short bones: f.e. vertebrae and diaphyses of the digital phalanges. Anatomically, this type of tuberculosis falls into three major forms:bodies of the short bones: f.e. vertebrae and diaphyses of the digital phalanges. Anatomically, this type of tuberculosis falls into three major forms:dies of the short bones: f.e. vertebrae and diaphyses of the digital phalanges. Anatomically, this type of tuberculosis falls into three major forms:f the short bones: f.e. vertebrae and diaphyses of the digital phalanges. Anatomically, this type of tuberculosis falls into three major forms:the short bones: f.e. vertebrae and diaphyses of the digital phalanges. Anatomically, this type of tuberculosis falls into three major forms:e short bones: f.e. vertebrae and diaphyses of the digital phalanges. Anatomically, this type of tuberculosis falls into three major forms: short bones: f.e. vertebrae and diaphyses of the digital phalanges. Anatomically, this type of tuberculosis falls into three major forms:hort bones: f.e. vertebrae and diaphyses of the digital phalanges. Anatomically, this type of tuberculosis falls into three major forms:rt bones: f.e. vertebrae and diaphyses of the digital phalanges. Anatomically, this type of tuberculosis falls into three major forms:diaphyses of the digital phalanges. Anatomically, this type of tuberculosis falls into three major forms:>
classasspan class="">
• tuberculous spondylitis, or spinal TB (40%);n class="">
• tuberculous spondylitis, or spinal TB (40%);class="">
• tuberculous spondylitis, or spinal TB (40%);class=""> tuberculous spondylitis, or spinal TB (40%);ass=""> tuberculous spondylitis, or spinal TB (40%);s=""> tuberculous spondylitis, or spinal TB (40%);""> tuberculous spondylitis, or spinal TB (40%);> tuberculous spondylitis, or spinal TB (40%);nbsp;tuberculous spondylitis, or spinal TB (40%);/span>tuberculous spondylitis, or spinal TB (40%);pan>tuberculous spondylitis, or spinal TB (40%);n>tuberculous spondylitis, or spinal TB (40%);berculous spondylitis, or spinal TB (40%);rculous spondylitis, or spinal TB (40%);ulous spondylitis, or spinal TB (40%);asss="txtt">n class="">
• tuberculous gonitis, or knee joint TB (15-20%).class="">
• tuberculous gonitis, or knee joint TB (15-20%).ass="">
• tuberculous gonitis, or knee joint TB (15-20%)."">
• tuberculous gonitis, or knee joint TB (15-20%).>
• tuberculous gonitis, or knee joint TB (15-20%).p>• tuberculous gonitis, or knee joint TB (15-20%).sp;tuberculous gonitis, or knee joint TB (15-20%).;tuberculous gonitis, or knee joint TB (15-20%)./span>tuberculous gonitis, or knee joint TB (15-20%).pan>tuberculous gonitis, or knee joint TB (15-20%).n>tuberculous gonitis, or knee joint TB (15-20%).tuberculous gonitis, or knee joint TB (15-20%).berculous gonitis, or knee joint TB (15-20%).rculous gonitis, or knee joint TB (15-20%).s gonitis, or knee joint TB (15-20%).gonitis, or knee joint TB (15-20%).nitis, or knee joint TB (15-20%).s, or knee joint TB (15-20%). or knee joint TB (15-20%).r knee joint TB (15-20%).>
pan class="">Tuberculosis of the bones and joints is usually due to a secondary TB infection, which implies its lymphohaematogenous spread from the primary pulmonary focus or that of other organs. M. tuberculosis bacteria induce specific inflammatory reaction within the bone marrow of epiphyses, the site known to have a slower blood flow. This results in primary ostiitis, or tuberculous osteomyelitis. The initial tuberculous focus undergoes caseous («cheese-like») necrosis around which further tubercles form to merge with the initial focus. Subsequently, granulation occurs which, in turn, is subject to necrosis. This leads to sequestration: the bony tissues involved in the necrosis become completely separated from the underlying bone.n class="">Tuberculosis of the bones and joints is usually due to a secondary TB infection, which implies its lymphohaematogenous spread from the primary pulmonary focus or that of other organs. M. tuberculosis bacteria induce specific inflammatory reaction within the bone marrow of epiphyses, the site known to have a slower blood flow. This results in primary ostiitis, or tuberculous osteomyelitis. The initial tuberculous focus undergoes caseous («cheese-like») necrosis around which further tubercles form to merge with the initial focus. Subsequently, granulation occurs which, in turn, is subject to necrosis. This leads to sequestration: the bony tissues involved in the necrosis become completely separated from the underlying bone.class="">Tuberculosis of the bones and joints is usually due to a secondary TB infection, which implies its lymphohaematogenous spread from the primary pulmonary focus or that of other organs. M. tuberculosis bacteria induce specific inflammatory reaction within the bone marrow of epiphyses, the site known to have a slower blood flow. This results in primary ostiitis, or tuberculous osteomyelitis. The initial tuberculous focus undergoes caseous («cheese-like») necrosis around which further tubercles form to merge with the initial focus. Subsequently, granulation occurs which, in turn, is subject to necrosis. This leads to sequestration: the bony tissues involved in the necrosis become completely separated from the underlying bone.ry pulmonary focus or that of other organs. M. tuberculosis bacteria induce specific inflammatory reaction within the bone marrow of epiphyses, the site known to have a slower blood flow. This results in primary ostiitis, or tuberculous osteomyelitis. The initial tuberculous focus undergoes caseous («cheese-like») necrosis around which further tubercles form to merge with the initial focus. Subsequently, granulation occurs which, in turn, is subject to necrosis. This leads to sequestration: the bony tissues involved in the necrosis become completely separated from the underlying bone. pulmonary focus or that of other organs. M. tuberculosis bacteria induce specific inflammatory reaction within the bone marrow of epiphyses, the site known to have a slower blood flow. This results in primary ostiitis, or tuberculous osteomyelitis. The initial tuberculous focus undergoes caseous («cheese-like») necrosis around which further tubercles form to merge with the initial focus. Subsequently, granulation occurs which, in turn, is subject to necrosis. This leads to sequestration: the bony tissues involved in the necrosis become completely separated from the underlying bone.ulmonary focus or that of other organs. M. tuberculosis bacteria induce specific inflammatory reaction within the bone marrow of epiphyses, the site known to have a slower blood flow. This results in primary ostiitis, or tuberculous osteomyelitis. The initial tuberculous focus undergoes caseous («cheese-like») necrosis around which further tubercles form to merge with the initial focus. Subsequently, granulation occurs which, in turn, is subject to necrosis. This leads to sequestration: the bony tissues involved in the necrosis become completely separated from the underlying bone.monary focus or that of other organs. M. tuberculosis bacteria induce specific inflammatory reaction within the bone marrow of epiphyses, the site known to have a slower blood flow. This results in primary ostiitis, or tuberculous osteomyelitis. The initial tuberculous focus undergoes caseous («cheese-like») necrosis around which further tubercles form to merge with the initial focus. Subsequently, granulation occurs which, in turn, is subject to necrosis. This leads to sequestration: the bony tissues involved in the necrosis become completely separated from the underlying bone.nary focus or that of other organs. M. tuberculosis bacteria induce specific inflammatory reaction within the bone marrow of epiphyses, the site known to have a slower blood flow. This results in primary ostiitis, or tuberculous osteomyelitis. The initial tuberculous focus undergoes caseous («cheese-like») necrosis around which further tubercles form to merge with the initial focus. Subsequently, granulation occurs which, in turn, is subject to necrosis. This leads to sequestration: the bony tissues involved in the necrosis become completely separated from the underlying bone.ry focus or that of other organs. M. tuberculosis bacteria induce specific inflammatory reaction within the bone marrow of epiphyses, the site known to have a slower blood flow. This results in primary ostiitis, or tuberculous osteomyelitis. The initial tuberculous focus undergoes caseous («cheese-like») necrosis around which further tubercles form to merge with the initial focus. Subsequently, granulation occurs which, in turn, is subject to necrosis. This leads to sequestration: the bony tissues involved in the necrosis become completely separated from the underlying bone. focus or that of other organs. M. tuberculosis bacteria induce specific inflammatory reaction within the bone marrow of epiphyses, the site known to have a slower blood flow. This results in primary ostiitis, or tuberculous osteomyelitis. The initial tuberculous focus undergoes caseous («cheese-like») necrosis around which further tubercles form to merge with the initial focus. Subsequently, granulation occurs which, in turn, is subject to necrosis. This leads to sequestration: the bony tissues involved in the necrosis become completely separated from the underlying bone.ocus or that of other organs. M. tuberculosis bacteria induce specific inflammatory reaction within the bone marrow of epiphyses, the site known to have a slower blood flow. This results in primary ostiitis, or tuberculous osteomyelitis. The initial tuberculous focus undergoes caseous («cheese-like») necrosis around which further tubercles form to merge with the initial focus. Subsequently, granulation occurs which, in turn, is subject to necrosis. This leads to sequestration: the bony tissues involved in the necrosis become completely separated from the underlying bone.r that of other organs. M. tuberculosis bacteria induce specific inflammatory reaction within the bone marrow of epiphyses, the site known to have a slower blood flow. This results in primary ostiitis, or tuberculous osteomyelitis. The initial tuberculous focus undergoes caseous («cheese-like») necrosis around which further tubercles form to merge with the initial focus. Subsequently, granulation occurs which, in turn, is subject to necrosis. This leads to sequestration: the bony tissues involved in the necrosis become completely separated from the underlying bone.that of other organs. M. tuberculosis bacteria induce specific inflammatory reaction within the bone marrow of epiphyses, the site known to have a slower blood flow. This results in primary ostiitis, or tuberculous osteomyelitis. The initial tuberculous focus undergoes caseous («cheese-like») necrosis around which further tubercles form to merge with the initial focus. Subsequently, granulation occurs which, in turn, is subject to necrosis. This leads to sequestration: the bony tissues involved in the necrosis become completely separated from the underlying bone.at of other organs. M. tuberculosis bacteria induce specific inflammatory reaction within the bone marrow of epiphyses, the site known to have a slower blood flow. This results in primary ostiitis, or tuberculous osteomyelitis. The initial tuberculous focus undergoes caseous («cheese-like») necrosis around which further tubercles form to merge with the initial focus. Subsequently, granulation occurs which, in turn, is subject to necrosis. This leads to sequestration: the bony tissues involved in the necrosis become completely separated from the underlying bone.f other organs. M. tuberculosis bacteria induce specific inflammatory reaction within the bone marrow of epiphyses, the site known to have a slower blood flow. This results in primary ostiitis, or tuberculous osteomyelitis. The initial tuberculous focus undergoes caseous («cheese-like») necrosis around which further tubercles form to merge with the initial focus. Subsequently, granulation occurs which, in turn, is subject to necrosis. This leads to sequestration: the bony tissues involved in the necrosis become completely separated from the underlying bone.other organs. M. tuberculosis bacteria induce specific inflammatory reaction within the bone marrow of epiphyses, the site known to have a slower blood flow. This results in primary ostiitis, or tuberculous osteomyelitis. The initial tuberculous focus undergoes caseous («cheese-like») necrosis around which further tubercles form to merge with the initial focus. Subsequently, granulation occurs which, in turn, is subject to necrosis. This leads to sequestration: the bony tissues involved in the necrosis become completely separated from the underlying bone.her organs. M. tuberculosis bacteria induce specific inflammatory reaction within the bone marrow of epiphyses, the site known to have a slower blood flow. This results in primary ostiitis, or tuberculous osteomyelitis. The initial tuberculous focus undergoes caseous («cheese-like») necrosis around which further tubercles form to merge with the initial focus. Subsequently, granulation occurs which, in turn, is subject to necrosis. This leads to sequestration: the bony tissues involved in the necrosis become completely separated from the underlying bone.ses, the site known to have a slower blood flow. This results in primary ostiitis, or tuberculous osteomyelitis. The initial tuberculous focus undergoes caseous («cheese-like») necrosis around which further tubercles form to merge with the initial focus. Subsequently, granulation occurs which, in turn, is subject to necrosis. This leads to sequestration: the bony tissues involved in the necrosis become completely separated from the underlying bone.s, the site known to have a slower blood flow. This results in primary ostiitis, or tuberculous osteomyelitis. The initial tuberculous focus undergoes caseous («cheese-like») necrosis around which further tubercles form to merge with the initial focus. Subsequently, granulation occurs which, in turn, is subject to necrosis. This leads to sequestration: the bony tissues involved in the necrosis become completely separated from the underlying bone. the site known to have a slower blood flow. This results in primary ostiitis, or tuberculous osteomyelitis. The initial tuberculous focus undergoes caseous («cheese-like») necrosis around which further tubercles form to merge with the initial focus. Subsequently, granulation occurs which, in turn, is subject to necrosis. This leads to sequestration: the bony tissues involved in the necrosis become completely separated from the underlying bone.he site known to have a slower blood flow. This results in primary ostiitis, or tuberculous osteomyelitis. The initial tuberculous focus undergoes caseous («cheese-like») necrosis around which further tubercles form to merge with the initial focus. Subsequently, granulation occurs which, in turn, is subject to necrosis. This leads to sequestration: the bony tissues involved in the necrosis become completely separated from the underlying bone. site known to have a slower blood flow. This results in primary ostiitis, or tuberculous osteomyelitis. The initial tuberculous focus undergoes caseous («cheese-like») necrosis around which further tubercles form to merge with the initial focus. Subsequently, granulation occurs which, in turn, is subject to necrosis. This leads to sequestration: the bony tissues involved in the necrosis become completely separated from the underlying bone.ite known to have a slower blood flow. This results in primary ostiitis, or tuberculous osteomyelitis. The initial tuberculous focus undergoes caseous («cheese-like») necrosis around which further tubercles form to merge with the initial focus. Subsequently, granulation occurs which, in turn, is subject to necrosis. This leads to sequestration: the bony tissues involved in the necrosis become completely separated from the underlying bone.e known to have a slower blood flow. This results in primary ostiitis, or tuberculous osteomyelitis. The initial tuberculous focus undergoes caseous («cheese-like») necrosis around which further tubercles form to merge with the initial focus. Subsequently, granulation occurs which, in turn, is subject to necrosis. This leads to sequestration: the bony tissues involved in the necrosis become completely separated from the underlying bone.known to have a slower blood flow. This results in primary ostiitis, or tuberculous osteomyelitis. The initial tuberculous focus undergoes caseous («cheese-like») necrosis around which further tubercles form to merge with the initial focus. Subsequently, granulation occurs which, in turn, is subject to necrosis. This leads to sequestration: the bony tissues involved in the necrosis become completely separated from the underlying bone.to have a slower blood flow. This results in primary ostiitis, or tuberculous osteomyelitis. The initial tuberculous focus undergoes caseous («cheese-like») necrosis around which further tubercles form to merge with the initial focus. Subsequently, granulation occurs which, in turn, is subject to necrosis. This leads to sequestration: the bony tissues involved in the necrosis become completely separated from the underlying bone. have a slower blood flow. This results in primary ostiitis, or tuberculous osteomyelitis. The initial tuberculous focus undergoes caseous («cheese-like») necrosis around which further tubercles form to merge with the initial focus. Subsequently, granulation occurs which, in turn, is subject to necrosis. This leads to sequestration: the bony tissues involved in the necrosis become completely separated from the underlying bone.ave a slower blood flow. This results in primary ostiitis, or tuberculous osteomyelitis. The initial tuberculous focus undergoes caseous («cheese-like») necrosis around which further tubercles form to merge with the initial focus. Subsequently, granulation occurs which, in turn, is subject to necrosis. This leads to sequestration: the bony tissues involved in the necrosis become completely separated from the underlying bone.a slower blood flow. This results in primary ostiitis, or tuberculous osteomyelitis. The initial tuberculous focus undergoes caseous («cheese-like») necrosis around which further tubercles form to merge with the initial focus. Subsequently, granulation occurs which, in turn, is subject to necrosis. This leads to sequestration: the bony tissues involved in the necrosis become completely separated from the underlying bone.slower blood flow. This results in primary ostiitis, or tuberculous osteomyelitis. The initial tuberculous focus undergoes caseous («cheese-like») necrosis around which further tubercles form to merge with the initial focus. Subsequently, granulation occurs which, in turn, is subject to necrosis. This leads to sequestration: the bony tissues involved in the necrosis become completely separated from the underlying bone.ower blood flow. This results in primary ostiitis, or tuberculous osteomyelitis. The initial tuberculous focus undergoes caseous («cheese-like») necrosis around which further tubercles form to merge with the initial focus. Subsequently, granulation occurs which, in turn, is subject to necrosis. This leads to sequestration: the bony tissues involved in the necrosis become completely separated from the underlying bone.eese-like») necrosis around which further tubercles form to merge with the initial focus. Subsequently, granulation occurs which, in turn, is subject to necrosis. This leads to sequestration: the bony tissues involved in the necrosis become completely separated from the underlying bone.se-like») necrosis around which further tubercles form to merge with the initial focus. Subsequently, granulation occurs which, in turn, is subject to necrosis. This leads to sequestration: the bony tissues involved in the necrosis become completely separated from the underlying bone.-like») necrosis around which further tubercles form to merge with the initial focus. Subsequently, granulation occurs which, in turn, is subject to necrosis. This leads to sequestration: the bony tissues involved in the necrosis become completely separated from the underlying bone.ike») necrosis around which further tubercles form to merge with the initial focus. Subsequently, granulation occurs which, in turn, is subject to necrosis. This leads to sequestration: the bony tissues involved in the necrosis become completely separated from the underlying bone.e») necrosis around which further tubercles form to merge with the initial focus. Subsequently, granulation occurs which, in turn, is subject to necrosis. This leads to sequestration: the bony tissues involved in the necrosis become completely separated from the underlying bone.raquo;) necrosis around which further tubercles form to merge with the initial focus. Subsequently, granulation occurs which, in turn, is subject to necrosis. This leads to sequestration: the bony tissues involved in the necrosis become completely separated from the underlying bone.quo;) necrosis around which further tubercles form to merge with the initial focus. Subsequently, granulation occurs which, in turn, is subject to necrosis. This leads to sequestration: the bony tissues involved in the necrosis become completely separated from the underlying bone.o;) necrosis around which further tubercles form to merge with the initial focus. Subsequently, granulation occurs which, in turn, is subject to necrosis. This leads to sequestration: the bony tissues involved in the necrosis become completely separated from the underlying bone.crosis around which further tubercles form to merge with the initial focus. Subsequently, granulation occurs which, in turn, is subject to necrosis. This leads to sequestration: the bony tissues involved in the necrosis become completely separated from the underlying bone.osis around which further tubercles form to merge with the initial focus. Subsequently, granulation occurs which, in turn, is subject to necrosis. This leads to sequestration: the bony tissues involved in the necrosis become completely separated from the underlying bone.is around which further tubercles form to merge with the initial focus. Subsequently, granulation occurs which, in turn, is subject to necrosis. This leads to sequestration: the bony tissues involved in the necrosis become completely separated from the underlying bone.round which further tubercles form to merge with the initial focus. Subsequently, granulation occurs which, in turn, is subject to necrosis. This leads to sequestration: the bony tissues involved in the necrosis become completely separated from the underlying bone.und which further tubercles form to merge with the initial focus. Subsequently, granulation occurs which, in turn, is subject to necrosis. This leads to sequestration: the bony tissues involved in the necrosis become completely separated from the underlying bone.d which further tubercles form to merge with the initial focus. Subsequently, granulation occurs which, in turn, is subject to necrosis. This leads to sequestration: the bony tissues involved in the necrosis become completely separated from the underlying bone. to sequestration: the bony tissues involved in the necrosis become completely separated from the underlying bone.o sequestration: the bony tissues involved in the necrosis become completely separated from the underlying bone.sequestration: the bony tissues involved in the necrosis become completely separated from the underlying bone.questration: the bony tissues involved in the necrosis become completely separated from the underlying bone.estration: the bony tissues involved in the necrosis become completely separated from the underlying bone.tration: the bony tissues involved in the necrosis become completely separated from the underlying bone.ation: the bony tissues involved in the necrosis become completely separated from the underlying bone.ion: the bony tissues involved in the necrosis become completely separated from the underlying bone.he bony tissues involved in the necrosis become completely separated from the underlying bone. bony tissues involved in the necrosis become completely separated from the underlying bone.ony tissues involved in the necrosis become completely separated from the underlying bone.tissues involved in the necrosis become completely separated from the underlying bone.ssues involved in the necrosis become completely separated from the underlying bone.ues involved in the necrosis become completely separated from the underlying bone.erculous inflammation into the joint forms an articular sac with subsequent caseous necrosis of the joint cartilage and contiguous osseous tissues.culous inflammation into the joint forms an articular sac with subsequent caseous necrosis of the joint cartilage and contiguous osseous tissues.lous inflammation into the joint forms an articular sac with subsequent caseous necrosis of the joint cartilage and contiguous osseous tissues.us inflammation into the joint forms an articular sac with subsequent caseous necrosis of the joint cartilage and contiguous osseous tissues. inflammation into the joint forms an articular sac with subsequent caseous necrosis of the joint cartilage and contiguous osseous tissues.nflammation into the joint forms an articular sac with subsequent caseous necrosis of the joint cartilage and contiguous osseous tissues.lammation into the joint forms an articular sac with subsequent caseous necrosis of the joint cartilage and contiguous osseous tissues.mmation into the joint forms an articular sac with subsequent caseous necrosis of the joint cartilage and contiguous osseous tissues.n into the joint forms an articular sac with subsequent caseous necrosis of the joint cartilage and contiguous osseous tissues.into the joint forms an articular sac with subsequent caseous necrosis of the joint cartilage and contiguous osseous tissues.to the joint forms an articular sac with subsequent caseous necrosis of the joint cartilage and contiguous osseous tissues.he joint forms an articular sac with subsequent caseous necrosis of the joint cartilage and contiguous osseous tissues. joint forms an articular sac with subsequent caseous necrosis of the joint cartilage and contiguous osseous tissues.oint forms an articular sac with subsequent caseous necrosis of the joint cartilage and contiguous osseous tissues.iitis) only occurs in about 5% of cases, with a favourable outcome as it is unlikely to cause the damage to the joint ends of the bones.tis) only occurs in about 5% of cases, with a favourable outcome as it is unlikely to cause the damage to the joint ends of the bones.s) only occurs in about 5% of cases, with a favourable outcome as it is unlikely to cause the damage to the joint ends of the bones. only occurs in about 5% of cases, with a favourable outcome as it is unlikely to cause the damage to the joint ends of the bones.nly occurs in about 5% of cases, with a favourable outcome as it is unlikely to cause the damage to the joint ends of the bones.y occurs in about 5% of cases, with a favourable outcome as it is unlikely to cause the damage to the joint ends of the bones.occurs in about 5% of cases, with a favourable outcome as it is unlikely to cause the damage to the joint ends of the bones.curs in about 5% of cases, with a favourable outcome as it is unlikely to cause the damage to the joint ends of the bones.n about 5% of cases, with a favourable outcome as it is unlikely to cause the damage to the joint ends of the bones.about 5% of cases, with a favourable outcome as it is unlikely to cause the damage to the joint ends of the bones.out 5% of cases, with a favourable outcome as it is unlikely to cause the damage to the joint ends of the bones.5% of cases, with a favourable outcome as it is unlikely to cause the damage to the joint ends of the bones. of cases, with a favourable outcome as it is unlikely to cause the damage to the joint ends of the bones.f cases, with a favourable outcome as it is unlikely to cause the damage to the joint ends of the bones.ones.es../span>pan>n>/div> effect">
© "STUDENT ADVISOR" LLS
Electronic Library System «Student Advisor»
Sadovnicheskaya Str. 11, bldg. 12, Moscow, Russia
Sales Department of "Student's Consultant" ELS
sale@studentlibrary.ru
+7(495)921-39-07 extension 648, 655
Technical support
support@studentlibrary.ru
+7(495)921-39-07
+7(917)550-49-08
Educational establishments