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minus;99%) of adult people of developed countries. The spine is a complex movable supporting structure. The main load in it falls on the intervertebral discs, consisting of an elastic cushioning nucleus pulposus and a fibrous ring surrounding the nucleus. The disc is connected to the upper and lower vertebrae through cartilaginous hyaline plates. With age, the water content in the discs and their elasticity decrease significantly. The small vessels supplying the disc are obliterated by 20−30 years, and metabolic processes in the disc are carried out by diffusion.nus;99%) of adult people of developed countries. The spine is a complex movable supporting structure. The main load in it falls on the intervertebral discs, consisting of an elastic cushioning nucleus pulposus and a fibrous ring surrounding the nucleus. The disc is connected to the upper and lower vertebrae through cartilaginous hyaline plates. With age, the water content in the discs and their elasticity decrease significantly. The small vessels supplying the disc are obliterated by 20−30 years, and metabolic processes in the disc are carried out by diffusion.s;99%) of adult people of developed countries. The spine is a complex movable supporting structure. The main load in it falls on the intervertebral discs, consisting of an elastic cushioning nucleus pulposus and a fibrous ring surrounding the nucleus. The disc is connected to the upper and lower vertebrae through cartilaginous hyaline plates. With age, the water content in the discs and their elasticity decrease significantly. The small vessels supplying the disc are obliterated by 20−30 years, and metabolic processes in the disc are carried out by diffusion. of adult people of developed countries. The spine is a complex movable supporting structure. The main load in it falls on the intervertebral discs, consisting of an elastic cushioning nucleus pulposus and a fibrous ring surrounding the nucleus. The disc is connected to the upper and lower vertebrae through cartilaginous hyaline plates. With age, the water content in the discs and their elasticity decrease significantly. The small vessels supplying the disc are obliterated by 20−30 years, and metabolic processes in the disc are carried out by diffusion.f adult people of developed countries. The spine is a complex movable supporting structure. The main load in it falls on the intervertebral discs, consisting of an elastic cushioning nucleus pulposus and a fibrous ring surrounding the nucleus. The disc is connected to the upper and lower vertebrae through cartilaginous hyaline plates. With age, the water content in the discs and their elasticity decrease significantly. The small vessels supplying the disc are obliterated by 20−30 years, and metabolic processes in the disc are carried out by diffusion.adult people of developed countries. The spine is a complex movable supporting structure. The main load in it falls on the intervertebral discs, consisting of an elastic cushioning nucleus pulposus and a fibrous ring surrounding the nucleus. The disc is connected to the upper and lower vertebrae through cartilaginous hyaline plates. With age, the water content in the discs and their elasticity decrease significantly. The small vessels supplying the disc are obliterated by 20−30 years, and metabolic processes in the disc are carried out by diffusion.ed countries. The spine is a complex movable supporting structure. The main load in it falls on the intervertebral discs, consisting of an elastic cushioning nucleus pulposus and a fibrous ring surrounding the nucleus. The disc is connected to the upper and lower vertebrae through cartilaginous hyaline plates. With age, the water content in the discs and their elasticity decrease significantly. The small vessels supplying the disc are obliterated by 20−30 years, and metabolic processes in the disc are carried out by diffusion. countries. The spine is a complex movable supporting structure. The main load in it falls on the intervertebral discs, consisting of an elastic cushioning nucleus pulposus and a fibrous ring surrounding the nucleus. The disc is connected to the upper and lower vertebrae through cartilaginous hyaline plates. With age, the water content in the discs and their elasticity decrease significantly. The small vessels supplying the disc are obliterated by 20−30 years, and metabolic processes in the disc are carried out by diffusion.ountries. The spine is a complex movable supporting structure. The main load in it falls on the intervertebral discs, consisting of an elastic cushioning nucleus pulposus and a fibrous ring surrounding the nucleus. The disc is connected to the upper and lower vertebrae through cartilaginous hyaline plates. With age, the water content in the discs and their elasticity decrease significantly. The small vessels supplying the disc are obliterated by 20−30 years, and metabolic processes in the disc are carried out by diffusion.ntries. The spine is a complex movable supporting structure. The main load in it falls on the intervertebral discs, consisting of an elastic cushioning nucleus pulposus and a fibrous ring surrounding the nucleus. The disc is connected to the upper and lower vertebrae through cartilaginous hyaline plates. With age, the water content in the discs and their elasticity decrease significantly. The small vessels supplying the disc are obliterated by 20−30 years, and metabolic processes in the disc are carried out by diffusion.ries. The spine is a complex movable supporting structure. The main load in it falls on the intervertebral discs, consisting of an elastic cushioning nucleus pulposus and a fibrous ring surrounding the nucleus. The disc is connected to the upper and lower vertebrae through cartilaginous hyaline plates. With age, the water content in the discs and their elasticity decrease significantly. The small vessels supplying the disc are obliterated by 20−30 years, and metabolic processes in the disc are carried out by diffusion.es. The spine is a complex movable supporting structure. The main load in it falls on the intervertebral discs, consisting of an elastic cushioning nucleus pulposus and a fibrous ring surrounding the nucleus. The disc is connected to the upper and lower vertebrae through cartilaginous hyaline plates. With age, the water content in the discs and their elasticity decrease significantly. The small vessels supplying the disc are obliterated by 20−30 years, and metabolic processes in the disc are carried out by diffusion.. The spine is a complex movable supporting structure. The main load in it falls on the intervertebral discs, consisting of an elastic cushioning nucleus pulposus and a fibrous ring surrounding the nucleus. The disc is connected to the upper and lower vertebrae through cartilaginous hyaline plates. With age, the water content in the discs and their elasticity decrease significantly. The small vessels supplying the disc are obliterated by 20−30 years, and metabolic processes in the disc are carried out by diffusion.The spine is a complex movable supporting structure. The main load in it falls on the intervertebral discs, consisting of an elastic cushioning nucleus pulposus and a fibrous ring surrounding the nucleus. The disc is connected to the upper and lower vertebrae through cartilaginous hyaline plates. With age, the water content in the discs and their elasticity decrease significantly. The small vessels supplying the disc are obliterated by 20−30 years, and metabolic processes in the disc are carried out by diffusion.e spine is a complex movable supporting structure. The main load in it falls on the intervertebral discs, consisting of an elastic cushioning nucleus pulposus and a fibrous ring surrounding the nucleus. The disc is connected to the upper and lower vertebrae through cartilaginous hyaline plates. With age, the water content in the discs and their elasticity decrease significantly. The small vessels supplying the disc are obliterated by 20−30 years, and metabolic processes in the disc are carried out by diffusion.ine is a complex movable supporting structure. The main load in it falls on the intervertebral discs, consisting of an elastic cushioning nucleus pulposus and a fibrous ring surrounding the nucleus. The disc is connected to the upper and lower vertebrae through cartilaginous hyaline plates. With age, the water content in the discs and their elasticity decrease significantly. The small vessels supplying the disc are obliterated by 20−30 years, and metabolic processes in the disc are carried out by diffusion.e is a complex movable supporting structure. The main load in it falls on the intervertebral discs, consisting of an elastic cushioning nucleus pulposus and a fibrous ring surrounding the nucleus. The disc is connected to the upper and lower vertebrae through cartilaginous hyaline plates. With age, the water content in the discs and their elasticity decrease significantly. The small vessels supplying the disc are obliterated by 20−30 years, and metabolic processes in the disc are carried out by diffusion. complex movable supporting structure. The main load in it falls on the intervertebral discs, consisting of an elastic cushioning nucleus pulposus and a fibrous ring surrounding the nucleus. The disc is connected to the upper and lower vertebrae through cartilaginous hyaline plates. With age, the water content in the discs and their elasticity decrease significantly. The small vessels supplying the disc are obliterated by 20−30 years, and metabolic processes in the disc are carried out by diffusion.omplex movable supporting structure. The main load in it falls on the intervertebral discs, consisting of an elastic cushioning nucleus pulposus and a fibrous ring surrounding the nucleus. The disc is connected to the upper and lower vertebrae through cartilaginous hyaline plates. With age, the water content in the discs and their elasticity decrease significantly. The small vessels supplying the disc are obliterated by 20−30 years, and metabolic processes in the disc are carried out by diffusion.plex movable supporting structure. The main load in it falls on the intervertebral discs, consisting of an elastic cushioning nucleus pulposus and a fibrous ring surrounding the nucleus. The disc is connected to the upper and lower vertebrae through cartilaginous hyaline plates. With age, the water content in the discs and their elasticity decrease significantly. The small vessels supplying the disc are obliterated by 20−30 years, and metabolic processes in the disc are carried out by diffusion.rting structure. The main load in it falls on the intervertebral discs, consisting of an elastic cushioning nucleus pulposus and a fibrous ring surrounding the nucleus. The disc is connected to the upper and lower vertebrae through cartilaginous hyaline plates. With age, the water content in the discs and their elasticity decrease significantly. The small vessels supplying the disc are obliterated by 20−30 years, and metabolic processes in the disc are carried out by diffusion.ing structure. The main load in it falls on the intervertebral discs, consisting of an elastic cushioning nucleus pulposus and a fibrous ring surrounding the nucleus. The disc is connected to the upper and lower vertebrae through cartilaginous hyaline plates. With age, the water content in the discs and their elasticity decrease significantly. The small vessels supplying the disc are obliterated by 20−30 years, and metabolic processes in the disc are carried out by diffusion.g structure. The main load in it falls on the intervertebral discs, consisting of an elastic cushioning nucleus pulposus and a fibrous ring surrounding the nucleus. The disc is connected to the upper and lower vertebrae through cartilaginous hyaline plates. With age, the water content in the discs and their elasticity decrease significantly. The small vessels supplying the disc are obliterated by 20−30 years, and metabolic processes in the disc are carried out by diffusion.structure. The main load in it falls on the intervertebral discs, consisting of an elastic cushioning nucleus pulposus and a fibrous ring surrounding the nucleus. The disc is connected to the upper and lower vertebrae through cartilaginous hyaline plates. With age, the water content in the discs and their elasticity decrease significantly. The small vessels supplying the disc are obliterated by 20−30 years, and metabolic processes in the disc are carried out by diffusion.ructure. The main load in it falls on the intervertebral discs, consisting of an elastic cushioning nucleus pulposus and a fibrous ring surrounding the nucleus. The disc is connected to the upper and lower vertebrae through cartilaginous hyaline plates. With age, the water content in the discs and their elasticity decrease significantly. The small vessels supplying the disc are obliterated by 20−30 years, and metabolic processes in the disc are carried out by diffusion.ure. The main load in it falls on the intervertebral discs, consisting of an elastic cushioning nucleus pulposus and a fibrous ring surrounding the nucleus. The disc is connected to the upper and lower vertebrae through cartilaginous hyaline plates. With age, the water content in the discs and their elasticity decrease significantly. The small vessels supplying the disc are obliterated by 20−30 years, and metabolic processes in the disc are carried out by diffusion.e. The main load in it falls on the intervertebral discs, consisting of an elastic cushioning nucleus pulposus and a fibrous ring surrounding the nucleus. The disc is connected to the upper and lower vertebrae through cartilaginous hyaline plates. With age, the water content in the discs and their elasticity decrease significantly. The small vessels supplying the disc are obliterated by 20−30 years, and metabolic processes in the disc are carried out by diffusion. The main load in it falls on the intervertebral discs, consisting of an elastic cushioning nucleus pulposus and a fibrous ring surrounding the nucleus. The disc is connected to the upper and lower vertebrae through cartilaginous hyaline plates. With age, the water content in the discs and their elasticity decrease significantly. The small vessels supplying the disc are obliterated by 20−30 years, and metabolic processes in the disc are carried out by diffusion.he main load in it falls on the intervertebral discs, consisting of an elastic cushioning nucleus pulposus and a fibrous ring surrounding the nucleus. The disc is connected to the upper and lower vertebrae through cartilaginous hyaline plates. With age, the water content in the discs and their elasticity decrease significantly. The small vessels supplying the disc are obliterated by 20−30 years, and metabolic processes in the disc are carried out by diffusion.ain load in it falls on the intervertebral discs, consisting of an elastic cushioning nucleus pulposus and a fibrous ring surrounding the nucleus. The disc is connected to the upper and lower vertebrae through cartilaginous hyaline plates. With age, the water content in the discs and their elasticity decrease significantly. The small vessels supplying the disc are obliterated by 20−30 years, and metabolic processes in the disc are carried out by diffusion.n load in it falls on the intervertebral discs, consisting of an elastic cushioning nucleus pulposus and a fibrous ring surrounding the nucleus. The disc is connected to the upper and lower vertebrae through cartilaginous hyaline plates. With age, the water content in the discs and their elasticity decrease significantly. The small vessels supplying the disc are obliterated by 20−30 years, and metabolic processes in the disc are carried out by diffusion.load in it falls on the intervertebral discs, consisting of an elastic cushioning nucleus pulposus and a fibrous ring surrounding the nucleus. The disc is connected to the upper and lower vertebrae through cartilaginous hyaline plates. With age, the water content in the discs and their elasticity decrease significantly. The small vessels supplying the disc are obliterated by 20−30 years, and metabolic processes in the disc are carried out by diffusion. in it falls on the intervertebral discs, consisting of an elastic cushioning nucleus pulposus and a fibrous ring surrounding the nucleus. The disc is connected to the upper and lower vertebrae through cartilaginous hyaline plates. With age, the water content in the discs and their elasticity decrease significantly. The small vessels supplying the disc are obliterated by 20−30 years, and metabolic processes in the disc are carried out by diffusion.n it falls on the intervertebral discs, consisting of an elastic cushioning nucleus pulposus and a fibrous ring surrounding the nucleus. The disc is connected to the upper and lower vertebrae through cartilaginous hyaline plates. With age, the water content in the discs and their elasticity decrease significantly. The small vessels supplying the disc are obliterated by 20−30 years, and metabolic processes in the disc are carried out by diffusion.alls on the intervertebral discs, consisting of an elastic cushioning nucleus pulposus and a fibrous ring surrounding the nucleus. The disc is connected to the upper and lower vertebrae through cartilaginous hyaline plates. With age, the water content in the discs and their elasticity decrease significantly. The small vessels supplying the disc are obliterated by 20−30 years, and metabolic processes in the disc are carried out by diffusion.ls on the intervertebral discs, consisting of an elastic cushioning nucleus pulposus and a fibrous ring surrounding the nucleus. The disc is connected to the upper and lower vertebrae through cartilaginous hyaline plates. With age, the water content in the discs and their elasticity decrease significantly. The small vessels supplying the disc are obliterated by 20−30 years, and metabolic processes in the disc are carried out by diffusion. on the intervertebral discs, consisting of an elastic cushioning nucleus pulposus and a fibrous ring surrounding the nucleus. The disc is connected to the upper and lower vertebrae through cartilaginous hyaline plates. With age, the water content in the discs and their elasticity decrease significantly. The small vessels supplying the disc are obliterated by 20−30 years, and metabolic processes in the disc are carried out by diffusion.rtebral discs, consisting of an elastic cushioning nucleus pulposus and a fibrous ring surrounding the nucleus. The disc is connected to the upper and lower vertebrae through cartilaginous hyaline plates. With age, the water content in the discs and their elasticity decrease significantly. The small vessels supplying the disc are obliterated by 20−30 years, and metabolic processes in the disc are carried out by diffusion.ebral discs, consisting of an elastic cushioning nucleus pulposus and a fibrous ring surrounding the nucleus. The disc is connected to the upper and lower vertebrae through cartilaginous hyaline plates. With age, the water content in the discs and their elasticity decrease significantly. The small vessels supplying the disc are obliterated by 20−30 years, and metabolic processes in the disc are carried out by diffusion.ral discs, consisting of an elastic cushioning nucleus pulposus and a fibrous ring surrounding the nucleus. The disc is connected to the upper and lower vertebrae through cartilaginous hyaline plates. With age, the water content in the discs and their elasticity decrease significantly. The small vessels supplying the disc are obliterated by 20−30 years, and metabolic processes in the disc are carried out by diffusion. discs, consisting of an elastic cushioning nucleus pulposus and a fibrous ring surrounding the nucleus. The disc is connected to the upper and lower vertebrae through cartilaginous hyaline plates. With age, the water content in the discs and their elasticity decrease significantly. The small vessels supplying the disc are obliterated by 20−30 years, and metabolic processes in the disc are carried out by diffusion.iscs, consisting of an elastic cushioning nucleus pulposus and a fibrous ring surrounding the nucleus. The disc is connected to the upper and lower vertebrae through cartilaginous hyaline plates. With age, the water content in the discs and their elasticity decrease significantly. The small vessels supplying the disc are obliterated by 20−30 years, and metabolic processes in the disc are carried out by diffusion.cs, consisting of an elastic cushioning nucleus pulposus and a fibrous ring surrounding the nucleus. The disc is connected to the upper and lower vertebrae through cartilaginous hyaline plates. With age, the water content in the discs and their elasticity decrease significantly. The small vessels supplying the disc are obliterated by 20−30 years, and metabolic processes in the disc are carried out by diffusion.ushioning nucleus pulposus and a fibrous ring surrounding the nucleus. The disc is connected to the upper and lower vertebrae through cartilaginous hyaline plates. With age, the water content in the discs and their elasticity decrease significantly. The small vessels supplying the disc are obliterated by 20−30 years, and metabolic processes in the disc are carried out by diffusion.hioning nucleus pulposus and a fibrous ring surrounding the nucleus. The disc is connected to the upper and lower vertebrae through cartilaginous hyaline plates. With age, the water content in the discs and their elasticity decrease significantly. The small vessels supplying the disc are obliterated by 20−30 years, and metabolic processes in the disc are carried out by diffusion.oning nucleus pulposus and a fibrous ring surrounding the nucleus. The disc is connected to the upper and lower vertebrae through cartilaginous hyaline plates. With age, the water content in the discs and their elasticity decrease significantly. The small vessels supplying the disc are obliterated by 20−30 years, and metabolic processes in the disc are carried out by diffusion.ing nucleus pulposus and a fibrous ring surrounding the nucleus. The disc is connected to the upper and lower vertebrae through cartilaginous hyaline plates. With age, the water content in the discs and their elasticity decrease significantly. The small vessels supplying the disc are obliterated by 20−30 years, and metabolic processes in the disc are carried out by diffusion.g nucleus pulposus and a fibrous ring surrounding the nucleus. The disc is connected to the upper and lower vertebrae through cartilaginous hyaline plates. With age, the water content in the discs and their elasticity decrease significantly. The small vessels supplying the disc are obliterated by 20−30 years, and metabolic processes in the disc are carried out by diffusion.nucleus pulposus and a fibrous ring surrounding the nucleus. The disc is connected to the upper and lower vertebrae through cartilaginous hyaline plates. With age, the water content in the discs and their elasticity decrease significantly. The small vessels supplying the disc are obliterated by 20−30 years, and metabolic processes in the disc are carried out by diffusion.us pulposus and a fibrous ring surrounding the nucleus. The disc is connected to the upper and lower vertebrae through cartilaginous hyaline plates. With age, the water content in the discs and their elasticity decrease significantly. The small vessels supplying the disc are obliterated by 20−30 years, and metabolic processes in the disc are carried out by diffusion. pulposus and a fibrous ring surrounding the nucleus. The disc is connected to the upper and lower vertebrae through cartilaginous hyaline plates. With age, the water content in the discs and their elasticity decrease significantly. The small vessels supplying the disc are obliterated by 20−30 years, and metabolic processes in the disc are carried out by diffusion.ulposus and a fibrous ring surrounding the nucleus. The disc is connected to the upper and lower vertebrae through cartilaginous hyaline plates. With age, the water content in the discs and their elasticity decrease significantly. The small vessels supplying the disc are obliterated by 20−30 years, and metabolic processes in the disc are carried out by diffusion.nected to the upper and lower vertebrae through cartilaginous hyaline plates. With age, the water content in the discs and their elasticity decrease significantly. The small vessels supplying the disc are obliterated by 20−30 years, and metabolic processes in the disc are carried out by diffusion.cted to the upper and lower vertebrae through cartilaginous hyaline plates. With age, the water content in the discs and their elasticity decrease significantly. The small vessels supplying the disc are obliterated by 20−30 years, and metabolic processes in the disc are carried out by diffusion.ed to the upper and lower vertebrae through cartilaginous hyaline plates. With age, the water content in the discs and their elasticity decrease significantly. The small vessels supplying the disc are obliterated by 20−30 years, and metabolic processes in the disc are carried out by diffusion.the upper and lower vertebrae through cartilaginous hyaline plates. With age, the water content in the discs and their elasticity decrease significantly. The small vessels supplying the disc are obliterated by 20−30 years, and metabolic processes in the disc are carried out by diffusion.e upper and lower vertebrae through cartilaginous hyaline plates. With age, the water content in the discs and their elasticity decrease significantly. The small vessels supplying the disc are obliterated by 20−30 years, and metabolic processes in the disc are carried out by diffusion.upper and lower vertebrae through cartilaginous hyaline plates. With age, the water content in the discs and their elasticity decrease significantly. The small vessels supplying the disc are obliterated by 20−30 years, and metabolic processes in the disc are carried out by diffusion.ertebrae through cartilaginous hyaline plates. With age, the water content in the discs and their elasticity decrease significantly. The small vessels supplying the disc are obliterated by 20−30 years, and metabolic processes in the disc are carried out by diffusion.tebrae through cartilaginous hyaline plates. With age, the water content in the discs and their elasticity decrease significantly. The small vessels supplying the disc are obliterated by 20−30 years, and metabolic processes in the disc are carried out by diffusion.brae through cartilaginous hyaline plates. With age, the water content in the discs and their elasticity decrease significantly. The small vessels supplying the disc are obliterated by 20−30 years, and metabolic processes in the disc are carried out by diffusion. With age, the water content in the discs and their elasticity decrease significantly. The small vessels supplying the disc are obliterated by 20−30 years, and metabolic processes in the disc are carried out by diffusion.ith age, the water content in the discs and their elasticity decrease significantly. The small vessels supplying the disc are obliterated by 20−30 years, and metabolic processes in the disc are carried out by diffusion.h age, the water content in the discs and their elasticity decrease significantly. The small vessels supplying the disc are obliterated by 20−30 years, and metabolic processes in the disc are carried out by diffusion.age, the water content in the discs and their elasticity decrease significantly. The small vessels supplying the disc are obliterated by 20−30 years, and metabolic processes in the disc are carried out by diffusion.e, the water content in the discs and their elasticity decrease significantly. The small vessels supplying the disc are obliterated by 20−30 years, and metabolic processes in the disc are carried out by diffusion. the water content in the discs and their elasticity decrease significantly. The small vessels supplying the disc are obliterated by 20−30 years, and metabolic processes in the disc are carried out by diffusion.he water content in the discs and their elasticity decrease significantly. The small vessels supplying the disc are obliterated by 20−30 years, and metabolic processes in the disc are carried out by diffusion.ater content in the discs and their elasticity decrease significantly. The small vessels supplying the disc are obliterated by 20−30 years, and metabolic processes in the disc are carried out by diffusion.er content in the discs and their elasticity decrease significantly. The small vessels supplying the disc are obliterated by 20−30 years, and metabolic processes in the disc are carried out by diffusion. content in the discs and their elasticity decrease significantly. The small vessels supplying the disc are obliterated by 20−30 years, and metabolic processes in the disc are carried out by diffusion.tent in the discs and their elasticity decrease significantly. The small vessels supplying the disc are obliterated by 20−30 years, and metabolic processes in the disc are carried out by diffusion.nt in the discs and their elasticity decrease significantly. The small vessels supplying the disc are obliterated by 20−30 years, and metabolic processes in the disc are carried out by diffusion.the discs and their elasticity decrease significantly. The small vessels supplying the disc are obliterated by 20−30 years, and metabolic processes in the disc are carried out by diffusion.e discs and their elasticity decrease significantly. The small vessels supplying the disc are obliterated by 20−30 years, and metabolic processes in the disc are carried out by diffusion.discs and their elasticity decrease significantly. The small vessels supplying the disc are obliterated by 20−30 years, and metabolic processes in the disc are carried out by diffusion. elasticity decrease significantly. The small vessels supplying the disc are obliterated by 20−30 years, and metabolic processes in the disc are carried out by diffusion.lasticity decrease significantly. The small vessels supplying the disc are obliterated by 20−30 years, and metabolic processes in the disc are carried out by diffusion.sticity decrease significantly. The small vessels supplying the disc are obliterated by 20−30 years, and metabolic processes in the disc are carried out by diffusion.city decrease significantly. The small vessels supplying the disc are obliterated by 20−30 years, and metabolic processes in the disc are carried out by diffusion.ty decrease significantly. The small vessels supplying the disc are obliterated by 20−30 years, and metabolic processes in the disc are carried out by diffusion. decrease significantly. The small vessels supplying the disc are obliterated by 20−30 years, and metabolic processes in the disc are carried out by diffusion.mall vessels supplying the disc are obliterated by 20−30 years, and metabolic processes in the disc are carried out by diffusion.ll vessels supplying the disc are obliterated by 20−30 years, and metabolic processes in the disc are carried out by diffusion. vessels supplying the disc are obliterated by 20−30 years, and metabolic processes in the disc are carried out by diffusion.essels supplying the disc are obliterated by 20−30 years, and metabolic processes in the disc are carried out by diffusion.sels supplying the disc are obliterated by 20−30 years, and metabolic processes in the disc are carried out by diffusion.ls supplying the disc are obliterated by 20−30 years, and metabolic processes in the disc are carried out by diffusion.pplying the disc are obliterated by 20−30 years, and metabolic processes in the disc are carried out by diffusion.lying the disc are obliterated by 20−30 years, and metabolic processes in the disc are carried out by diffusion.ing the disc are obliterated by 20−30 years, and metabolic processes in the disc are carried out by diffusion.ocesses in the disc are carried out by diffusion.esses in the disc are carried out by diffusion.ses in the disc are carried out by diffusion. the disc are carried out by diffusion.he disc are carried out by diffusion. disc are carried out by diffusion. out by diffusion.ut by diffusion. by diffusion. classasss="txtthe causes of this pathology have not been definitively clarified. Since degenerative changes of the spine occur more often in residents of developed countries, it is believed that they are based on the peculiarities of nutrition and lifestyle. Thus, among the poorest segments of the population of Southeast Asia, the frequency of pronounced degenerative changes in the spine is only about 30%, while among the affluent residents of the same regions, almost 100% of adults experienced back pain. causes of this pathology have not been definitively clarified. Since degenerative changes of the spine occur more often in residents of developed countries, it is believed that they are based on the peculiarities of nutrition and lifestyle. Thus, among the poorest segments of the population of Southeast Asia, the frequency of pronounced degenerative changes in the spine is only about 30%, while among the affluent residents of the same regions, almost 100% of adults experienced back pain.f pronounced degenerative changes in the spine is only about 30%, while among the affluent residents of the same regions, almost 100% of adults experienced back pain.pronounced degenerative changes in the spine is only about 30%, while among the affluent residents of the same regions, almost 100% of adults experienced back pain.onounced degenerative changes in the spine is only about 30%, while among the affluent residents of the same regions, almost 100% of adults experienced back pain.ed degenerative changes in the spine is only about 30%, while among the affluent residents of the same regions, almost 100% of adults experienced back pain. degenerative changes in the spine is only about 30%, while among the affluent residents of the same regions, almost 100% of adults experienced back pain.egenerative changes in the spine is only about 30%, while among the affluent residents of the same regions, almost 100% of adults experienced back pain.the spine is only about 30%, while among the affluent residents of the same regions, almost 100% of adults experienced back pain.e spine is only about 30%, while among the affluent residents of the same regions, almost 100% of adults experienced back pain.spine is only about 30%, while among the affluent residents of the same regions, almost 100% of adults experienced back pain.ine is only about 30%, while among the affluent residents of the same regions, almost 100% of adults experienced back pain.e is only about 30%, while among the affluent residents of the same regions, almost 100% of adults experienced back pain.is only about 30%, while among the affluent residents of the same regions, almost 100% of adults experienced back pain. only about 30%, while among the affluent residents of the same regions, almost 100% of adults experienced back pain.nly about 30%, while among the affluent residents of the same regions, almost 100% of adults experienced back pain.y about 30%, while among the affluent residents of the same regions, almost 100% of adults experienced back pain.out 30%, while among the affluent residents of the same regions, almost 100% of adults experienced back pain.t 30%, while among the affluent residents of the same regions, almost 100% of adults experienced back pain. while among the affluent residents of the same regions, almost 100% of adults experienced back pain.hile among the affluent residents of the same regions, almost 100% of adults experienced back pain.le among the affluent residents of the same regions, almost 100% of adults experienced back pain.ent residents of the same regions, almost 100% of adults experienced back pain.t residents of the same regions, almost 100% of adults experienced back pain.residents of the same regions, almost 100% of adults experienced back pain.sidents of the same regions, almost 100% of adults experienced back pain.dents of the same regions, almost 100% of adults experienced back pain.s of the same regions, almost 100% of adults experienced back pain.of the same regions, almost 100% of adults experienced back pain. the same regions, almost 100% of adults experienced back pain.he same regions, almost 100% of adults experienced back pain.ame regions, almost 100% of adults experienced back pain.e regions, almost 100% of adults experienced back pain.regions, almost 100% of adults experienced back pain.ons, almost 100% of adults experienced back pain.s, almost 100% of adults experienced back pain.ost 100% of adults experienced back pain.t 100% of adults experienced back pain.100% of adults experienced back pain.experienced back pain.perienced back pain.rienced back pain.nced back pain.ed back pain. back pain.s="txtt"iomechanics of the spine affects the localization and severity of degenerative changes. The normal cervical and lumbar lordoses provide greater pressure on the anterior parts of the disc, and as degenerative changes in the fibrous ring progress, the gelatinous nucleus (also degeneratively altered) begins to shift in the posterior direction. The bulging of the posterior parts of the disc into the spinal canal leads to the displacement of the periosteum and the formation of reactive bone changes in this zone referred to as osteophytes. The X-ray picture of such changes is referred to as spondylosis. If intervertebral joints are involved in the process, spondyloarthrosis is diagnosed.mechanics of the spine affects the localization and severity of degenerative changes. The normal cervical and lumbar lordoses provide greater pressure on the anterior parts of the disc, and as degenerative changes in the fibrous ring progress, the gelatinous nucleus (also degeneratively altered) begins to shift in the posterior direction. The bulging of the posterior parts of the disc into the spinal canal leads to the displacement of the periosteum and the formation of reactive bone changes in this zone referred to as osteophytes. The X-ray picture of such changes is referred to as spondylosis. If intervertebral joints are involved in the process, spondyloarthrosis is diagnosed.chanics of the spine affects the localization and severity of degenerative changes. The normal cervical and lumbar lordoses provide greater pressure on the anterior parts of the disc, and as degenerative changes in the fibrous ring progress, the gelatinous nucleus (also degeneratively altered) begins to shift in the posterior direction. The bulging of the posterior parts of the disc into the spinal canal leads to the displacement of the periosteum and the formation of reactive bone changes in this zone referred to as osteophytes. The X-ray picture of such changes is referred to as spondylosis. If intervertebral joints are involved in the process, spondyloarthrosis is diagnosed.erative changes. The normal cervical and lumbar lordoses provide greater pressure on the anterior parts of the disc, and as degenerative changes in the fibrous ring progress, the gelatinous nucleus (also degeneratively altered) begins to shift in the posterior direction. The bulging of the posterior parts of the disc into the spinal canal leads to the displacement of the periosteum and the formation of reactive bone changes in this zone referred to as osteophytes. The X-ray picture of such changes is referred to as spondylosis. If intervertebral joints are involved in the process, spondyloarthrosis is diagnosed.ative changes. The normal cervical and lumbar lordoses provide greater pressure on the anterior parts of the disc, and as degenerative changes in the fibrous ring progress, the gelatinous nucleus (also degeneratively altered) begins to shift in the posterior direction. The bulging of the posterior parts of the disc into the spinal canal leads to the displacement of the periosteum and the formation of reactive bone changes in this zone referred to as osteophytes. The X-ray picture of such changes is referred to as spondylosis. If intervertebral joints are involved in the process, spondyloarthrosis is diagnosed.ive changes. The normal cervical and lumbar lordoses provide greater pressure on the anterior parts of the disc, and as degenerative changes in the fibrous ring progress, the gelatinous nucleus (also degeneratively altered) begins to shift in the posterior direction. The bulging of the posterior parts of the disc into the spinal canal leads to the displacement of the periosteum and the formation of reactive bone changes in this zone referred to as osteophytes. The X-ray picture of such changes is referred to as spondylosis. If intervertebral joints are involved in the process, spondyloarthrosis is diagnosed.anges. The normal cervical and lumbar lordoses provide greater pressure on the anterior parts of the disc, and as degenerative changes in the fibrous ring progress, the gelatinous nucleus (also degeneratively altered) begins to shift in the posterior direction. The bulging of the posterior parts of the disc into the spinal canal leads to the displacement of the periosteum and the formation of reactive bone changes in this zone referred to as osteophytes. The X-ray picture of such changes is referred to as spondylosis. If intervertebral joints are involved in the process, spondyloarthrosis is diagnosed.ges. The normal cervical and lumbar lordoses provide greater pressure on the anterior parts of the disc, and as degenerative changes in the fibrous ring progress, the gelatinous nucleus (also degeneratively altered) begins to shift in the posterior direction. The bulging of the posterior parts of the disc into the spinal canal leads to the displacement of the periosteum and the formation of reactive bone changes in this zone referred to as osteophytes. The X-ray picture of such changes is referred to as spondylosis. If intervertebral joints are involved in the process, spondyloarthrosis is diagnosed.s. The normal cervical and lumbar lordoses provide greater pressure on the anterior parts of the disc, and as degenerative changes in the fibrous ring progress, the gelatinous nucleus (also degeneratively altered) begins to shift in the posterior direction. The bulging of the posterior parts of the disc into the spinal canal leads to the displacement of the periosteum and the formation of reactive bone changes in this zone referred to as osteophytes. The X-ray picture of such changes is referred to as spondylosis. If intervertebral joints are involved in the process, spondyloarthrosis is diagnosed.vical and lumbar lordoses provide greater pressure on the anterior parts of the disc, and as degenerative changes in the fibrous ring progress, the gelatinous nucleus (also degeneratively altered) begins to shift in the posterior direction. The bulging of the posterior parts of the disc into the spinal canal leads to the displacement of the periosteum and the formation of reactive bone changes in this zone referred to as osteophytes. The X-ray picture of such changes is referred to as spondylosis. If intervertebral joints are involved in the process, spondyloarthrosis is diagnosed.cal and lumbar lordoses provide greater pressure on the anterior parts of the disc, and as degenerative changes in the fibrous ring progress, the gelatinous nucleus (also degeneratively altered) begins to shift in the posterior direction. The bulging of the posterior parts of the disc into the spinal canal leads to the displacement of the periosteum and the formation of reactive bone changes in this zone referred to as osteophytes. The X-ray picture of such changes is referred to as spondylosis. If intervertebral joints are involved in the process, spondyloarthrosis is diagnosed.l and lumbar lordoses provide greater pressure on the anterior parts of the disc, and as degenerative changes in the fibrous ring progress, the gelatinous nucleus (also degeneratively altered) begins to shift in the posterior direction. The bulging of the posterior parts of the disc into the spinal canal leads to the displacement of the periosteum and the formation of reactive bone changes in this zone referred to as osteophytes. The X-ray picture of such changes is referred to as spondylosis. If intervertebral joints are involved in the process, spondyloarthrosis is diagnosed. provide greater pressure on the anterior parts of the disc, and as degenerative changes in the fibrous ring progress, the gelatinous nucleus (also degeneratively altered) begins to shift in the posterior direction. The bulging of the posterior parts of the disc into the spinal canal leads to the displacement of the periosteum and the formation of reactive bone changes in this zone referred to as osteophytes. The X-ray picture of such changes is referred to as spondylosis. If intervertebral joints are involved in the process, spondyloarthrosis is diagnosed.rovide greater pressure on the anterior parts of the disc, and as degenerative changes in the fibrous ring progress, the gelatinous nucleus (also degeneratively altered) begins to shift in the posterior direction. The bulging of the posterior parts of the disc into the spinal canal leads to the displacement of the periosteum and the formation of reactive bone changes in this zone referred to as osteophytes. The X-ray picture of such changes is referred to as spondylosis. If intervertebral joints are involved in the process, spondyloarthrosis is diagnosed.vide greater pressure on the anterior parts of the disc, and as degenerative changes in the fibrous ring progress, the gelatinous nucleus (also degeneratively altered) begins to shift in the posterior direction. The bulging of the posterior parts of the disc into the spinal canal leads to the displacement of the periosteum and the formation of reactive bone changes in this zone referred to as osteophytes. The X-ray picture of such changes is referred to as spondylosis. If intervertebral joints are involved in the process, spondyloarthrosis is diagnosed.de greater pressure on the anterior parts of the disc, and as degenerative changes in the fibrous ring progress, the gelatinous nucleus (also degeneratively altered) begins to shift in the posterior direction. The bulging of the posterior parts of the disc into the spinal canal leads to the displacement of the periosteum and the formation of reactive bone changes in this zone referred to as osteophytes. The X-ray picture of such changes is referred to as spondylosis. If intervertebral joints are involved in the process, spondyloarthrosis is diagnosed. greater pressure on the anterior parts of the disc, and as degenerative changes in the fibrous ring progress, the gelatinous nucleus (also degeneratively altered) begins to shift in the posterior direction. The bulging of the posterior parts of the disc into the spinal canal leads to the displacement of the periosteum and the formation of reactive bone changes in this zone referred to as osteophytes. The X-ray picture of such changes is referred to as spondylosis. If intervertebral joints are involved in the process, spondyloarthrosis is diagnosed.reater pressure on the anterior parts of the disc, and as degenerative changes in the fibrous ring progress, the gelatinous nucleus (also degeneratively altered) begins to shift in the posterior direction. The bulging of the posterior parts of the disc into the spinal canal leads to the displacement of the periosteum and the formation of reactive bone changes in this zone referred to as osteophytes. The X-ray picture of such changes is referred to as spondylosis. If intervertebral joints are involved in the process, spondyloarthrosis is diagnosed.ater pressure on the anterior parts of the disc, and as degenerative changes in the fibrous ring progress, the gelatinous nucleus (also degeneratively altered) begins to shift in the posterior direction. The bulging of the posterior parts of the disc into the spinal canal leads to the displacement of the periosteum and the formation of reactive bone changes in this zone referred to as osteophytes. The X-ray picture of such changes is referred to as spondylosis. If intervertebral joints are involved in the process, spondyloarthrosis is diagnosed.er pressure on the anterior parts of the disc, and as degenerative changes in the fibrous ring progress, the gelatinous nucleus (also degeneratively altered) begins to shift in the posterior direction. The bulging of the posterior parts of the disc into the spinal canal leads to the displacement of the periosteum and the formation of reactive bone changes in this zone referred to as osteophytes. The X-ray picture of such changes is referred to as spondylosis. If intervertebral joints are involved in the process, spondyloarthrosis is diagnosed.essure on the anterior parts of the disc, and as degenerative changes in the fibrous ring progress, the gelatinous nucleus (also degeneratively altered) begins to shift in the posterior direction. The bulging of the posterior parts of the disc into the spinal canal leads to the displacement of the periosteum and the formation of reactive bone changes in this zone referred to as osteophytes. The X-ray picture of such changes is referred to as spondylosis. If intervertebral joints are involved in the process, spondyloarthrosis is diagnosed.sure on the anterior parts of the disc, and as degenerative changes in the fibrous ring progress, the gelatinous nucleus (also degeneratively altered) begins to shift in the posterior direction. The bulging of the posterior parts of the disc into the spinal canal leads to the displacement of the periosteum and the formation of reactive bone changes in this zone referred to as osteophytes. The X-ray picture of such changes is referred to as spondylosis. If intervertebral joints are involved in the process, spondyloarthrosis is diagnosed.re on the anterior parts of the disc, and as degenerative changes in the fibrous ring progress, the gelatinous nucleus (also degeneratively altered) begins to shift in the posterior direction. The bulging of the posterior parts of the disc into the spinal canal leads to the displacement of the periosteum and the formation of reactive bone changes in this zone referred to as osteophytes. The X-ray picture of such changes is referred to as spondylosis. If intervertebral joints are involved in the process, spondyloarthrosis is diagnosed.reactive bone changes in this zone referred to as osteophytes. The X-ray picture of such changes is referred to as spondylosis. If intervertebral joints are involved in the process, spondyloarthrosis is diagnosed.active bone changes in this zone referred to as osteophytes. The X-ray picture of such changes is referred to as spondylosis. If intervertebral joints are involved in the process, spondyloarthrosis is diagnosed.tive bone changes in this zone referred to as osteophytes. The X-ray picture of such changes is referred to as spondylosis. If intervertebral joints are involved in the process, spondyloarthrosis is diagnosed.one changes in this zone referred to as osteophytes. The X-ray picture of such changes is referred to as spondylosis. If intervertebral joints are involved in the process, spondyloarthrosis is diagnosed.e changes in this zone referred to as osteophytes. The X-ray picture of such changes is referred to as spondylosis. If intervertebral joints are involved in the process, spondyloarthrosis is diagnosed.changes in this zone referred to as osteophytes. The X-ray picture of such changes is referred to as spondylosis. If intervertebral joints are involved in the process, spondyloarthrosis is diagnosed.ferred to as osteophytes. The X-ray picture of such changes is referred to as spondylosis. If intervertebral joints are involved in the process, spondyloarthrosis is diagnosed.rred to as osteophytes. The X-ray picture of such changes is referred to as spondylosis. 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If intervertebral joints are involved in the process, spondyloarthrosis is diagnosed.ertebral joints are involved in the process, spondyloarthrosis is diagnosed.tebral joints are involved in the process, spondyloarthrosis is diagnosed.bral joints are involved in the process, spondyloarthrosis is diagnosed.l joints are involved in the process, spondyloarthrosis is diagnosed.joints are involved in the process, spondyloarthrosis is diagnosed.ints are involved in the process, spondyloarthrosis is diagnosed.ss, spondyloarthrosis is diagnosed., spondyloarthrosis is diagnosed.spondyloarthrosis is diagnosed.>spondyloarthrosis is diagnosed.pondyloarthrosis is diagnosed.ndyloarthrosis is diagnosed.arthrosis is diagnosed.throsis is diagnosed.rosis is diagnosed.tudinal connective tissue ligaments course along the anterior and posterior surfaces of the spine, which make the spine structure even more durable. The posterior longitudinal ligament strengthens the middle sections of the surface of the fibrous ring facing the spinal canal, preventing the displacement of the intervertebral disc in this direction. At the same time, the lateral surfaces of the discs, especially in the lumbar region, where the posterior longitudinal ligament narrows, are less durable. Thus, in most cases, the bulging of the cervical or lumbar intervertebral disc occurs in the posterolateral direction (Fig. 13.1).dinal connective tissue ligaments course along the anterior and posterior surfaces of the spine, which make the spine structure even more durable. The posterior longitudinal ligament strengthens the middle sections of the surface of the fibrous ring facing the spinal canal, preventing the displacement of the intervertebral disc in this direction. At the same time, the lateral surfaces of the discs, especially in the lumbar region, where the posterior longitudinal ligament narrows, are less durable. Thus, in most cases, the bulging of the cervical or lumbar intervertebral disc occurs in the posterolateral direction (Fig. 13.1).nal connective tissue ligaments course along the anterior and posterior surfaces of the spine, which make the spine structure even more durable. The posterior longitudinal ligament strengthens the middle sections of the surface of the fibrous ring facing the spinal canal, preventing the displacement of the intervertebral disc in this direction. At the same time, the lateral surfaces of the discs, especially in the lumbar region, where the posterior longitudinal ligament narrows, are less durable. Thus, in most cases, the bulging of the cervical or lumbar intervertebral disc occurs in the posterolateral direction (Fig. 13.1).nnective tissue ligaments course along the anterior and posterior surfaces of the spine, which make the spine structure even more durable. The posterior longitudinal ligament strengthens the middle sections of the surface of the fibrous ring facing the spinal canal, preventing the displacement of the intervertebral disc in this direction. At the same time, the lateral surfaces of the discs, especially in the lumbar region, where the posterior longitudinal ligament narrows, are less durable. Thus, in most cases, the bulging of the cervical or lumbar intervertebral disc occurs in the posterolateral direction (Fig. 13.1).ective tissue ligaments course along the anterior and posterior surfaces of the spine, which make the spine structure even more durable. The posterior longitudinal ligament strengthens the middle sections of the surface of the fibrous ring facing the spinal canal, preventing the displacement of the intervertebral disc in this direction. At the same time, the lateral surfaces of the discs, especially in the lumbar region, where the posterior longitudinal ligament narrows, are less durable. Thus, in most cases, the bulging of the cervical or lumbar intervertebral disc occurs in the posterolateral direction (Fig. 13.1).tive tissue ligaments course along the anterior and posterior surfaces of the spine, which make the spine structure even more durable. The posterior longitudinal ligament strengthens the middle sections of the surface of the fibrous ring facing the spinal canal, preventing the displacement of the intervertebral disc in this direction. At the same time, the lateral surfaces of the discs, especially in the lumbar region, where the posterior longitudinal ligament narrows, are less durable. Thus, in most cases, the bulging of the cervical or lumbar intervertebral disc occurs in the posterolateral direction (Fig. 13.1).ents course along the anterior and posterior surfaces of the spine, which make the spine structure even more durable. The posterior longitudinal ligament strengthens the middle sections of the surface of the fibrous ring facing the spinal canal, preventing the displacement of the intervertebral disc in this direction. At the same time, the lateral surfaces of the discs, especially in the lumbar region, where the posterior longitudinal ligament narrows, are less durable. Thus, in most cases, the bulging of the cervical or lumbar intervertebral disc occurs in the posterolateral direction (Fig. 13.1).ts course along the anterior and posterior surfaces of the spine, which make the spine structure even more durable. The posterior longitudinal ligament strengthens the middle sections of the surface of the fibrous ring facing the spinal canal, preventing the displacement of the intervertebral disc in this direction. At the same time, the lateral surfaces of the discs, especially in the lumbar region, where the posterior longitudinal ligament narrows, are less durable. Thus, in most cases, the bulging of the cervical or lumbar intervertebral disc occurs in the posterolateral direction (Fig. 13.1). course along the anterior and posterior surfaces of the spine, which make the spine structure even more durable. The posterior longitudinal ligament strengthens the middle sections of the surface of the fibrous ring facing the spinal canal, preventing the displacement of the intervertebral disc in this direction. At the same time, the lateral surfaces of the discs, especially in the lumbar region, where the posterior longitudinal ligament narrows, are less durable. Thus, in most cases, the bulging of the cervical or lumbar intervertebral disc occurs in the posterolateral direction (Fig. 13.1).e even more durable. The posterior longitudinal ligament strengthens the middle sections of the surface of the fibrous ring facing the spinal canal, preventing the displacement of the intervertebral disc in this direction. At the same time, the lateral surfaces of the discs, especially in the lumbar region, where the posterior longitudinal ligament narrows, are less durable. Thus, in most cases, the bulging of the cervical or lumbar intervertebral disc occurs in the posterolateral direction (Fig. 13.1).even more durable. The posterior longitudinal ligament strengthens the middle sections of the surface of the fibrous ring facing the spinal canal, preventing the displacement of the intervertebral disc in this direction. At the same time, the lateral surfaces of the discs, especially in the lumbar region, where the posterior longitudinal ligament narrows, are less durable. Thus, in most cases, the bulging of the cervical or lumbar intervertebral disc occurs in the posterolateral direction (Fig. 13.1).en more durable. The posterior longitudinal ligament strengthens the middle sections of the surface of the fibrous ring facing the spinal canal, preventing the displacement of the intervertebral disc in this direction. At the same time, the lateral surfaces of the discs, especially in the lumbar region, where the posterior longitudinal ligament narrows, are less durable. Thus, in most cases, the bulging of the cervical or lumbar intervertebral disc occurs in the posterolateral direction (Fig. 13.1). more durable. The posterior longitudinal ligament strengthens the middle sections of the surface of the fibrous ring facing the spinal canal, preventing the displacement of the intervertebral disc in this direction. At the same time, the lateral surfaces of the discs, especially in the lumbar region, where the posterior longitudinal ligament narrows, are less durable. Thus, in most cases, the bulging of the cervical or lumbar intervertebral disc occurs in the posterolateral direction (Fig. 13.1).ore durable. The posterior longitudinal ligament strengthens the middle sections of the surface of the fibrous ring facing the spinal canal, preventing the displacement of the intervertebral disc in this direction. At the same time, the lateral surfaces of the discs, especially in the lumbar region, where the posterior longitudinal ligament narrows, are less durable. Thus, in most cases, the bulging of the cervical or lumbar intervertebral disc occurs in the posterolateral direction (Fig. 13.1).e durable. The posterior longitudinal ligament strengthens the middle sections of the surface of the fibrous ring facing the spinal canal, preventing the displacement of the intervertebral disc in this direction. At the same time, the lateral surfaces of the discs, especially in the lumbar region, where the posterior longitudinal ligament narrows, are less durable. Thus, in most cases, the bulging of the cervical or lumbar intervertebral disc occurs in the posterolateral direction (Fig. 13.1).durable. The posterior longitudinal ligament strengthens the middle sections of the surface of the fibrous ring facing the spinal canal, preventing the displacement of the intervertebral disc in this direction. At the same time, the lateral surfaces of the discs, especially in the lumbar region, where the posterior longitudinal ligament narrows, are less durable. Thus, in most cases, the bulging of the cervical or lumbar intervertebral disc occurs in the posterolateral direction (Fig. 13.1).ble. The posterior longitudinal ligament strengthens the middle sections of the surface of the fibrous ring facing the spinal canal, preventing the displacement of the intervertebral disc in this direction. At the same time, the lateral surfaces of the discs, especially in the lumbar region, where the posterior longitudinal ligament narrows, are less durable. Thus, in most cases, the bulging of the cervical or lumbar intervertebral disc occurs in the posterolateral direction (Fig. 13.1).e. The posterior longitudinal ligament strengthens the middle sections of the surface of the fibrous ring facing the spinal canal, preventing the displacement of the intervertebral disc in this direction. At the same time, the lateral surfaces of the discs, especially in the lumbar region, where the posterior longitudinal ligament narrows, are less durable. Thus, in most cases, the bulging of the cervical or lumbar intervertebral disc occurs in the posterolateral direction (Fig. 13.1). The posterior longitudinal ligament strengthens the middle sections of the surface of the fibrous ring facing the spinal canal, preventing the displacement of the intervertebral disc in this direction. At the same time, the lateral surfaces of the discs, especially in the lumbar region, where the posterior longitudinal ligament narrows, are less durable. Thus, in most cases, the bulging of the cervical or lumbar intervertebral disc occurs in the posterolateral direction (Fig. 13.1). posterior longitudinal ligament strengthens the middle sections of the surface of the fibrous ring facing the spinal canal, preventing the displacement of the intervertebral disc in this direction. At the same time, the lateral surfaces of the discs, especially in the lumbar region, where the posterior longitudinal ligament narrows, are less durable. Thus, in most cases, the bulging of the cervical or lumbar intervertebral disc occurs in the posterolateral direction (Fig. 13.1).osterior longitudinal ligament strengthens the middle sections of the surface of the fibrous ring facing the spinal canal, preventing the displacement of the intervertebral disc in this direction. At the same time, the lateral surfaces of the discs, especially in the lumbar region, where the posterior longitudinal ligament narrows, are less durable. Thus, in most cases, the bulging of the cervical or lumbar intervertebral disc occurs in the posterolateral direction (Fig. 13.1).or longitudinal ligament strengthens the middle sections of the surface of the fibrous ring facing the spinal canal, preventing the displacement of the intervertebral disc in this direction. At the same time, the lateral surfaces of the discs, especially in the lumbar region, where the posterior longitudinal ligament narrows, are less durable. Thus, in most cases, the bulging of the cervical or lumbar intervertebral disc occurs in the posterolateral direction (Fig. 13.1). longitudinal ligament strengthens the middle sections of the surface of the fibrous ring facing the spinal canal, preventing the displacement of the intervertebral disc in this direction. At the same time, the lateral surfaces of the discs, especially in the lumbar region, where the posterior longitudinal ligament narrows, are less durable. Thus, in most cases, the bulging of the cervical or lumbar intervertebral disc occurs in the posterolateral direction (Fig. 13.1).ongitudinal ligament strengthens the middle sections of the surface of the fibrous ring facing the spinal canal, preventing the displacement of the intervertebral disc in this direction. At the same time, the lateral surfaces of the discs, especially in the lumbar region, where the posterior longitudinal ligament narrows, are less durable. Thus, in most cases, the bulging of the cervical or lumbar intervertebral disc occurs in the posterolateral direction (Fig. 13.1).ament strengthens the middle sections of the surface of the fibrous ring facing the spinal canal, preventing the displacement of the intervertebral disc in this direction. At the same time, the lateral surfaces of the discs, especially in the lumbar region, where the posterior longitudinal ligament narrows, are less durable. Thus, in most cases, the bulging of the cervical or lumbar intervertebral disc occurs in the posterolateral direction (Fig. 13.1).ent strengthens the middle sections of the surface of the fibrous ring facing the spinal canal, preventing the displacement of the intervertebral disc in this direction. At the same time, the lateral surfaces of the discs, especially in the lumbar region, where the posterior longitudinal ligament narrows, are less durable. Thus, in most cases, the bulging of the cervical or lumbar intervertebral disc occurs in the posterolateral direction (Fig. 13.1).t strengthens the middle sections of the surface of the fibrous ring facing the spinal canal, preventing the displacement of the intervertebral disc in this direction. At the same time, the lateral surfaces of the discs, especially in the lumbar region, where the posterior longitudinal ligament narrows, are less durable. Thus, in most cases, the bulging of the cervical or lumbar intervertebral disc occurs in the posterolateral direction (Fig. 13.1).trengthens the middle sections of the surface of the fibrous ring facing the spinal canal, preventing the displacement of the intervertebral disc in this direction. At the same time, the lateral surfaces of the discs, especially in the lumbar region, where the posterior longitudinal ligament narrows, are less durable. Thus, in most cases, the bulging of the cervical or lumbar intervertebral disc occurs in the posterolateral direction (Fig. 13.1).engthens the middle sections of the surface of the fibrous ring facing the spinal canal, preventing the displacement of the intervertebral disc in this direction. At the same time, the lateral surfaces of the discs, especially in the lumbar region, where the posterior longitudinal ligament narrows, are less durable. Thus, in most cases, the bulging of the cervical or lumbar intervertebral disc occurs in the posterolateral direction (Fig. 13.1).gthens the middle sections of the surface of the fibrous ring facing the spinal canal, preventing the displacement of the intervertebral disc in this direction. At the same time, the lateral surfaces of the discs, especially in the lumbar region, where the posterior longitudinal ligament narrows, are less durable. Thus, in most cases, the bulging of the cervical or lumbar intervertebral disc occurs in the posterolateral direction (Fig. 13.1).the surface of the fibrous ring facing the spinal canal, preventing the displacement of the intervertebral disc in this direction. At the same time, the lateral surfaces of the discs, especially in the lumbar region, where the posterior longitudinal ligament narrows, are less durable. Thus, in most cases, the bulging of the cervical or lumbar intervertebral disc occurs in the posterolateral direction (Fig. 13.1).e surface of the fibrous ring facing the spinal canal, preventing the displacement of the intervertebral disc in this direction. At the same time, the lateral surfaces of the discs, especially in the lumbar region, where the posterior longitudinal ligament narrows, are less durable. Thus, in most cases, the bulging of the cervical or lumbar intervertebral disc occurs in the posterolateral direction (Fig. 13.1).surface of the fibrous ring facing the spinal canal, preventing the displacement of the intervertebral disc in this direction. At the same time, the lateral surfaces of the discs, especially in the lumbar region, where the posterior longitudinal ligament narrows, are less durable. Thus, in most cases, the bulging of the cervical or lumbar intervertebral disc occurs in the posterolateral direction (Fig. 13.1).rface of the fibrous ring facing the spinal canal, preventing the displacement of the intervertebral disc in this direction. At the same time, the lateral surfaces of the discs, especially in the lumbar region, where the posterior longitudinal ligament narrows, are less durable. Thus, in most cases, the bulging of the cervical or lumbar intervertebral disc occurs in the posterolateral direction (Fig. 13.1).ace of the fibrous ring facing the spinal canal, preventing the displacement of the intervertebral disc in this direction. At the same time, the lateral surfaces of the discs, especially in the lumbar region, where the posterior longitudinal ligament narrows, are less durable. Thus, in most cases, the bulging of the cervical or lumbar intervertebral disc occurs in the posterolateral direction (Fig. 13.1).e of the fibrous ring facing the spinal canal, preventing the displacement of the intervertebral disc in this direction. At the same time, the lateral surfaces of the discs, especially in the lumbar region, where the posterior longitudinal ligament narrows, are less durable. Thus, in most cases, the bulging of the cervical or lumbar intervertebral disc occurs in the posterolateral direction (Fig. 13.1).the fibrous ring facing the spinal canal, preventing the displacement of the intervertebral disc in this direction. At the same time, the lateral surfaces of the discs, especially in the lumbar region, where the posterior longitudinal ligament narrows, are less durable. Thus, in most cases, the bulging of the cervical or lumbar intervertebral disc occurs in the posterolateral direction (Fig. 13.1).e fibrous ring facing the spinal canal, preventing the displacement of the intervertebral disc in this direction. At the same time, the lateral surfaces of the discs, especially in the lumbar region, where the posterior longitudinal ligament narrows, are less durable. Thus, in most cases, the bulging of the cervical or lumbar intervertebral disc occurs in the posterolateral direction (Fig. 13.1).fibrous ring facing the spinal canal, preventing the displacement of the intervertebral disc in this direction. At the same time, the lateral surfaces of the discs, especially in the lumbar region, where the posterior longitudinal ligament narrows, are less durable. Thus, in most cases, the bulging of the cervical or lumbar intervertebral disc occurs in the posterolateral direction (Fig. 13.1).f the intervertebral disc in this direction. At the same time, the lateral surfaces of the discs, especially in the lumbar region, where the posterior longitudinal ligament narrows, are less durable. Thus, in most cases, the bulging of the cervical or lumbar intervertebral disc occurs in the posterolateral direction (Fig. 13.1).the intervertebral disc in this direction. At the same time, the lateral surfaces of the discs, especially in the lumbar region, where the posterior longitudinal ligament narrows, are less durable. Thus, in most cases, the bulging of the cervical or lumbar intervertebral disc occurs in the posterolateral direction (Fig. 13.1).e intervertebral disc in this direction. At the same time, the lateral surfaces of the discs, especially in the lumbar region, where the posterior longitudinal ligament narrows, are less durable. Thus, in most cases, the bulging of the cervical or lumbar intervertebral disc occurs in the posterolateral direction (Fig. 13.1).rvertebral disc in this direction. At the same time, the lateral surfaces of the discs, especially in the lumbar region, where the posterior longitudinal ligament narrows, are less durable. Thus, in most cases, the bulging of the cervical or lumbar intervertebral disc occurs in the posterolateral direction (Fig. 13.1).ertebral disc in this direction. At the same time, the lateral surfaces of the discs, especially in the lumbar region, where the posterior longitudinal ligament narrows, are less durable. Thus, in most cases, the bulging of the cervical or lumbar intervertebral disc occurs in the posterolateral direction (Fig. 13.1).tebral disc in this direction. At the same time, the lateral surfaces of the discs, especially in the lumbar region, where the posterior longitudinal ligament narrows, are less durable. Thus, in most cases, the bulging of the cervical or lumbar intervertebral disc occurs in the posterolateral direction (Fig. 13.1).is direction. At the same time, the lateral surfaces of the discs, especially in the lumbar region, where the posterior longitudinal ligament narrows, are less durable. Thus, in most cases, the bulging of the cervical or lumbar intervertebral disc occurs in the posterolateral direction (Fig. 13.1). direction. At the same time, the lateral surfaces of the discs, especially in the lumbar region, where the posterior longitudinal ligament narrows, are less durable. Thus, in most cases, the bulging of the cervical or lumbar intervertebral disc occurs in the posterolateral direction (Fig. 13.1).irection. At the same time, the lateral surfaces of the discs, especially in the lumbar region, where the posterior longitudinal ligament narrows, are less durable. Thus, in most cases, the bulging of the cervical or lumbar intervertebral disc occurs in the posterolateral direction (Fig. 13.1).the discs, especially in the lumbar region, where the posterior longitudinal ligament narrows, are less durable. Thus, in most cases, the bulging of the cervical or lumbar intervertebral disc occurs in the posterolateral direction (Fig. 13.1).e discs, especially in the lumbar region, where the posterior longitudinal ligament narrows, are less durable. Thus, in most cases, the bulging of the cervical or lumbar intervertebral disc occurs in the posterolateral direction (Fig. 13.1).discs, especially in the lumbar region, where the posterior longitudinal ligament narrows, are less durable. Thus, in most cases, the bulging of the cervical or lumbar intervertebral disc occurs in the posterolateral direction (Fig. 13.1).scs, especially in the lumbar region, where the posterior longitudinal ligament narrows, are less durable. Thus, in most cases, the bulging of the cervical or lumbar intervertebral disc occurs in the posterolateral direction (Fig. 13.1).s, especially in the lumbar region, where the posterior longitudinal ligament narrows, are less durable. 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Thus, in most cases, the bulging of the cervical or lumbar intervertebral disc occurs in the posterolateral direction (Fig. 13.1).erior longitudinal ligament narrows, are less durable. Thus, in most cases, the bulging of the cervical or lumbar intervertebral disc occurs in the posterolateral direction (Fig. 13.1).ior longitudinal ligament narrows, are less durable. Thus, in most cases, the bulging of the cervical or lumbar intervertebral disc occurs in the posterolateral direction (Fig. 13.1).r longitudinal ligament narrows, are less durable. Thus, in most cases, the bulging of the cervical or lumbar intervertebral disc occurs in the posterolateral direction (Fig. 13.1).s, are less durable. Thus, in most cases, the bulging of the cervical or lumbar intervertebral disc occurs in the posterolateral direction (Fig. 13.1). are less durable. Thus, in most cases, the bulging of the cervical or lumbar intervertebral disc occurs in the posterolateral direction (Fig. 13.1).re less durable. 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#c0c0c0;cursor:hand;">73726-0015,POPUP-Xz17-img214.png,567,567)');}return false;}" style="padding:3 3 3 3px;border:1px dotted #c0c0c0;cursor:hand;">726-0015,POPUP-Xz17-img214.png,567,567)');}return false;}" style="padding:3 3 3 3px;border:1px dotted #c0c0c0;cursor:hand;">0015,POPUP-Xz17-img214.png,567,567)');}return false;}" style="padding:3 3 3 3px;border:1px dotted #c0c0c0;cursor:hand;">15,POPUP-Xz17-img214.png,567,567)');}return false;}" style="padding:3 3 3 3px;border:1px dotted #c0c0c0;cursor:hand;">UP-Xz17-img214.png,567,567)');}return false;}" style="padding:3 3 3 3px;border:1px dotted #c0c0c0;cursor:hand;">-Xz17-img214.png,567,567)');}return false;}" style="padding:3 3 3 3px;border:1px dotted #c0c0c0;cursor:hand;">z17-img214.png,567,567)');}return false;}" style="padding:3 3 3 3px;border:1px dotted #c0c0c0;cursor:hand;">567,567)');}return false;}" style="padding:3 3 3 3px;border:1px dotted #c0c0c0;cursor:hand;">7,567)');}return false;}" style="padding:3 3 3 3px;border:1px dotted #c0c0c0;cursor:hand;">567)');}return false;}" style="padding:3 3 3 3px;border:1px dotted #c0c0c0;cursor:hand;">)');}return false;}" style="padding:3 3 3 3px;border:1px dotted #c0c0c0;cursor:hand;">);}return false;}" style="padding:3 3 3 3px;border:1px dotted #c0c0c0;cursor:hand;">}return false;}" style="padding:3 3 3 3px;border:1px dotted #c0c0c0;cursor:hand;">g:3 3 3 3px;border:1px dotted #c0c0c0;cursor:hand;">3 3 3 3px;border:1px dotted #c0c0c0;cursor:hand;">3 3 3px;border:1px dotted #c0c0c0;cursor:hand;">3 3px;border:1px dotted #c0c0c0;cursor:hand;">3px;border:1px dotted #c0c0c0;cursor:hand;">x;border:1px dotted #c0c0c0;cursor:hand;">der:1px dotted #c0c0c0;cursor:hand;">r:1px dotted #c0c0c0;cursor:hand;">1px dotted #c0c0c0;cursor:hand;">b>Fig. 13.1. The mechanism of formation of a herniated intervertebral disc: 1 — fibrous ring; 2 — gelatinous nucleus; 3 — herniated discFig. 13.1. The mechanism of formation of a herniated intervertebral disc: 1 — fibrous ring; 2 — gelatinous nucleus; 3 — herniated discg. 13.1. The mechanism of formation of a herniated intervertebral disc: 1 — fibrous ring; 2 — gelatinous nucleus; 3 — herniated disc1. The mechanism of formation of a herniated intervertebral disc: 1 — fibrous ring; 2 — gelatinous nucleus; 3 — herniated disc The mechanism of formation of a herniated intervertebral disc: 1 — fibrous ring; 2 — gelatinous nucleus; 3 — herniated disc/b>The mechanism of formation of a herniated intervertebral disc: 1 — fibrous ring; 2 — gelatinous nucleus; 3 — herniated discof formation of a herniated intervertebral disc: 1 — fibrous ring; 2 — gelatinous nucleus; 3 — herniated disc formation of a herniated intervertebral disc: 1 — fibrous ring; 2 — gelatinous nucleus; 3 — herniated discormation of a herniated intervertebral disc: 1 — fibrous ring; 2 — gelatinous nucleus; 3 — herniated discal disc: 1 — fibrous ring; 2 — gelatinous nucleus; 3 — herniated disc disc: 1 — fibrous ring; 2 — gelatinous nucleus; 3 — herniated discisc: 1 — fibrous ring; 2 — gelatinous nucleus; 3 — herniated discc: 1 — fibrous ring; 2 — gelatinous nucleus; 3 — herniated disc 1 — fibrous ring; 2 — gelatinous nucleus; 3 — herniated disc — fibrous ring; 2 — gelatinous nucleus; 3 — herniated discmdash; fibrous ring; 2 — gelatinous nucleus; 3 — herniated disch; fibrous ring; 2 — gelatinous nucleus; 3 — herniated disc fibrous ring; 2 — gelatinous nucleus; 3 — herniated discibrous ring; 2 — gelatinous nucleus; 3 — herniated discus ring; 2 — gelatinous nucleus; 3 — herniated disc ring; 2 — gelatinous nucleus; 3 — herniated disc 2 — gelatinous nucleus; 3 — herniated disc — gelatinous nucleus; 3 — herniated discmdash; gelatinous nucleus; 3 — herniated discus nucleus; 3 — herniated disc nucleus; 3 — herniated discucleus; 3 — herniated disceus; 3 — herniated discs; 3 — herniated disc 3 — herniated discp>

At stage II, fragments (sequesters) of the nucleus pulposus are squeezed into the spinal canal through cracks in the fibrous ring. The thickness of the intervertebral disc decreases (“disc settlement”).

At stage III (final), the disc is completely replaced by coarse-fibrous connective tissue, which is often ossified.

re is a decrease in the concentration and a change in the properties of glycosaminoglycans and collagen of the nucleus pulposus, which leads to a decrease in intradiscal pressure and a decrease in the water content in the nucleus; at the same time, a fibrous process develops, as a result, the cushioning properties of the nucleus decrease, and with any significant load, microtraumatization of the fibrous ring occurs with the appearance of cracks in it.

At stage II, fragments (sequesters) of the nucleus pulposus are squeezed into the spinal canal through cracks in the fibrous ring. The thickness of the intervertebral disc decreases (“disc settlement”).

At stage III (final), the disc is completely replaced by coarse-fibrous connective tissue, which is often ossified.

is a decrease in the concentration and a change in the properties of glycosaminoglycans and collagen of the nucleus pulposus, which leads to a decrease in intradiscal pressure and a decrease in the water content in the nucleus; at the same time, a fibrous process develops, as a result, the cushioning properties of the nucleus decrease, and with any significant load, microtraumatization of the fibrous ring occurs with the appearance of cracks in it.

At stage II, fragments (sequesters) of the nucleus pulposus are squeezed into the spinal canal through cracks in the fibrous ring. The thickness of the intervertebral disc decreases (“disc settlement”).

At stage III (final), the disc is completely replaced by coarse-fibrous connective tissue, which is often ossified.

decrease in the concentration and a change in the properties of glycosaminoglycans and collagen of the nucleus pulposus, which leads to a decrease in intradiscal pressure and a decrease in the water content in the nucleus; at the same time, a fibrous process develops, as a result, the cushioning properties of the nucleus decrease, and with any significant load, microtraumatization of the fibrous ring occurs with the appearance of cracks in it.

At stage II, fragments (sequesters) of the nucleus pulposus are squeezed into the spinal canal through cracks in the fibrous ring. The thickness of the intervertebral disc decreases (“disc settlement”).

At stage III (final), the disc is completely replaced by coarse-fibrous connective tissue, which is often ossified.

crease in the concentration and a change in the properties of glycosaminoglycans and collagen of the nucleus pulposus, which leads to a decrease in intradiscal pressure and a decrease in the water content in the nucleus; at the same time, a fibrous process develops, as a result, the cushioning properties of the nucleus decrease, and with any significant load, microtraumatization of the fibrous ring occurs with the appearance of cracks in it.

At stage II, fragments (sequesters) of the nucleus pulposus are squeezed into the spinal canal through cracks in the fibrous ring. The thickness of the intervertebral disc decreases (“disc settlement”).

At stage III (final), the disc is completely replaced by coarse-fibrous connective tissue, which is often ossified.

ease in the concentration and a change in the properties of glycosaminoglycans and collagen of the nucleus pulposus, which leads to a decrease in intradiscal pressure and a decrease in the water content in the nucleus; at the same time, a fibrous process develops, as a result, the cushioning properties of the nucleus decrease, and with any significant load, microtraumatization of the fibrous ring occurs with the appearance of cracks in it.

At stage II, fragments (sequesters) of the nucleus pulposus are squeezed into the spinal canal through cracks in the fibrous ring. The thickness of the intervertebral disc decreases (“disc settlement”).

At stage III (final), the disc is completely replaced by coarse-fibrous connective tissue, which is often ossified.

n and a change in the properties of glycosaminoglycans and collagen of the nucleus pulposus, which leads to a decrease in intradiscal pressure and a decrease in the water content in the nucleus; at the same time, a fibrous process develops, as a result, the cushioning properties of the nucleus decrease, and with any significant load, microtraumatization of the fibrous ring occurs with the appearance of cracks in it.

At stage II, fragments (sequesters) of the nucleus pulposus are squeezed into the spinal canal through cracks in the fibrous ring. The thickness of the intervertebral disc decreases (“disc settlement”).

At stage III (final), the disc is completely replaced by coarse-fibrous connective tissue, which is often ossified.

and a change in the properties of glycosaminoglycans and collagen of the nucleus pulposus, which leads to a decrease in intradiscal pressure and a decrease in the water content in the nucleus; at the same time, a fibrous process develops, as a result, the cushioning properties of the nucleus decrease, and with any significant load, microtraumatization of the fibrous ring occurs with the appearance of cracks in it.

At stage II, fragments (sequesters) of the nucleus pulposus are squeezed into the spinal canal through cracks in the fibrous ring. The thickness of the intervertebral disc decreases (“disc settlement”).

At stage III (final), the disc is completely replaced by coarse-fibrous connective tissue, which is often ossified.

d a change in the properties of glycosaminoglycans and collagen of the nucleus pulposus, which leads to a decrease in intradiscal pressure and a decrease in the water content in the nucleus; at the same time, a fibrous process develops, as a result, the cushioning properties of the nucleus decrease, and with any significant load, microtraumatization of the fibrous ring occurs with the appearance of cracks in it.

At stage II, fragments (sequesters) of the nucleus pulposus are squeezed into the spinal canal through cracks in the fibrous ring. The thickness of the intervertebral disc decreases (“disc settlement”).

At stage III (final), the disc is completely replaced by coarse-fibrous connective tissue, which is often ossified.

a change in the properties of glycosaminoglycans and collagen of the nucleus pulposus, which leads to a decrease in intradiscal pressure and a decrease in the water content in the nucleus; at the same time, a fibrous process develops, as a result, the cushioning properties of the nucleus decrease, and with any significant load, microtraumatization of the fibrous ring occurs with the appearance of cracks in it.

At stage II, fragments (sequesters) of the nucleus pulposus are squeezed into the spinal canal through cracks in the fibrous ring. The thickness of the intervertebral disc decreases (“disc settlement”).

At stage III (final), the disc is completely replaced by coarse-fibrous connective tissue, which is often ossified.

change in the properties of glycosaminoglycans and collagen of the nucleus pulposus, which leads to a decrease in intradiscal pressure and a decrease in the water content in the nucleus; at the same time, a fibrous process develops, as a result, the cushioning properties of the nucleus decrease, and with any significant load, microtraumatization of the fibrous ring occurs with the appearance of cracks in it.

At stage II, fragments (sequesters) of the nucleus pulposus are squeezed into the spinal canal through cracks in the fibrous ring. The thickness of the intervertebral disc decreases (“disc settlement”).

At stage III (final), the disc is completely replaced by coarse-fibrous connective tissue, which is often ossified.

ange in the properties of glycosaminoglycans and collagen of the nucleus pulposus, which leads to a decrease in intradiscal pressure and a decrease in the water content in the nucleus; at the same time, a fibrous process develops, as a result, the cushioning properties of the nucleus decrease, and with any significant load, microtraumatization of the fibrous ring occurs with the appearance of cracks in it.

At stage II, fragments (sequesters) of the nucleus pulposus are squeezed into the spinal canal through cracks in the fibrous ring. The thickness of the intervertebral disc decreases (“disc settlement”).

At stage III (final), the disc is completely replaced by coarse-fibrous connective tissue, which is often ossified.

ge in the properties of glycosaminoglycans and collagen of the nucleus pulposus, which leads to a decrease in intradiscal pressure and a decrease in the water content in the nucleus; at the same time, a fibrous process develops, as a result, the cushioning properties of the nucleus decrease, and with any significant load, microtraumatization of the fibrous ring occurs with the appearance of cracks in it.

At stage II, fragments (sequesters) of the nucleus pulposus are squeezed into the spinal canal through cracks in the fibrous ring. The thickness of the intervertebral disc decreases (“disc settlement”).

At stage III (final), the disc is completely replaced by coarse-fibrous connective tissue, which is often ossified.

in the properties of glycosaminoglycans and collagen of the nucleus pulposus, which leads to a decrease in intradiscal pressure and a decrease in the water content in the nucleus; at the same time, a fibrous process develops, as a result, the cushioning properties of the nucleus decrease, and with any significant load, microtraumatization of the fibrous ring occurs with the appearance of cracks in it.

At stage II, fragments (sequesters) of the nucleus pulposus are squeezed into the spinal canal through cracks in the fibrous ring. The thickness of the intervertebral disc decreases (“disc settlement”).

At stage III (final), the disc is completely replaced by coarse-fibrous connective tissue, which is often ossified.

n the properties of glycosaminoglycans and collagen of the nucleus pulposus, which leads to a decrease in intradiscal pressure and a decrease in the water content in the nucleus; at the same time, a fibrous process develops, as a result, the cushioning properties of the nucleus decrease, and with any significant load, microtraumatization of the fibrous ring occurs with the appearance of cracks in it.

At stage II, fragments (sequesters) of the nucleus pulposus are squeezed into the spinal canal through cracks in the fibrous ring. The thickness of the intervertebral disc decreases (“disc settlement”).

At stage III (final), the disc is completely replaced by coarse-fibrous connective tissue, which is often ossified.

e properties of glycosaminoglycans and collagen of the nucleus pulposus, which leads to a decrease in intradiscal pressure and a decrease in the water content in the nucleus; at the same time, a fibrous process develops, as a result, the cushioning properties of the nucleus decrease, and with any significant load, microtraumatization of the fibrous ring occurs with the appearance of cracks in it.

At stage II, fragments (sequesters) of the nucleus pulposus are squeezed into the spinal canal through cracks in the fibrous ring. The thickness of the intervertebral disc decreases (“disc settlement”).

At stage III (final), the disc is completely replaced by coarse-fibrous connective tissue, which is often ossified.

properties of glycosaminoglycans and collagen of the nucleus pulposus, which leads to a decrease in intradiscal pressure and a decrease in the water content in the nucleus; at the same time, a fibrous process develops, as a result, the cushioning properties of the nucleus decrease, and with any significant load, microtraumatization of the fibrous ring occurs with the appearance of cracks in it.

At stage II, fragments (sequesters) of the nucleus pulposus are squeezed into the spinal canal through cracks in the fibrous ring. The thickness of the intervertebral disc decreases (“disc settlement”).

At stage III (final), the disc is completely replaced by coarse-fibrous connective tissue, which is often ossified.

ties of glycosaminoglycans and collagen of the nucleus pulposus, which leads to a decrease in intradiscal pressure and a decrease in the water content in the nucleus; at the same time, a fibrous process develops, as a result, the cushioning properties of the nucleus decrease, and with any significant load, microtraumatization of the fibrous ring occurs with the appearance of cracks in it.

At stage II, fragments (sequesters) of the nucleus pulposus are squeezed into the spinal canal through cracks in the fibrous ring. The thickness of the intervertebral disc decreases (“disc settlement”).

At stage III (final), the disc is completely replaced by coarse-fibrous connective tissue, which is often ossified.

es of glycosaminoglycans and collagen of the nucleus pulposus, which leads to a decrease in intradiscal pressure and a decrease in the water content in the nucleus; at the same time, a fibrous process develops, as a result, the cushioning properties of the nucleus decrease, and with any significant load, microtraumatization of the fibrous ring occurs with the appearance of cracks in it.

At stage II, fragments (sequesters) of the nucleus pulposus are squeezed into the spinal canal through cracks in the fibrous ring. The thickness of the intervertebral disc decreases (“disc settlement”).

At stage III (final), the disc is completely replaced by coarse-fibrous connective tissue, which is often ossified.

of glycosaminoglycans and collagen of the nucleus pulposus, which leads to a decrease in intradiscal pressure and a decrease in the water content in the nucleus; at the same time, a fibrous process develops, as a result, the cushioning properties of the nucleus decrease, and with any significant load, microtraumatization of the fibrous ring occurs with the appearance of cracks in it.

At stage II, fragments (sequesters) of the nucleus pulposus are squeezed into the spinal canal through cracks in the fibrous ring. The thickness of the intervertebral disc decreases (“disc settlement”).

At stage III (final), the disc is completely replaced by coarse-fibrous connective tissue, which is often ossified.

cans and collagen of the nucleus pulposus, which leads to a decrease in intradiscal pressure and a decrease in the water content in the nucleus; at the same time, a fibrous process develops, as a result, the cushioning properties of the nucleus decrease, and with any significant load, microtraumatization of the fibrous ring occurs with the appearance of cracks in it.

At stage II, fragments (sequesters) of the nucleus pulposus are squeezed into the spinal canal through cracks in the fibrous ring. The thickness of the intervertebral disc decreases (“disc settlement”).

At stage III (final), the disc is completely replaced by coarse-fibrous connective tissue, which is often ossified.

ns and collagen of the nucleus pulposus, which leads to a decrease in intradiscal pressure and a decrease in the water content in the nucleus; at the same time, a fibrous process develops, as a result, the cushioning properties of the nucleus decrease, and with any significant load, microtraumatization of the fibrous ring occurs with the appearance of cracks in it.

At stage II, fragments (sequesters) of the nucleus pulposus are squeezed into the spinal canal through cracks in the fibrous ring. The thickness of the intervertebral disc decreases (“disc settlement”).

At stage III (final), the disc is completely replaced by coarse-fibrous connective tissue, which is often ossified.

and collagen of the nucleus pulposus, which leads to a decrease in intradiscal pressure and a decrease in the water content in the nucleus; at the same time, a fibrous process develops, as a result, the cushioning properties of the nucleus decrease, and with any significant load, microtraumatization of the fibrous ring occurs with the appearance of cracks in it.

At stage II, fragments (sequesters) of the nucleus pulposus are squeezed into the spinal canal through cracks in the fibrous ring. The thickness of the intervertebral disc decreases (“disc settlement”).

At stage III (final), the disc is completely replaced by coarse-fibrous connective tissue, which is often ossified.

nd collagen of the nucleus pulposus, which leads to a decrease in intradiscal pressure and a decrease in the water content in the nucleus; at the same time, a fibrous process develops, as a result, the cushioning properties of the nucleus decrease, and with any significant load, microtraumatization of the fibrous ring occurs with the appearance of cracks in it.

At stage II, fragments (sequesters) of the nucleus pulposus are squeezed into the spinal canal through cracks in the fibrous ring. The thickness of the intervertebral disc decreases (“disc settlement”).

At stage III (final), the disc is completely replaced by coarse-fibrous connective tissue, which is often ossified.

ollagen of the nucleus pulposus, which leads to a decrease in intradiscal pressure and a decrease in the water content in the nucleus; at the same time, a fibrous process develops, as a result, the cushioning properties of the nucleus decrease, and with any significant load, microtraumatization of the fibrous ring occurs with the appearance of cracks in it.

At stage II, fragments (sequesters) of the nucleus pulposus are squeezed into the spinal canal through cracks in the fibrous ring. The thickness of the intervertebral disc decreases (“disc settlement”).

At stage III (final), the disc is completely replaced by coarse-fibrous connective tissue, which is often ossified.

lagen of the nucleus pulposus, which leads to a decrease in intradiscal pressure and a decrease in the water content in the nucleus; at the same time, a fibrous process develops, as a result, the cushioning properties of the nucleus decrease, and with any significant load, microtraumatization of the fibrous ring occurs with the appearance of cracks in it.

At stage II, fragments (sequesters) of the nucleus pulposus are squeezed into the spinal canal through cracks in the fibrous ring. The thickness of the intervertebral disc decreases (“disc settlement”).

At stage III (final), the disc is completely replaced by coarse-fibrous connective tissue, which is often ossified.

of the nucleus pulposus, which leads to a decrease in intradiscal pressure and a decrease in the water content in the nucleus; at the same time, a fibrous process develops, as a result, the cushioning properties of the nucleus decrease, and with any significant load, microtraumatization of the fibrous ring occurs with the appearance of cracks in it.

At stage II, fragments (sequesters) of the nucleus pulposus are squeezed into the spinal canal through cracks in the fibrous ring. The thickness of the intervertebral disc decreases (“disc settlement”).

At stage III (final), the disc is completely replaced by coarse-fibrous connective tissue, which is often ossified.

the nucleus pulposus, which leads to a decrease in intradiscal pressure and a decrease in the water content in the nucleus; at the same time, a fibrous process develops, as a result, the cushioning properties of the nucleus decrease, and with any significant load, microtraumatization of the fibrous ring occurs with the appearance of cracks in it.

At stage II, fragments (sequesters) of the nucleus pulposus are squeezed into the spinal canal through cracks in the fibrous ring. The thickness of the intervertebral disc decreases (“disc settlement”).

At stage III (final), the disc is completely replaced by coarse-fibrous connective tissue, which is often ossified.

he nucleus pulposus, which leads to a decrease in intradiscal pressure and a decrease in the water content in the nucleus; at the same time, a fibrous process develops, as a result, the cushioning properties of the nucleus decrease, and with any significant load, microtraumatization of the fibrous ring occurs with the appearance of cracks in it.

At stage II, fragments (sequesters) of the nucleus pulposus are squeezed into the spinal canal through cracks in the fibrous ring. The thickness of the intervertebral disc decreases (“disc settlement”).

At stage III (final), the disc is completely replaced by coarse-fibrous connective tissue, which is often ossified.

ch leads to a decrease in intradiscal pressure and a decrease in the water content in the nucleus; at the same time, a fibrous process develops, as a result, the cushioning properties of the nucleus decrease, and with any significant load, microtraumatization of the fibrous ring occurs with the appearance of cracks in it.

At stage II, fragments (sequesters) of the nucleus pulposus are squeezed into the spinal canal through cracks in the fibrous ring. The thickness of the intervertebral disc decreases (“disc settlement”).

At stage III (final), the disc is completely replaced by coarse-fibrous connective tissue, which is often ossified.

leads to a decrease in intradiscal pressure and a decrease in the water content in the nucleus; at the same time, a fibrous process develops, as a result, the cushioning properties of the nucleus decrease, and with any significant load, microtraumatization of the fibrous ring occurs with the appearance of cracks in it.

At stage II, fragments (sequesters) of the nucleus pulposus are squeezed into the spinal canal through cracks in the fibrous ring. The thickness of the intervertebral disc decreases (“disc settlement”).

At stage III (final), the disc is completely replaced by coarse-fibrous connective tissue, which is often ossified.

eads to a decrease in intradiscal pressure and a decrease in the water content in the nucleus; at the same time, a fibrous process develops, as a result, the cushioning properties of the nucleus decrease, and with any significant load, microtraumatization of the fibrous ring occurs with the appearance of cracks in it.

At stage II, fragments (sequesters) of the nucleus pulposus are squeezed into the spinal canal through cracks in the fibrous ring. The thickness of the intervertebral disc decreases (“disc settlement”).

At stage III (final), the disc is completely replaced by coarse-fibrous connective tissue, which is often ossified.

ds to a decrease in intradiscal pressure and a decrease in the water content in the nucleus; at the same time, a fibrous process develops, as a result, the cushioning properties of the nucleus decrease, and with any significant load, microtraumatization of the fibrous ring occurs with the appearance of cracks in it.

At stage II, fragments (sequesters) of the nucleus pulposus are squeezed into the spinal canal through cracks in the fibrous ring. The thickness of the intervertebral disc decreases (“disc settlement”).

At stage III (final), the disc is completely replaced by coarse-fibrous connective tissue, which is often ossified.

to a decrease in intradiscal pressure and a decrease in the water content in the nucleus; at the same time, a fibrous process develops, as a result, the cushioning properties of the nucleus decrease, and with any significant load, microtraumatization of the fibrous ring occurs with the appearance of cracks in it.

At stage II, fragments (sequesters) of the nucleus pulposus are squeezed into the spinal canal through cracks in the fibrous ring. The thickness of the intervertebral disc decreases (“disc settlement”).

At stage III (final), the disc is completely replaced by coarse-fibrous connective tissue, which is often ossified.

o a decrease in intradiscal pressure and a decrease in the water content in the nucleus; at the same time, a fibrous process develops, as a result, the cushioning properties of the nucleus decrease, and with any significant load, microtraumatization of the fibrous ring occurs with the appearance of cracks in it.

At stage II, fragments (sequesters) of the nucleus pulposus are squeezed into the spinal canal through cracks in the fibrous ring. The thickness of the intervertebral disc decreases (“disc settlement”).

At stage III (final), the disc is completely replaced by coarse-fibrous connective tissue, which is often ossified.

ecrease in intradiscal pressure and a decrease in the water content in the nucleus; at the same time, a fibrous process develops, as a result, the cushioning properties of the nucleus decrease, and with any significant load, microtraumatization of the fibrous ring occurs with the appearance of cracks in it.

At stage II, fragments (sequesters) of the nucleus pulposus are squeezed into the spinal canal through cracks in the fibrous ring. The thickness of the intervertebral disc decreases (“disc settlement”).

At stage III (final), the disc is completely replaced by coarse-fibrous connective tissue, which is often ossified.

rease in intradiscal pressure and a decrease in the water content in the nucleus; at the same time, a fibrous process develops, as a result, the cushioning properties of the nucleus decrease, and with any significant load, microtraumatization of the fibrous ring occurs with the appearance of cracks in it.

At stage II, fragments (sequesters) of the nucleus pulposus are squeezed into the spinal canal through cracks in the fibrous ring. The thickness of the intervertebral disc decreases (“disc settlement”).

At stage III (final), the disc is completely replaced by coarse-fibrous connective tissue, which is often ossified.

ase in intradiscal pressure and a decrease in the water content in the nucleus; at the same time, a fibrous process develops, as a result, the cushioning properties of the nucleus decrease, and with any significant load, microtraumatization of the fibrous ring occurs with the appearance of cracks in it.

At stage II, fragments (sequesters) of the nucleus pulposus are squeezed into the spinal canal through cracks in the fibrous ring. The thickness of the intervertebral disc decreases (“disc settlement”).

At stage III (final), the disc is completely replaced by coarse-fibrous connective tissue, which is often ossified.

nucleus; at the same time, a fibrous process develops, as a result, the cushioning properties of the nucleus decrease, and with any significant load, microtraumatization of the fibrous ring occurs with the appearance of cracks in it.

At stage II, fragments (sequesters) of the nucleus pulposus are squeezed into the spinal canal through cracks in the fibrous ring. The thickness of the intervertebral disc decreases (“disc settlement”).

At stage III (final), the disc is completely replaced by coarse-fibrous connective tissue, which is often ossified.

cleus; at the same time, a fibrous process develops, as a result, the cushioning properties of the nucleus decrease, and with any significant load, microtraumatization of the fibrous ring occurs with the appearance of cracks in it.

At stage II, fragments (sequesters) of the nucleus pulposus are squeezed into the spinal canal through cracks in the fibrous ring. The thickness of the intervertebral disc decreases (“disc settlement”).

At stage III (final), the disc is completely replaced by coarse-fibrous connective tissue, which is often ossified.

eus; at the same time, a fibrous process develops, as a result, the cushioning properties of the nucleus decrease, and with any significant load, microtraumatization of the fibrous ring occurs with the appearance of cracks in it.

At stage II, fragments (sequesters) of the nucleus pulposus are squeezed into the spinal canal through cracks in the fibrous ring. The thickness of the intervertebral disc decreases (“disc settlement”).

At stage III (final), the disc is completely replaced by coarse-fibrous connective tissue, which is often ossified.

t the same time, a fibrous process develops, as a result, the cushioning properties of the nucleus decrease, and with any significant load, microtraumatization of the fibrous ring occurs with the appearance of cracks in it.

At stage II, fragments (sequesters) of the nucleus pulposus are squeezed into the spinal canal through cracks in the fibrous ring. The thickness of the intervertebral disc decreases (“disc settlement”).

At stage III (final), the disc is completely replaced by coarse-fibrous connective tissue, which is often ossified.

the same time, a fibrous process develops, as a result, the cushioning properties of the nucleus decrease, and with any significant load, microtraumatization of the fibrous ring occurs with the appearance of cracks in it.

At stage II, fragments (sequesters) of the nucleus pulposus are squeezed into the spinal canal through cracks in the fibrous ring. The thickness of the intervertebral disc decreases (“disc settlement”).

At stage III (final), the disc is completely replaced by coarse-fibrous connective tissue, which is often ossified.

e same time, a fibrous process develops, as a result, the cushioning properties of the nucleus decrease, and with any significant load, microtraumatization of the fibrous ring occurs with the appearance of cracks in it.

At stage II, fragments (sequesters) of the nucleus pulposus are squeezed into the spinal canal through cracks in the fibrous ring. The thickness of the intervertebral disc decreases (“disc settlement”).

At stage III (final), the disc is completely replaced by coarse-fibrous connective tissue, which is often ossified.

ss develops, as a result, the cushioning properties of the nucleus decrease, and with any significant load, microtraumatization of the fibrous ring occurs with the appearance of cracks in it.

At stage II, fragments (sequesters) of the nucleus pulposus are squeezed into the spinal canal through cracks in the fibrous ring. The thickness of the intervertebral disc decreases (“disc settlement”).

At stage III (final), the disc is completely replaced by coarse-fibrous connective tissue, which is often ossified.

develops, as a result, the cushioning properties of the nucleus decrease, and with any significant load, microtraumatization of the fibrous ring occurs with the appearance of cracks in it.

At stage II, fragments (sequesters) of the nucleus pulposus are squeezed into the spinal canal through cracks in the fibrous ring. The thickness of the intervertebral disc decreases (“disc settlement”).

At stage III (final), the disc is completely replaced by coarse-fibrous connective tissue, which is often ossified.

evelops, as a result, the cushioning properties of the nucleus decrease, and with any significant load, microtraumatization of the fibrous ring occurs with the appearance of cracks in it.

At stage II, fragments (sequesters) of the nucleus pulposus are squeezed into the spinal canal through cracks in the fibrous ring. The thickness of the intervertebral disc decreases (“disc settlement”).

At stage III (final), the disc is completely replaced by coarse-fibrous connective tissue, which is often ossified.

es of the nucleus decrease, and with any significant load, microtraumatization of the fibrous ring occurs with the appearance of cracks in it.

At stage II, fragments (sequesters) of the nucleus pulposus are squeezed into the spinal canal through cracks in the fibrous ring. The thickness of the intervertebral disc decreases (“disc settlement”).

At stage III (final), the disc is completely replaced by coarse-fibrous connective tissue, which is often ossified.

of the nucleus decrease, and with any significant load, microtraumatization of the fibrous ring occurs with the appearance of cracks in it.

At stage II, fragments (sequesters) of the nucleus pulposus are squeezed into the spinal canal through cracks in the fibrous ring. The thickness of the intervertebral disc decreases (“disc settlement”).

At stage III (final), the disc is completely replaced by coarse-fibrous connective tissue, which is often ossified.

f the nucleus decrease, and with any significant load, microtraumatization of the fibrous ring occurs with the appearance of cracks in it.

At stage II, fragments (sequesters) of the nucleus pulposus are squeezed into the spinal canal through cracks in the fibrous ring. The thickness of the intervertebral disc decreases (“disc settlement”).

At stage III (final), the disc is completely replaced by coarse-fibrous connective tissue, which is often ossified.

the nucleus decrease, and with any significant load, microtraumatization of the fibrous ring occurs with the appearance of cracks in it.

At stage II, fragments (sequesters) of the nucleus pulposus are squeezed into the spinal canal through cracks in the fibrous ring. The thickness of the intervertebral disc decreases (“disc settlement”).

At stage III (final), the disc is completely replaced by coarse-fibrous connective tissue, which is often ossified.

e nucleus decrease, and with any significant load, microtraumatization of the fibrous ring occurs with the appearance of cracks in it.

At stage II, fragments (sequesters) of the nucleus pulposus are squeezed into the spinal canal through cracks in the fibrous ring. The thickness of the intervertebral disc decreases (“disc settlement”).

At stage III (final), the disc is completely replaced by coarse-fibrous connective tissue, which is often ossified.

nucleus decrease, and with any significant load, microtraumatization of the fibrous ring occurs with the appearance of cracks in it.

At stage II, fragments (sequesters) of the nucleus pulposus are squeezed into the spinal canal through cracks in the fibrous ring. The thickness of the intervertebral disc decreases (“disc settlement”).

At stage III (final), the disc is completely replaced by coarse-fibrous connective tissue, which is often ossified.

cleus decrease, and with any significant load, microtraumatization of the fibrous ring occurs with the appearance of cracks in it.

At stage II, fragments (sequesters) of the nucleus pulposus are squeezed into the spinal canal through cracks in the fibrous ring. The thickness of the intervertebral disc decreases (“disc settlement”).

At stage III (final), the disc is completely replaced by coarse-fibrous connective tissue, which is often ossified.

s decrease, and with any significant load, microtraumatization of the fibrous ring occurs with the appearance of cracks in it.

At stage II, fragments (sequesters) of the nucleus pulposus are squeezed into the spinal canal through cracks in the fibrous ring. The thickness of the intervertebral disc decreases (“disc settlement”).

At stage III (final), the disc is completely replaced by coarse-fibrous connective tissue, which is often ossified.

decrease, and with any significant load, microtraumatization of the fibrous ring occurs with the appearance of cracks in it.

At stage II, fragments (sequesters) of the nucleus pulposus are squeezed into the spinal canal through cracks in the fibrous ring. The thickness of the intervertebral disc decreases (“disc settlement”).

At stage III (final), the disc is completely replaced by coarse-fibrous connective tissue, which is often ossified.

crease, and with any significant load, microtraumatization of the fibrous ring occurs with the appearance of cracks in it.

At stage II, fragments (sequesters) of the nucleus pulposus are squeezed into the spinal canal through cracks in the fibrous ring. The thickness of the intervertebral disc decreases (“disc settlement”).

At stage III (final), the disc is completely replaced by coarse-fibrous connective tissue, which is often ossified.

se, and with any significant load, microtraumatization of the fibrous ring occurs with the appearance of cracks in it.

At stage II, fragments (sequesters) of the nucleus pulposus are squeezed into the spinal canal through cracks in the fibrous ring. The thickness of the intervertebral disc decreases (“disc settlement”).

At stage III (final), the disc is completely replaced by coarse-fibrous connective tissue, which is often ossified.

, and with any significant load, microtraumatization of the fibrous ring occurs with the appearance of cracks in it.

At stage II, fragments (sequesters) of the nucleus pulposus are squeezed into the spinal canal through cracks in the fibrous ring. The thickness of the intervertebral disc decreases (“disc settlement”).

At stage III (final), the disc is completely replaced by coarse-fibrous connective tissue, which is often ossified.

with any significant load, microtraumatization of the fibrous ring occurs with the appearance of cracks in it.

At stage II, fragments (sequesters) of the nucleus pulposus are squeezed into the spinal canal through cracks in the fibrous ring. The thickness of the intervertebral disc decreases (“disc settlement”).

At stage III (final), the disc is completely replaced by coarse-fibrous connective tissue, which is often ossified.

th any significant load, microtraumatization of the fibrous ring occurs with the appearance of cracks in it.

At stage II, fragments (sequesters) of the nucleus pulposus are squeezed into the spinal canal through cracks in the fibrous ring. The thickness of the intervertebral disc decreases (“disc settlement”).

At stage III (final), the disc is completely replaced by coarse-fibrous connective tissue, which is often ossified.

any significant load, microtraumatization of the fibrous ring occurs with the appearance of cracks in it.

At stage II, fragments (sequesters) of the nucleus pulposus are squeezed into the spinal canal through cracks in the fibrous ring. The thickness of the intervertebral disc decreases (“disc settlement”).

At stage III (final), the disc is completely replaced by coarse-fibrous connective tissue, which is often ossified.

icrotraumatization of the fibrous ring occurs with the appearance of cracks in it.

At stage II, fragments (sequesters) of the nucleus pulposus are squeezed into the spinal canal through cracks in the fibrous ring. The thickness of the intervertebral disc decreases (“disc settlement”).

At stage III (final), the disc is completely replaced by coarse-fibrous connective tissue, which is often ossified.

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Nerves. 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13. 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